Annual review of developmental psychology最新文献

Human culture is unique among animals in its complexity, variability, and cumulative quality. This article describes the development and diversity of cumulative cultural learning. Children inhabit cultural ecologies that consist of group-specific knowledge, practices, and technologies that are inherited and modified over generations. The learning processes that enable cultural acquisition and transmission are universal but are sufficiently flexible to accommodate the highly diverse cultural repertoires of human populations. Children learn culture in several complementary ways, including through exploration, observation, participation, imitation, and instruction. These methods of learning vary in frequency and kind within and between populations due to variation in socialization values and practices associated with specific educational institutions, skill sets, and knowledge systems. The processes by which children acquire and transmit the cumulative culture of their communities provide unique insight into the evolution and ontogeny of human cognition and culture.

This article summarizes empirical findings and theoretical concepts in cognitive aging and late-life dementia research. Major emphases are placed on (1) person-to-person heterogeneity in trajectories of cognitive change over time, (2) how trajectories of child cognitive development determine peak levels of adult cognitive function from which aging-related cognitive declines occur, and (3) how lifelong trajectories of cognitive function relate to the timing of severe cognitive impairments characteristic of dementia. I consider conceptual issues surrounding categorical vs. dimensional models of late-life dementia, and how current diagnostic approaches affect inferences in the empirical study of disease progression. The incomplete current understanding of the biological foundations of aging-related cognitive declines and the continuous nature of many biomarkers commonly used in dementia diagnosis and classification together pose both opportunities and challenges in the current research landscape. Research moving forward will benefit from accurately measuring and analyzing continuous variation in longitudinal trajectories of cognitive function.

Social categorization is a universal mechanism for making sense of a vast social world with roots in perceptual, conceptual, and social systems. These systems emerge strikingly early in life and undergo important developmental changes across childhood. The development of social categorization entails identifying which ways of classifying people are culturally meaningful, how these categories might be used to predict, explain, and evaluate the behavior of other people, and how one's own identity relates to these systems of categorization and representation. Social categorization can help children simplify and understand their social environment but has detrimental consequences in the forms of stereotyping, prejudice, and discrimination. Thus, understanding how social categorization develops is a central problem for the cognitive, social, and developmental sciences. This review details the multiple developmental processes that underlie this core psychological capacity.

An extensive literature on childhood adversity and neurodevelopment has emerged over the past decade. We evaluate two conceptual models of adversity and neurodevelopment-the dimensional model of adversity and stress acceleration model-in a systematic review of 109 studies using MRI-based measures of neural structure and function in children and adolescents. Consistent with the dimensional model, children exposed to threat had reduced amygdala, medial prefrontal cortex (mPFC), and hippocampal volume and heightened amygdala activation to threat in a majority of studies; these patterns were not observed consistently in children exposed to deprivation. In contrast, reduced volume and altered function in frontoparietal regions were observed consistently in children exposed to deprivation but not children exposed to threat. Evidence for accelerated development in amygdala-mPFC circuits was limited but emerged in other metrics of neurodevelopment. Progress in charting neurodevelopmental consequences of adversity requires larger samples, longitudinal designs, and more precise assessments of adversity.