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Expression of concern "DAMPs in immunosenescence and cancer" [Semin. Cancer Biol. 106-107 (2024) 123-142]. “DAMPs在免疫衰老和癌症中的表达”[Semin.]中国生物医学工程学报[j].中国生物医学工程杂志。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.014
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引用次数: 0
Expression of concern "Immunosenescence in digestive system cancers: Mechanisms, research advances, and therapeutic strategies" [Semin. Cancer Biol. 106-107 (2024) 234-250]. “消化系统癌症中的免疫衰老：机制、研究进展和治疗策略”[Semin]。中国生物医学工程学报[j].中国生物医学工程杂志。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.011
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引用次数: 0
Expression of concern "Brain macrophage senescence in glioma" [Semin. Cancer Biol. 104-105 (2024) 46-60]. “脑巨噬细胞衰老在胶质瘤中的表达”[Semin。中国生物医学工程学报[j].中国生物医学工程学报，2016,32(5):559 - 561。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-08 DOI: 10.1016/j.semcancer.2025.11.020
{"title":"Expression of concern \"Brain macrophage senescence in glioma\" [Semin. Cancer Biol. 104-105 (2024) 46-60].","authors":"","doi":"10.1016/j.semcancer.2025.11.020","DOIUrl":"https://doi.org/10.1016/j.semcancer.2025.11.020","url":null,"abstract":"","PeriodicalId":21594,"journal":{"name":"Seminars in cancer biology","volume":"117 ","pages":"126"},"PeriodicalIF":15.7,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145715529","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Expression of concern "Mechanisms and strategies of immunosenescence effects on non-small cell lung cancer (NSCLC) treatment: A comprehensive analysis and future directions" [Semin. Cancer Biol. 109 (2025) 44-66]. “免疫衰老对非小细胞肺癌（NSCLC）治疗的机制和策略：综合分析和未来方向”[Semin。中国生物医学工程学报，2009(5):444 - 466。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.016
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引用次数: 0
Expression of concern "Psychological stress on cancer progression and immunosenescence" [Semin. Cancer Biol. 113 (2025) 85-99]. “心理压力对癌症进展和免疫衰老的影响”[Semin.]中国生物医学工程学报，2015(5):389 - 391。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.007
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引用次数: 0
Expression of concern "T-cell senescence: Unlocking the tumor immune "Dark Box" - A multidimensional analysis from mechanism to tumor immunotherapeutic intervention" [Semin. Cancer Biol. 113 (2025) 190-209]. “t细胞衰老：打开肿瘤免疫“黑箱”——从机制到肿瘤免疫治疗干预的多维分析”[Semin.]中国生物医学工程学报，2016,31(5):391 - 391。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.005
{"title":"Expression of concern \"T-cell senescence: Unlocking the tumor immune \"Dark Box\" - A multidimensional analysis from mechanism to tumor immunotherapeutic intervention\" [Semin. Cancer Biol. 113 (2025) 190-209].","authors":"","doi":"10.1016/j.semcancer.2025.11.005","DOIUrl":"https://doi.org/10.1016/j.semcancer.2025.11.005","url":null,"abstract":"","PeriodicalId":21594,"journal":{"name":"Seminars in cancer biology","volume":"117 ","pages":"125"},"PeriodicalIF":15.7,"publicationDate":"2025-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"145715518","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":1,"RegionCategory":"医学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Expression of concern "Immune senescence: A key player in cancer biology" [Semin. Cancer Biol. 108 (2025) 71-82]. 关注表达“免疫衰老：癌症生物学中的关键角色”[Semin。中国生物医学工程学报，2015(5):387 - 391。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.017
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引用次数: 0
Expression of concern "Study of immunosenescence in the occurrence and immunotherapy of gastrointestinal malignancies" [Semin. Cancer Biol. 111 (2025) 16-35]. “免疫衰老在胃肠道恶性肿瘤发生及免疫治疗中的研究”[Semin。中国生物医学工程杂志，2011(5):357 - 357。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-12-01 Epub Date: 2025-11-07 DOI: 10.1016/j.semcancer.2025.11.010
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引用次数: 0
Lipid metabolism in cancer is regulated by the integrated natural product–miRNA–target axis 肿瘤中的脂质代谢受天然产物- mirna -靶标轴的综合调控。
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-11-29 DOI: 10.1016/j.semcancer.2025.11.024
Ya-Ting Chuang , Ching-Yu Yen , Yi-Hong Tsai , Fang-Rong Chang , Ammad Ahmad Farooqi , Hsueh-Wei Chang
Signaling lipids have been shown to play multifaceted roles ranging from cell proliferation, metabolism, invasion, migration, and apoptosis. Mechanistically, these enzymes and their downstream targets constitute complicated and intertwined lipid signaling networks with multiple nodes of interaction and cross-regulations. Dysregulated lipid metabolism is one of the hallmarks of cancer. Targeting such lipid metabolism is a potential anticancer strategy. Several natural products and miRNAs have been reported to modulate cancer cell fatty acid (FA) metabolisms; however, the integrated connections between these natural products, miRNAs, and targets have yet to be evaluated in detail. This review uses the bioinformatic STRING tool to explore target-target interactions between Google Scholar-retrieval targets of dysregulated FA metabolism in cancer cells. miRNAs and natural products that dysregulate FA metabolism were also retrieved and connected to the STRING–target network with Google Scholar and the bioinformatic databases (miRTarBase and miRDB), which retrieves targets for miRNAs. Moreover, the knowledge gaps between miRNA- and natural product-modulated FA metabolism are intricately connected. Consequently, this review highlights and shows interactions within the natural product–miRNA–target axis in counteracting the dysregulated FA metabolism in cancer cells.
信号脂质在细胞增殖、代谢、侵袭、迁移和凋亡等方面发挥着多方面的作用。从机制上讲，这些酶及其下游靶点构成了复杂而交织的脂质信号网络，具有多个相互作用和交叉调节的节点。脂质代谢失调是癌症的标志之一。靶向这种脂质代谢是一种潜在的抗癌策略。一些天然产物和mirna已经被报道可以调节癌细胞脂肪酸（FA）代谢；然而，这些天然产物、mirna和靶标之间的综合联系尚未得到详细的评估。本文利用生物信息学的STRING工具来探讨肿瘤细胞中FA代谢失调的谷歌学者检索靶标之间的靶标相互作用。通过谷歌Scholar和生物信息学数据库（miRTarBase和miRDB）检索并连接到STRING-target网络，检索mirna的靶标。此外，miRNA和天然产物调节的FA代谢之间的知识差距是错综复杂的。因此，本综述强调并展示了天然产物- mirna -靶标轴在对抗癌细胞中失调的FA代谢中的相互作用。
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引用次数: 0
Microbiota metabolism and immune regulation: From mechanisms to immunotherapeutic applications in cancer 微生物群代谢和免疫调节：从机制到癌症的免疫治疗应用
IF 15.7 1区 医学 Q1 ONCOLOGY
Seminars in cancer biology
Pub Date : 2025-11-19 DOI: 10.1016/j.semcancer.2025.11.023
Sichen Yuan , Alexa Bremmer , Xicheng Yang , Quanyin Hu
The human microbiome plays a pivotal role in shaping immune responses by producing bioactive metabolites such as bile acids (BAs) and short-chain fatty acids (SCFAs). These microbial metabolites influence a broad range of cell types, including innate and adaptive immune cells and non-immune cells. Emerging evidence suggests that microbiome metabolism is a crucial factor affecting the success of cancer immunotherapy. Promising therapeutic approaches, such as the use of probiotics, engineered bacteria, metabolite supplementation, and bile acid sequestrants, are being explored to enhance immunotherapeutic outcomes. This review critically evaluates current insights into how microbiome-derived metabolites modulate cellular functions, with a particular focus on immune cells within the tumor microenvironment, and highlights their potential to improve the efficacy of cancer immunotherapy.
人类微生物组通过产生生物活性代谢物，如胆汁酸（BAs）和短链脂肪酸（SCFAs），在形成免疫反应中起着关键作用。这些微生物代谢物影响广泛的细胞类型，包括先天和适应性免疫细胞和非免疫细胞。新出现的证据表明，微生物组代谢是影响癌症免疫治疗成功的关键因素。目前正在探索有前景的治疗方法，如使用益生菌、工程菌、代谢物补充和胆汁酸隔离剂，以提高免疫治疗效果。这篇综述批判性地评估了微生物组衍生代谢物如何调节细胞功能的当前见解，特别关注肿瘤微环境中的免疫细胞，并强调了它们提高癌症免疫治疗疗效的潜力。
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引用次数: 0
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