Pub Date : 2025-03-01DOI: 10.1016/S1619-9987(25)00023-6
{"title":"Neuafnahmen (Stand 01.01.25)","authors":"","doi":"10.1016/S1619-9987(25)00023-6","DOIUrl":"10.1016/S1619-9987(25)00023-6","url":null,"abstract":"","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Page 67"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679878","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2025-03-01DOI: 10.1016/j.fuspru.2025.01.002
M. Jordan
{"title":"Incidence of complications with precontoured allograft wedges in foot and ankle surgery","authors":"M. Jordan","doi":"10.1016/j.fuspru.2025.01.002","DOIUrl":"10.1016/j.fuspru.2025.01.002","url":null,"abstract":"","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Page 61"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679870","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2025-03-01DOI: 10.1016/j.fuspru.2025.03.001
{"title":"Knochenmarködem und Osteonekrosen an Fuß und Sprunggelenk","authors":"","doi":"10.1016/j.fuspru.2025.03.001","DOIUrl":"10.1016/j.fuspru.2025.03.001","url":null,"abstract":"","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Page 1"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679874","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2025-03-01DOI: 10.1016/j.fuspru.2025.01.005
Alena Richter, Ulrich Wiebking, Steffen Böddeker, Tobias Finck, Sophie Hügel, Christina Stukenborg-Colsman, Christian Plaaß
Avascular necrosis of the sesamoids is a rare differential diagnosis for plantar pain of the first metatarsal head. A prevalence of 1% is assumed. The exact etiolog is not known, but it correlates with increased forefoot loading in athletes or in pes cavus. MRI allows early diagnosis, whereas specific pathologic changes with consolidation and fragmentation are visible in radiography only after 6 to 12 month and have to be differentiated from fractures and bipartide sesamoides. Treatment is primarily conservative comprising off-loading of the forefoot, immobilization in a walker and the use of NSAIDs. Infiltrations with PRP and shockwave therapy have also shown promising results in individual cases. If symptoms persist, sesamoidectomy has shown satisfactory results. Complications such as postoperative hallux valgus or hallux varus seldom occur, but have to be considered.
{"title":"Avaskuläre Nekrosen der Sesambeine","authors":"Alena Richter, Ulrich Wiebking, Steffen Böddeker, Tobias Finck, Sophie Hügel, Christina Stukenborg-Colsman, Christian Plaaß","doi":"10.1016/j.fuspru.2025.01.005","DOIUrl":"10.1016/j.fuspru.2025.01.005","url":null,"abstract":"<div><div>Avascular necrosis of the sesamoids is a rare differential diagnosis for plantar pain of the first metatarsal head. A prevalence of 1% is assumed. The exact etiolog is not known, but it correlates with increased forefoot loading in athletes or in pes cavus. MRI allows early diagnosis, whereas specific pathologic changes with consolidation and fragmentation are visible in radiography only after 6 to 12 month and have to be differentiated from fractures and bipartide sesamoides. Treatment is primarily conservative comprising off-loading of the forefoot, immobilization in a walker and the use of NSAIDs. Infiltrations with PRP and shockwave therapy have also shown promising results in individual cases. If symptoms persist, sesamoidectomy has shown satisfactory results. Complications such as postoperative hallux valgus or hallux varus seldom occur, but have to be considered.</div></div>","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Pages 15-24"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679875","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2025-03-01DOI: 10.1016/j.fuspru.2025.01.003
Lubomir Kopp , Vit Baba , Christine Marx , Stefan Rammelt
About three of four cases of avascular necrosis (AVN) of the talus are of posttraumatic origin. Risk factors include displacement, open fractures and dislocations, patient age, high BMI and smoking. For the management of adverse sequelae of talar fractures like malunion and nonunion, the distinction between partial and total AVN with collapse of the talar dome is of great relevance.
Treatment options for precollapse AVN include protection, medication, extracorporeal shock wave therapy, bone marrow aspirate, drilling, and (vascularized) bone grafting. In the presence of partial AVN, malunions or nonunions of the talar neck and body may be treated with joint-preserving corrections in active, compliant patients, provided a sufficient vital cartilage and bone stock is present.
Complete talar AVN with collapse may be salvaged with necrectomy, bone grafting and fusion of arthritic joints. Recently, custom 3D prostheses providing either total talar replacement or talar body replacement with preservation of the talar head, have gained more attention, but long-term results are missing. Talar replacement may also be combined with ankle replacement and / or subtalar fusion.
Septic AVN of the talar body as the worst case scenario warrants staged treatment with radical debridements until negative swabs are obtained. Besides allograft and autograft bone, customized cages, trabecular metal, or biomaterials like bioglass with antiinfective properties may be used for secondary defect filling. Reconstruction and fusion is obtained with internal or external fixation. With critical soft tissue conditions, partial or total astragalectomy and tibiocalcaneal fusion may serve as a salvage procedure.
{"title":"Post-traumatic osteonecrosis of the talus","authors":"Lubomir Kopp , Vit Baba , Christine Marx , Stefan Rammelt","doi":"10.1016/j.fuspru.2025.01.003","DOIUrl":"10.1016/j.fuspru.2025.01.003","url":null,"abstract":"<div><div>About three of four cases of avascular necrosis (AVN) of the talus are of posttraumatic origin. Risk factors include displacement, open fractures and dislocations, patient age, high BMI and smoking. For the management of adverse sequelae of talar fractures like malunion and nonunion, the distinction between partial and total AVN with collapse of the talar dome is of great relevance.</div><div>Treatment options for precollapse AVN include protection, medication, extracorporeal shock wave therapy, bone marrow aspirate, drilling, and (vascularized) bone grafting. In the presence of partial AVN, malunions or nonunions of the talar neck and body may be treated with joint-preserving corrections in active, compliant patients, provided a sufficient vital cartilage and bone stock is present.</div><div>Complete talar AVN with collapse may be salvaged with necrectomy, bone grafting and fusion of arthritic joints. Recently, custom 3D prostheses providing either total talar replacement or talar body replacement with preservation of the talar head, have gained more attention, but long-term results are missing. Talar replacement may also be combined with ankle replacement and / or subtalar fusion.</div><div>Septic AVN of the talar body as the worst case scenario warrants staged treatment with radical debridements until negative swabs are obtained. Besides allograft and autograft bone, customized cages, trabecular metal, or biomaterials like bioglass with antiinfective properties may be used for secondary defect filling. Reconstruction and fusion is obtained with internal or external fixation. With critical soft tissue conditions, partial or total astragalectomy and tibiocalcaneal fusion may serve as a salvage procedure.</div></div>","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Pages 41-59"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679877","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2025-03-01DOI: 10.1016/j.fuspru.2025.01.006
Manuel Monteagudo , Ernesto Maceira , Ángel Orejana , Gastón Slullitel , Pilar Martínez-de-Albornoz , Anna Oller
Müller-Weiss disease (MWD) is a condition that involves a dysplastic navicular bone with asymmetric talonavicular arthritis and secondary hindfoot varus deformity. Although its prevalence is supposed to be low, it is much more common than reported as many cases are asymptomatic, undiagnosed, or misdiagnosed. Understanding aetiology and epidemiology is important to know about the background of MWD. Although it has traditionally been presented as a necrosis of the navicular, we now know it is apparently the result of mechanical disturbances occurring around the bone that ultimately cause deformity and an asymmetric arthritis of the talonavicular joint. The uneven wear and progressive thinning of the lateral part of the joint gradually shifts the talus over the calcaneus thus creating subtalar/hindfoot varus. Rotational and medializing moments acting on the dysplastic navicular cause medial protrusion and plantar collapse of the bone. It is essential to understand pathogenesis as it directly relates to diagnosis and management of this condition. Conservative treatment involves correcting subtalar varus as a deforming force around the midfoot and hindfoot. Insoles with a pronatory wedge from the heel to the metatarsal region and longitudinal arch support are usually effective in providing pain relief and improving function. Patients who fail to respond to insoles may be treated surgically. Conventional talonavicular (or extended) arthrodesis does not address the underlying pathomechanics. An isolated valgus (Dwyer-type with lateral slide) osteotomy of the os calcis has shown to be effective for most patients. After 30 years studying MWD, we will try to provide a basic understanding of the pathogenesis, pathomechanics, diagnosis, and ultimately both conservative and surgical treatment of this challenging and not so uncommon condition.
{"title":"Müller Weiss Disease: Our 30-year-experience","authors":"Manuel Monteagudo , Ernesto Maceira , Ángel Orejana , Gastón Slullitel , Pilar Martínez-de-Albornoz , Anna Oller","doi":"10.1016/j.fuspru.2025.01.006","DOIUrl":"10.1016/j.fuspru.2025.01.006","url":null,"abstract":"<div><div>Müller-Weiss disease (MWD) is a condition that involves a dysplastic navicular bone with asymmetric talonavicular arthritis and secondary hindfoot varus deformity. Although its prevalence is supposed to be low, it is much more common than reported as many cases are asymptomatic, undiagnosed, or misdiagnosed. Understanding aetiology and epidemiology is important to know about the background of MWD. Although it has traditionally been presented as a necrosis of the navicular, we now know it is apparently the result of mechanical disturbances occurring around the bone that ultimately cause deformity and an asymmetric arthritis of the talonavicular joint. The uneven wear and progressive thinning of the lateral part of the joint gradually shifts the talus over the calcaneus thus creating subtalar/hindfoot varus. Rotational and medializing moments acting on the dysplastic navicular cause medial protrusion and plantar collapse of the bone. It is essential to understand pathogenesis as it directly relates to diagnosis and management of this condition. Conservative treatment involves correcting subtalar varus as a deforming force around the midfoot and hindfoot. Insoles with a pronatory wedge from the heel to the metatarsal region and longitudinal arch support are usually effective in providing pain relief and improving function. Patients who fail to respond to insoles may be treated surgically. Conventional talonavicular (or extended) arthrodesis does not address the underlying pathomechanics. An isolated valgus (Dwyer-type with lateral slide) osteotomy of the os calcis has shown to be effective for most patients. After 30 years studying MWD, we will try to provide a basic understanding of the pathogenesis, pathomechanics, diagnosis, and ultimately both conservative and surgical treatment of this challenging and not so uncommon condition.</div></div>","PeriodicalId":39776,"journal":{"name":"Fuss und Sprunggelenk","volume":"23 1","pages":"Pages 25-40"},"PeriodicalIF":0.0,"publicationDate":"2025-03-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143679876","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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