Research on Child and Adolescent Psychopathology最新文献

This study examined the interplay of psychopathic traits, executive functioning, and antisocial behavior among adjudicated youth, with a focus on the potential moderating role of executive function. The current study uses data from the Pathways to Desistance dataset was examined, utilizing the Psychopathy Checklist: Youth Version (PCL-YV) and the Stroop Color-Word Task to measure psychopathic traits and executive functioning, respectively. Violent and property offending frequencies were self-reported. Both psychopathic traits and lower executive functioning were initially associated with higher frequencies of both violent and property offending. Crucially, a significant interaction emerged: Youth exhibiting higher socially deviant/lifestyle psychopathic traits and weaker executive function were most likely to engage in property offenses. These findings offer insights into specific risk profiles for offending behaviors and underscore the importance of interventions promoting executive function, especially for youth with these characteristics. This study highlights the complex ways in which individual differences contribute to antisocial outcomes.

Preschool-onset major depressive disorder (PO-MDD) is an impairing pediatric mental health disorder that impacts children as young as three years old. There is limited work dedicated to uncovering neural measures of this early childhood disorder which could be leveraged to further understand both treatment responsiveness and future depression risk. Event-related potentials (ERPs) such as the P300 have been employed extensively in adult populations to examine depression-related deficits in cognitive and motivational systems. Few studies examine the prospective relationships between depression and P300, especially in young children. Moreover, limited research examines the relationship between P300 with psychotherapy treatment responsiveness in youths. The current study sought to examine the prospective relationships between pre-intervention P300 (i.e., choice-locked) elicited from the doors task in depressed preschool children (i.e., PO-MDD; ages 3-to-6) with reductions in depressive symptoms after completing an 18-week long dyadic psychotherapy intervention (n = 59). We also explored relations to risk for depression assessed at a follow-up visit during preadolescence (ages 8-to-12; n = 82). Those with PO-MDD exhibiting reduced choice (doors)-locked P300 demonstrated worse treatment response to psychotherapy and were more likely to meet criteria for depression during preadolescence. Moreover, the relationship between pre-intervention P300 and later preadolescence depression was significantly mediated by response to treatment. These findings suggest that deficits in brain systems linked to the choice-locked P300 component (i.e., cognitive and motivational) might be indicative of non-responsiveness to early dyadic psychotherapeutic intervention efforts for depression which impacts risk for recurrent patterns of depression in youths.

Developmental changes in youth sleep preferences (chronotype) and pubertal development are consequential for youth risk for depression. Previous research has identified individual differences in chronotype in risk for psychopathology. However, little is known regarding how the timing of chronotype may confer risk in youth. This study addressed this gap by examining associations between chronotypal timing and symptoms of depression in youth. Community youth (N = 155; $M_{\mathrm{age}}$ =12.7) completed self-report measures of chronotype, pubertal status and depression every six months for a period of one year (three assessment points). Regression analyses showed that chronotypal timing predicted change in depressive symptoms across six months (b = -0.66, p = 0.019), but not across any other timeframe. Findings suggested that youth experiencing more of a morning preference compared to same-aged peers were at increased risk for later depression across six months. Chronotypal timing continued to predict changes in symptoms of depression controlling for gender (b = -0.63, p = 0.023) and pubertal timing (b = -0.72, p = 0.012). These findings suggest that chronotypal timing is prospectively related to changes in youth symptoms of depression. Results indicate that attention to the timing of normative changes in chronotype is warranted, in addition to mean-level differences.

Neurodivergent youth often experience anxiety and depressive symptoms that may hamper adaptive functioning and well-being. There is little knowledge of how anxiety and depression are related in neurodivergent youth. Therefore, we aimed to examine whether the relationship between anxiety and depressive symptoms is uni- or bidirectional in neurodiverse youth. We assessed self-reported anxiety and depressive symptoms over time in 173 youth (M_{baseline age} = 11.7 years, SD = 2.1, 64% males, 36% females). The sample comprised 38 autistic youth, 85 youth diagnosed with attention-deficit/hyperactivity disorder (ADHD), and 50 comparison youth assessed at baseline (T1), 2-year follow-up (T2, 97% retention), and 10-year follow-up (T3, 73% retention). We used cross-lagged models to analyze the data. In neurodivergent youth, more anxiety symptoms at T1 and T2 predicted more depressive symptoms at T2 and T3. Preceding anxiety symptoms were linked with later depressive symptoms, even after accounting for autoregressive effects of depressive symptoms. The results are consistent with a prodromal model in which anxiety symptoms can independently foreshadow the emergence of depressive symptoms over the course of development among neurodivergent youth. Potentially, addressing anxiety symptoms among youth with autism or ADHD could play a role in preventing the onset of youth depression.

Youth with complex health needs (CHNs; e.g., requiring daily assistance or equipment for care) and their parents face heightened vulnerabilities during natural disasters, potentially leading to poorer mental health outcomes compared to those without CHNs. However, limited research has focused on this group's disaster-related experiences and their impact on mental health outcomes. This study aimed to investigate the disaster experiences, perceptions, and mental health outcomes of youth with CHNs and their parents' post-hurricane and to evaluate the unique influence of CHN- and disaster-related factors on their psychological functioning. Parents (N = 142) of youth with CHNs (n = 48) and without CHNs (n = 94) who experienced a hurricane reported on their youth's and their own psychological functioning, disaster perceptions, experiences, and CHN-related information. Youth with CHNs exhibited greater perceived life threat compared to youth without CHNs. Families of youth with CHNs were more likely to evacuate and faced greater evacuation barriers. They also exhibited greater PTS, depressive, and anxiety symptom severity compared to those without CHNs. Financial healthcare concerns were not associated with youth with CHNs or their parents' mental health symptomatology. Hurricane-impeded access to healthcare necessities was associated with youth and parent PTS and depressive symptom severity and youth anxiety symptom severity. These findings underscore the vulnerabilities of youth with CHNs and their parents' post-hurricane, emphasizing the need for tailored mental health services and improved disaster planning resources to support this population effectively.

Parental depression is associated with offspring depression and sleep problems are prospectively associated with the development of depression. However, little work has examined sleep problems in the offspring of depressed parents and whether these problems partially account for the association between parent and offspring depression. This longitudinal study examined the indirect effect of sleep problems on the association between parent psychopathology and offspring depression in a sample of 10,953 10 to 12-year-old children participating in the Adolescent Brain and Cognitive Development (ABCD) study. Controlling for age, sex, and other forms of parent psychopathology, we found significant indirect effects of parent to offspring depression through parent and youth reports of youth insomnia and hypersomnia. We also found indirect effects of parent history of anxiety and drug use problems to offspring depression through insomnia, and indirect effects of parent history of anxiety, drug use problems, and alcohol use problems to offspring depression through hypersomnia. Our findings show that sleep may be a mechanism of the transmission of parent depression, anxiety, drug use problems, and alcohol use problems to offspring depression. Mitigating sleep problems represents a potential avenue for preventative interventions in youth with a heightened susceptibility to depression.

Despite frequent reliance on teacher and parent ratings of children's behavior for multi-informant assessment, agreement between teachers' and parents' ratings is low. This study examined the predictive utility of teacher and parent ratings for children's self-regulatory outcomes (i.e., executive function, continuous performance task) in four studies. Study 1 included 163 children ranging from 31 to 84 months of age (M = 55.58 months, SD = 8.43). Study 2 included 1,088 children ranging from 48 to 63 months of age (M = 55.15, SD = 3.65). Study 3 included 246 bilingual Spanish-speaking children ranging from 40 to 72 months of age (M = 56.66, SD = 6.06). Study 4 included 280 children ranging from 38 to 75 months of age (M = 55.92, SD = 4.16). Across studies, parents and teachers rated children's externalizing behaviors on the Conners' Rating Scale, the Strength and Weaknesses of ADHD-Symptoms and Normal Behavior Scale, or both; children completed a variety of performance-based self-regulation tasks. The strength of associations of parent and teacher ratings with self-regulatory outcomes was compared, and regression analyses determined the unique and overlapping variance accounted for by different raters. Teachers' ratings had larger associations with self-regulation than did parents' ratings across outcomes and studies-except for two instances in Study 4 where the associations for teachers' and parents' ratings were equal. These findings indicate that teachers supply more useful information than parents, possibly because teachers have better-informed expectations of children's behavior, and they raise questions about the utility of multi-informant assessment, at least with preschool children.

The aim of this study is to investigate the impact of using probiotics with strains related to dopamine and gamma-aminobutyric acid production on clinical features of autism spectrum disorder (ASD) and/or attention deficit/hyperactivity disorder (ADHD). This randomized, controlled trial involved 38 children with ADHD and 42 children with ASD, aged 5-16 years, who received probiotics (Lactiplantibacillus plantarum and Levilactobacillus brevis 109/cfu/daily) or placebo for 12 weeks. Parent-reported symptoms were assessed using Conners' 3rd-Ed and the Social Responsiveness Scale Test, 2nd-Ed (SRS-2), and children completed the Conners Continuous Performance Test, 3rd-Ed (CPT 3) or Conners Kiddie CPT, 2nd-Ed (K-CPT 2). Executive functions, quality of life and sleep patterns were also parent-assessed. Intention-to-treat analyses, controlling for sociodemographic and nutritional covariates, revealed no significant inter-group differences in parent-reported or neuropsychological data after the probiotic intervention. However, age-stratified analyses showed improved hyperactivity-impulsivity symptoms in younger children with ASD (Cohen's d = 1.245) and ADHD (Cohen's d = 0.692). Intra-group analyses supported these findings in the aforementioned age and intervention group for both diagnoses. An improvement in impulsivity for children with ASD was also observed in the intra-group analysis of the CPT commissions scores (probiotic: p = 0.001, Cohen's d = -1.216; placebo: p = 0.013, Cohen's d = -0.721). A better comfort score (quality of life) was shown in children with ASD (probiotic: p = 0.010, Cohen's d = 0.722; placebo: p = 0.099, Cohen's d = 0.456). The probiotics used, may improve hyperactivity-impulsivity in children with ASD or/and ADHD and quality of life in children with ASD. Further research is warranted to explore probiotics as an adjunctive therapeutic intervention for NDs.Trial registration: clinicaltrials.gov Identifier: NCT05167110.

To prevent young children's injuries, studies have considered both child (e.g., temperament, age, sex) and parent factors (e.g., parental supervision and style, attachment) associated with risk-taking behaviors. Building on risk-taking theory literature, Jonas and Kochanska (Jonas & Kochanska, Journal of Abnormal Child Psychology 46:1573-1583, 2018) adapted the dual systems model (Steinberg, Developmental Review 28:78-106, 2008) to children and suggested that risk-taking propensity arises from an imbalance between the overactivation of the child's socioemotional system (sensation seeking or traits of surgency) and the lower cognitive control system (lack of self-regulation or of effortful control). However, from an intergenerational transmission perspective, it is relevant to consider the role both parents' and the children's socioemotional and cognitive control systems have on a child's risk-taking behaviors. The current longitudinal study is the first to examines sensation seeking and lack of self-regulation in parents in addition to the child's surgency-effortful control imbalance to understand the child's risk-taking behaviors. The sample comprised 177 two-parent families (89 boys) observed at two time points (child age ranges: 12-18 months and 24-30 months). Both parents provided sociodemographic information and completed self-reported questionnaires on sensation seeking and self-regulation, child's temperament and risk-taking behaviors. Results showed that fathers' higher sensation-seeking and mothers' lack of self-regulation were associated with higher children's risk-taking behaviors. After controlling for these parent factors and child sex, child surgency-effortful imbalance was strongly associated with higher children's risk-taking behaviors. An adapted dual systems model including both parents (sensation seeking and self-regulation) and children (surgency-effortful imbalance) seems a promising avenue to a fuller understanding of children's risk-taking behaviors.

Recently, an association between cognitive disengagement syndrome (CDS), formerly sluggish cognitive tempo, and autism has been documented, but it is not known if the association is due to overlapping autism and CDS traits or if CDS is empirically distinct from autism. Mothers rated 2,209 children 4-17 years (1,177 with autism, 725 with ADHD-Combined type, and 307 with ADHD-Inattentive type) on the Pediatric Behavior Scale. Factor analysis of the Pediatric Behavior Scale items indicated that CDS and autism traits are empirically distinct from each other without cross-loading and are distinct from eight other factors (attention deficit, impulsivity, hyperactivity, oppositional behavior, irritability/anger, conduct problems, depression, and anxiety). CDS total scores were significantly higher in the autism + ADHD-Inattentive and autism + ADHD-Combined groups than in the autism, ADHD-Combined, and ADHD-Inattentive only groups with a nonsignificant difference between the latter three groups. CDS and autism are empirically distinct from each other and from other psychopathology dimensions. Overlapping traits do not explain the association between autism and CDS. Autism in combination with ADHD-Combined or ADHD-Inattentive increases the likelihood of CDS relative to youth who have autism, ADHD-Combined, or ADHD-Inattentive only. Because of the known associations between autism, CDS, and ADHD, both autism and ADHD must be assessed in CDS research and clinically to better understand and explain research findings and provide targeted clinical intervention.