Pub Date : 2022-10-12DOI: 10.1177/26324636221123366
Gautam Kumar Bunker, Manoj Patidar, Devendra Atal, R. Meena
Background: The incidence of premature coronary artery disease (PCAD) is on rising trend in low- and middle-income countries. Considering the limited reports, the current study was conducted to evaluate the prevalence, various predisposing factors, and coronary angiographic findings of PCAD in North India. Methods: Study design—hospital-based analytical cross-sectional study. Setting: Outpatient and inpatient units of a tertiary cardiac center in North India. Two hundred sequential patients (170 males and 30 females) with acute coronary syndrome were evaluated for history of risk factors, measurement of anthropometric parameters, biochemical blood examination, electrocardiogram, complete dental examination, and angiographic assessment of coronary artery lesion. Patients with acute infection or chronic inflammatory disorders were excluded. Patients were divided into PCAD (age ≤45 years) and mature CAD. Results: The prevalence of PCAD was 15% (N = 30). PCAD had higher rates of family history of CAD (P < 0.001), tobacco consumption (P = 0.01), smoking (P = 0.004), and single-vessel disease (SVD) (P < 0.001). Premature CAD group had significantly lower mean fasting blood glucose (105 ± 41 mg/dL vs 127 ± 45 mg/dL) levels. Conclusion: Family history of CAD, smoking, and tobacco consumption are important risk factors of PCAD. SVD is more common in PCAD. Screening the susceptible population at risk and controlling the cardiovascular risk factors will help reduce the epidemic and consequences of PCAD.
{"title":"Evaluation of Risk Factors of Premature Coronary Artery Disease in Patients From North India: A Rising Epidemic","authors":"Gautam Kumar Bunker, Manoj Patidar, Devendra Atal, R. Meena","doi":"10.1177/26324636221123366","DOIUrl":"https://doi.org/10.1177/26324636221123366","url":null,"abstract":"Background: The incidence of premature coronary artery disease (PCAD) is on rising trend in low- and middle-income countries. Considering the limited reports, the current study was conducted to evaluate the prevalence, various predisposing factors, and coronary angiographic findings of PCAD in North India. Methods: Study design—hospital-based analytical cross-sectional study. Setting: Outpatient and inpatient units of a tertiary cardiac center in North India. Two hundred sequential patients (170 males and 30 females) with acute coronary syndrome were evaluated for history of risk factors, measurement of anthropometric parameters, biochemical blood examination, electrocardiogram, complete dental examination, and angiographic assessment of coronary artery lesion. Patients with acute infection or chronic inflammatory disorders were excluded. Patients were divided into PCAD (age ≤45 years) and mature CAD. Results: The prevalence of PCAD was 15% (N = 30). PCAD had higher rates of family history of CAD (P < 0.001), tobacco consumption (P = 0.01), smoking (P = 0.004), and single-vessel disease (SVD) (P < 0.001). Premature CAD group had significantly lower mean fasting blood glucose (105 ± 41 mg/dL vs 127 ± 45 mg/dL) levels. Conclusion: Family history of CAD, smoking, and tobacco consumption are important risk factors of PCAD. SVD is more common in PCAD. Screening the susceptible population at risk and controlling the cardiovascular risk factors will help reduce the epidemic and consequences of PCAD.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"69 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-10-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"114682931","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-10-05DOI: 10.1177/26324636221123367
Hendsun Hendsun, Y. Firmansyah, Irene Setiawan
Brugada phenocopies (BrP) are clinical commodities that are etiologically distinguishable from genuine congenital Brugada syndrome. BrP is marked by type-1 or type-2 Brugada electrocardiogram (ECG) patterns in precordial V1 to V3, which is provoked by various underlying clinical disorders. Hemodialysis may improve the BrP ECG image. This case report describes the occurrence of a transient Brugada pattern that was seen on the ECG of an asymptomatic 64-years-old woman after administering hemodialysis due to renal failure. Diagnosis of Brugada phenocopy and BrS is necessary to achieve appropriate further treatment. Hemodialysis may improve the BrP ECG image.
{"title":"Brugada Phenocopy That Lurks After Hemodialysis: A Case Report","authors":"Hendsun Hendsun, Y. Firmansyah, Irene Setiawan","doi":"10.1177/26324636221123367","DOIUrl":"https://doi.org/10.1177/26324636221123367","url":null,"abstract":"Brugada phenocopies (BrP) are clinical commodities that are etiologically distinguishable from genuine congenital Brugada syndrome. BrP is marked by type-1 or type-2 Brugada electrocardiogram (ECG) patterns in precordial V1 to V3, which is provoked by various underlying clinical disorders. Hemodialysis may improve the BrP ECG image. This case report describes the occurrence of a transient Brugada pattern that was seen on the ECG of an asymptomatic 64-years-old woman after administering hemodialysis due to renal failure. Diagnosis of Brugada phenocopy and BrS is necessary to achieve appropriate further treatment. Hemodialysis may improve the BrP ECG image.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"3 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-10-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"130628708","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-10-04DOI: 10.1177/26324636221128453
A. Singhi, S. Mohapatra, E. Bari
Double outlet right ventricle is a very rare association in a patient with tricuspid atresia. The anomaly has been documented in a young boy with illustrative echocardiogram, angiographic images and cardiac computed tomographic imaging. The images describing the rare anomaly would be good to understand the importance of segmental analysis in a complex heart disease.
{"title":"Tricuspid Atresia with Double Outlet Right Ventricle and L Malposed Great Arteries","authors":"A. Singhi, S. Mohapatra, E. Bari","doi":"10.1177/26324636221128453","DOIUrl":"https://doi.org/10.1177/26324636221128453","url":null,"abstract":"Double outlet right ventricle is a very rare association in a patient with tricuspid atresia. The anomaly has been documented in a young boy with illustrative echocardiogram, angiographic images and cardiac computed tomographic imaging. The images describing the rare anomaly would be good to understand the importance of segmental analysis in a complex heart disease.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"80 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-10-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121817891","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-10-03DOI: 10.1177/26324636221123187
S. Umarje, Asawari Raut, P. Dave, N. M. James
Objective To conduct risk assessment and assess the choice of statins with regard to cardiovascular diseases (CVDs). Methodology This is a cross-sectional observation study conducted on 500 patients visiting a teaching hospital. Medical records were used to obtain patient characteristics and type, dose, and regimen of prescribed statin. Prescribed statin dose was evaluated using standard prescribing guidelines by American College of Cardiology/American Heart Association. Result Out of the 500 patients studied, 70% (350) of patients were prescribed statin therapy for a CVD and 30% had at least 1 CVD risk factor. Proportion of males was higher. Mean age of patients with statin therapy for coexisting CVD was 61.16 ± 12.87. Among those with at least 1 cardiovascular risk factor but without CVD, 66% had risk score more than 7.5 out of 10 suggestive of high risk for CVDs within 10 years. Atorvastatin and rosuvastatin were the preferred choice of statin therapy, with higher preference for atorvastatin. Dosing criteria were well-met for secondary prevention; whereas, underdosing was prevalent among users of fixed-dose combinations and among patients prescribed a statin for primary prevention of CVDs. Conclusion Lack of use of risk scores may lead to underdosing and underutilization of statins.
{"title":"Statin Utilization Trend in Primary and Secondary Prevention of Cardiovascular Diseases in a Teaching Hospital","authors":"S. Umarje, Asawari Raut, P. Dave, N. M. James","doi":"10.1177/26324636221123187","DOIUrl":"https://doi.org/10.1177/26324636221123187","url":null,"abstract":"Objective To conduct risk assessment and assess the choice of statins with regard to cardiovascular diseases (CVDs). Methodology This is a cross-sectional observation study conducted on 500 patients visiting a teaching hospital. Medical records were used to obtain patient characteristics and type, dose, and regimen of prescribed statin. Prescribed statin dose was evaluated using standard prescribing guidelines by American College of Cardiology/American Heart Association. Result Out of the 500 patients studied, 70% (350) of patients were prescribed statin therapy for a CVD and 30% had at least 1 CVD risk factor. Proportion of males was higher. Mean age of patients with statin therapy for coexisting CVD was 61.16 ± 12.87. Among those with at least 1 cardiovascular risk factor but without CVD, 66% had risk score more than 7.5 out of 10 suggestive of high risk for CVDs within 10 years. Atorvastatin and rosuvastatin were the preferred choice of statin therapy, with higher preference for atorvastatin. Dosing criteria were well-met for secondary prevention; whereas, underdosing was prevalent among users of fixed-dose combinations and among patients prescribed a statin for primary prevention of CVDs. Conclusion Lack of use of risk scores may lead to underdosing and underutilization of statins.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"516 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-10-03","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"123089713","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-09-30DOI: 10.1177/26324636221123376
J. Gupta, S. Singh
Left ventricular noncompaction cardiomyopathy (LVNC) is a type of primary genetic cardiomyopathy, which occurs during embryogenesis by the arrest in the ventricular myocardium compaction. LVNC is characterized by prominent wall trabeculations and intertrabecular recesses that communicate with the ventricular cavity. There are 2 types of cardiomyopathy: the first one is associated with other primary cardiac structural abnormalities like malfunctional cardiac valves as mentioned in the case report below and the second type is in which there are no other associated cardiac structural abnormalities also called isolated LV noncompaction cardiomyopathy. We report an association of severe rheumatic mitral valve disease and LV noncompaction with significantly reduced ejection fraction which is rare.
{"title":"A Unique Association of Left Ventricular Noncompaction With Rheumatic Heart Disease","authors":"J. Gupta, S. Singh","doi":"10.1177/26324636221123376","DOIUrl":"https://doi.org/10.1177/26324636221123376","url":null,"abstract":"Left ventricular noncompaction cardiomyopathy (LVNC) is a type of primary genetic cardiomyopathy, which occurs during embryogenesis by the arrest in the ventricular myocardium compaction. LVNC is characterized by prominent wall trabeculations and intertrabecular recesses that communicate with the ventricular cavity. There are 2 types of cardiomyopathy: the first one is associated with other primary cardiac structural abnormalities like malfunctional cardiac valves as mentioned in the case report below and the second type is in which there are no other associated cardiac structural abnormalities also called isolated LV noncompaction cardiomyopathy. We report an association of severe rheumatic mitral valve disease and LV noncompaction with significantly reduced ejection fraction which is rare.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"1 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"130823592","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-09-07DOI: 10.1177/26324636221122238
A. Andreou
A 60-year-old female patient, a cigarette smoker with a history of hyperlipidemia presented to the hospital with a 1-h episode of retrosternal chest pain. Physical examination revealed nothing remarkable. Electrocardiography (ECG) showed >0.1 mV ST-segment elevation (STE) in II, aVF, III, and V6, and ST-segment depression (STD) in aVL, I, and V1 to V4 ( Figure 1A ), ≥0.05 mV STE in V5R and V6R ( Figure 1B ), and >0.05 mV STE in V7 to V9 ( Figure 1C ). The patient received a diagnosis of infero-postero-lateral wall STEmyocardial infarction (MI) and was referred for emergency coronary angiography. Which is the culprit artery, based on the ECG findings? Interpretation of the ECG with use of vector concepts reveals an ST-segment vector pointing downward and somewhat rightward between +90° and +120° (STE III > II and STD aVL > I) as well as backward with less STD in V1 to V3 than STE in the inferior leads. Consequently, based on conventional ECG criteria, the right coronary artery (RCA) was most likely the culprit artery. 1-3 Importantly, the ECG also reveals about 0.1 mV STD in aVR, that is a lead facing through the left ventricular cavity, the apex, and lateral wall and is directionally opposite to I, II, V5, and V6, with the latter showing about 0.2 mV STE in this case. Therefore, the ECG indicates extension of the infarction to the apical inferior and apical lateral walls thereby suggesting the presence of a large posterior-lateral left ventricular branch (PLVB). 4 The overall ECG evidence, including an isoelectric ST-segment in V4R and STD in V3 to V4 and STE in V7 to V9, which indicate extension of the infarction to the inferobasal (formerly posterior) wall can be justified by distal occlusion of a dominant RCA supplying a large PLVB. Nonetheless, the inferior-lateral wall is also supplied by the LCx artery, the occlusion of which may also result in STD in aVR. Indeed, the latter ECG sign has been reported to be more common in LCx artery-related than RCA-related MI. 4 Furthermore, in a
60岁女性患者，吸烟，有高脂血症病史，以1小时胸后胸痛就诊。体检没有发现异常。心电图显示II、aVF、III、V6的st段抬高(STE) >0.1 mV, aVL、I、V1至V4的st段降低(STD)(图1A)， V5R、V6R的st段升高≥0.05 mV(图1B)， V7至V9的st段升高>0.05 mV(图1C)。患者被诊断为下后侧壁心肌梗死(MI)，并被转诊进行紧急冠状动脉造影。根据心电图结果，哪条动脉是罪魁祸首?使用矢量概念解释心电图显示，st段矢量在+90°至+120°之间向下且略向右(STE III > II和STD aVL > I)，以及向后，V1至V3的STD少于下导联的STE。因此，根据常规心电图标准，右冠状动脉(RCA)最有可能是罪魁祸首动脉。1-3重要的是，心电图还显示aVR中约0.1 mV STD，这是一个穿过左心室腔，心尖和侧壁的导联，方向与I, II, V5和V6相反，后者在本例中显示约0.2 mV STE。因此，心电图显示梗死延伸至心尖下壁和心尖外壁，从而提示存在较大的左心室后外侧支(PLVB)。4总的心电图证据，包括V4R的等电st段，V3至V4的STD和V7至V9的STE，表明梗死延伸到基底间壁(以前的后壁)，可以通过远端阻断供应大PLVB的优势RCA来证明。尽管如此，下侧壁也由LCx动脉供应，其闭塞也可能导致aVR中的性病。事实上，据报道后一种ECG征象在LCx动脉相关的心肌梗死中比rca相关的心肌梗死更常见
{"title":"Electrocardiographic Prediction of Culprit Artery in Inferior ST-Segment Elevation Myocardial Infarction: Looks can Be Deceiving","authors":"A. Andreou","doi":"10.1177/26324636221122238","DOIUrl":"https://doi.org/10.1177/26324636221122238","url":null,"abstract":"A 60-year-old female patient, a cigarette smoker with a history of hyperlipidemia presented to the hospital with a 1-h episode of retrosternal chest pain. Physical examination revealed nothing remarkable. Electrocardiography (ECG) showed >0.1 mV ST-segment elevation (STE) in II, aVF, III, and V6, and ST-segment depression (STD) in aVL, I, and V1 to V4 ( Figure 1A ), ≥0.05 mV STE in V5R and V6R ( Figure 1B ), and >0.05 mV STE in V7 to V9 ( Figure 1C ). The patient received a diagnosis of infero-postero-lateral wall STEmyocardial infarction (MI) and was referred for emergency coronary angiography. Which is the culprit artery, based on the ECG findings? Interpretation of the ECG with use of vector concepts reveals an ST-segment vector pointing downward and somewhat rightward between +90° and +120° (STE III > II and STD aVL > I) as well as backward with less STD in V1 to V3 than STE in the inferior leads. Consequently, based on conventional ECG criteria, the right coronary artery (RCA) was most likely the culprit artery. 1-3 Importantly, the ECG also reveals about 0.1 mV STD in aVR, that is a lead facing through the left ventricular cavity, the apex, and lateral wall and is directionally opposite to I, II, V5, and V6, with the latter showing about 0.2 mV STE in this case. Therefore, the ECG indicates extension of the infarction to the apical inferior and apical lateral walls thereby suggesting the presence of a large posterior-lateral left ventricular branch (PLVB). 4 The overall ECG evidence, including an isoelectric ST-segment in V4R and STD in V3 to V4 and STE in V7 to V9, which indicate extension of the infarction to the inferobasal (formerly posterior) wall can be justified by distal occlusion of a dominant RCA supplying a large PLVB. Nonetheless, the inferior-lateral wall is also supplied by the LCx artery, the occlusion of which may also result in STD in aVR. Indeed, the latter ECG sign has been reported to be more common in LCx artery-related than RCA-related MI. 4 Furthermore, in a","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"11 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"123814220","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-09-01DOI: 10.1177/26324636221122084
B. Rao
Corresponding author: B Hygriv Rao, KIMS Hospitals, Hyderabad, Telangana 500003, India. E-mail: hygriv@hotmail.com Left ventricular dysfunction is an established marker in heart failure (HF) patients predicting poor clinical outcomes, sudden death, and overall mortality.1 Over the last few decades, various pharmacological agents as guideline directed medical treatment (GDMT) have been introduced serially in the management of HF resulting in incremental benefit in HF hospitalizations, quality of life, symptom alleviation, and mortality. Large data has established the use of beta blockers, angiotensin converting enzyme inhibitors, angiotensin receptor blockers, angiotensin receptor– neprilysin inhibitors, and mineralocorticoid receptor antagonists, in these patients. The ongoing battle against HF was consolidated by amalgamation of sacubitril–valsartan and SGLT-2 inhibitors in the GDMT by data from large trials—PARDIGM HF, DAPA HF, EMPEROR Reduced. 2,3,4 Despite the scintillating advances in pharmacotherapy in this area, the twin clinical problems of worsening HF and renal failure continue to cause abundant frustration in patient management. Patients with a recent HF hospitalization or worsening HF constitute a particularly vulnerable cohort as they are associated with high subsequent event rates and mortality. Moreover HF is frequently associated with impaired renal function and/or high serum potassium concentrations. Kidney is truly the Achilles heel in the management of HF as almost all the routine medications used in these patients require monitoring of renal function and electrolytes.5 Impaired estimated glomerular filtration rate (eGFR), with HF, presents a serious therapeutic challenge as it precludes prescription of all components of GDMT, makes it difficult to up-titrate them to optimal doses, and frequently results in their discontinuation. The most difficult cohort of patients to initiate and maintain GDMT are patients with a lower eGFR, higher N-terminal probrain natriuretic peptide (NTproBNP), and elevated serum potassium concentrations. These are the patients who have a higher risk of cardiovascular death and hospitalizations for HF and in a greater need for these treatments. Accordingly, an unmet need exists for effective therapies in patients with severe heart failure with reduced ejection fraction (HFrEF) and advanced chronic
{"title":"Vericiguat—Filling the Gaps in Heart Failure Management","authors":"B. Rao","doi":"10.1177/26324636221122084","DOIUrl":"https://doi.org/10.1177/26324636221122084","url":null,"abstract":"Corresponding author: B Hygriv Rao, KIMS Hospitals, Hyderabad, Telangana 500003, India. E-mail: hygriv@hotmail.com Left ventricular dysfunction is an established marker in heart failure (HF) patients predicting poor clinical outcomes, sudden death, and overall mortality.1 Over the last few decades, various pharmacological agents as guideline directed medical treatment (GDMT) have been introduced serially in the management of HF resulting in incremental benefit in HF hospitalizations, quality of life, symptom alleviation, and mortality. Large data has established the use of beta blockers, angiotensin converting enzyme inhibitors, angiotensin receptor blockers, angiotensin receptor– neprilysin inhibitors, and mineralocorticoid receptor antagonists, in these patients. The ongoing battle against HF was consolidated by amalgamation of sacubitril–valsartan and SGLT-2 inhibitors in the GDMT by data from large trials—PARDIGM HF, DAPA HF, EMPEROR Reduced. 2,3,4 Despite the scintillating advances in pharmacotherapy in this area, the twin clinical problems of worsening HF and renal failure continue to cause abundant frustration in patient management. Patients with a recent HF hospitalization or worsening HF constitute a particularly vulnerable cohort as they are associated with high subsequent event rates and mortality. Moreover HF is frequently associated with impaired renal function and/or high serum potassium concentrations. Kidney is truly the Achilles heel in the management of HF as almost all the routine medications used in these patients require monitoring of renal function and electrolytes.5 Impaired estimated glomerular filtration rate (eGFR), with HF, presents a serious therapeutic challenge as it precludes prescription of all components of GDMT, makes it difficult to up-titrate them to optimal doses, and frequently results in their discontinuation. The most difficult cohort of patients to initiate and maintain GDMT are patients with a lower eGFR, higher N-terminal probrain natriuretic peptide (NTproBNP), and elevated serum potassium concentrations. These are the patients who have a higher risk of cardiovascular death and hospitalizations for HF and in a greater need for these treatments. Accordingly, an unmet need exists for effective therapies in patients with severe heart failure with reduced ejection fraction (HFrEF) and advanced chronic","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"7 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121144134","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Case of wide QRS tachycardia discussing differentiation of supra ventricular tachycardia with aberrancy from ventricular tachyarrhythmia.
宽QRS型心动过速1例，探讨室性心动过速与室性心动过速异常的鉴别。
{"title":"Wide QRS Tachycardia, What Is the Diagnosis","authors":"Somasekhar Ghanta, Manohar Reddy Paluru, Raghuram Palaparti, Sudarshan Palaparthi","doi":"10.1177/26324636221121459","DOIUrl":"https://doi.org/10.1177/26324636221121459","url":null,"abstract":"Case of wide QRS tachycardia discussing differentiation of supra ventricular tachycardia with aberrancy from ventricular tachyarrhythmia.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"3 6","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"113958130","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2022-08-22DOI: 10.1177/26324636221118422
T. John, Michelle da Silva, H. Weich
Hemoptysis from ruptured Rasmussen’s aneurysms is a not an uncommon source of bleeding (5-10%) in patients with active or previous tuberculosis. Previous reports describe the use of coils, glue, covered stents, and detachable balloons as part of the endovascular management of Rassmusen’s aneurysms. Although vascular plugs have been used in the management of other pulmonary aneurysms and pulmonary arteriovenous malformations, its use within the inflammatory milieu as well as architectural lung parenchymal changes of tuberculosis has not been reported. We report and describe, to our knowledge, the first case of successful management of a Rasmussen’s aneurysm with a vascular plug with good success.
{"title":"Vascular Plugs—Novel Therapeutic Management of Hemoptysis Secondary to Ruptured Rasmussen’s Aneurysm?","authors":"T. John, Michelle da Silva, H. Weich","doi":"10.1177/26324636221118422","DOIUrl":"https://doi.org/10.1177/26324636221118422","url":null,"abstract":"Hemoptysis from ruptured Rasmussen’s aneurysms is a not an uncommon source of bleeding (5-10%) in patients with active or previous tuberculosis. Previous reports describe the use of coils, glue, covered stents, and detachable balloons as part of the endovascular management of Rassmusen’s aneurysms. Although vascular plugs have been used in the management of other pulmonary aneurysms and pulmonary arteriovenous malformations, its use within the inflammatory milieu as well as architectural lung parenchymal changes of tuberculosis has not been reported. We report and describe, to our knowledge, the first case of successful management of a Rasmussen’s aneurysm with a vascular plug with good success.","PeriodicalId":429933,"journal":{"name":"Indian Journal of Clinical Cardiology","volume":"16 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2022-08-22","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"126619723","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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