Pub Date : 2015-08-10DOI: 10.17554/j.issn.2309-6861.2015.02.80
Testa Marzia, Chauvie Stephane, Biggi Alberto, Terulla Alessandra, F. Mauro
AIM: 123I-Iobenguane myocardial scintigraphy (MIBG), which identifies sympathetic nervous system dysfunction, has been shown to be an effective predictor of sudden cardiac mortality in patient with congestive heart failure (CHF) and could be a useful instrument to early identify adverse clinical events. Methods: patients with CHF, a New York Heart Association classification (NYHA) of II or III and a left ventricular ejection fraction (LVEF) ≤ 35% were eligible for cardiac defibrillator (ICD) placement under current LVEF-based criteria in the guidelines. All patients listed for ICD underwent a MIBG before the implantation, in order to assess the prognostic value of an early cardiac MIBG to predict appropriate ICD indication. Results: 11 patients (100% males; age 65.1±8.9 years) with mean LVEF 29.1±4.9% entered the study. All patients had a coronary artery disease (CAD) treated by coronary artery bypass graft (CABG) in six cases, by a percutaneous coronary angioplasty (PTCA) in seven and both procedures in two patients. In all subjects the heart/mediastinum (H/M) ratio was calculated 35%. An ICD was implanted in six patients (54.5%). The sensitivity, specificity, postive and negative predictive value of an H/M ratio ≤ 1.25 at MIBG was calculated in 75%, 75%; 85,7%, 60%. Conclusion: MIBG might be a promising method for excluding a 3-month left ventricular functional recovery.
{"title":"Role of 123-Iobenguane Myocardial Scintigraphy in Predicting Short Term Left Ventricular Functional Recovery and Indication to ICD Implantation after Coronary Revascularization: A Work Hypothesis","authors":"Testa Marzia, Chauvie Stephane, Biggi Alberto, Terulla Alessandra, F. Mauro","doi":"10.17554/j.issn.2309-6861.2015.02.80","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2015.02.80","url":null,"abstract":"AIM: 123I-Iobenguane myocardial scintigraphy (MIBG), which identifies sympathetic nervous system dysfunction, has been shown to be an effective predictor of sudden cardiac mortality in patient with congestive heart failure (CHF) and could be a useful instrument to early identify adverse clinical events. Methods: patients with CHF, a New York Heart Association classification (NYHA) of II or III and a left ventricular ejection fraction (LVEF) ≤ 35% were eligible for cardiac defibrillator (ICD) placement under current LVEF-based criteria in the guidelines. All patients listed for ICD underwent a MIBG before the implantation, in order to assess the prognostic value of an early cardiac MIBG to predict appropriate ICD indication. Results: 11 patients (100% males; age 65.1±8.9 years) with mean LVEF 29.1±4.9% entered the study. All patients had a coronary artery disease (CAD) treated by coronary artery bypass graft (CABG) in six cases, by a percutaneous coronary angioplasty (PTCA) in seven and both procedures in two patients. In all subjects the heart/mediastinum (H/M) ratio was calculated 35%. An ICD was implanted in six patients (54.5%). The sensitivity, specificity, postive and negative predictive value of an H/M ratio ≤ 1.25 at MIBG was calculated in 75%, 75%; 85,7%, 60%. Conclusion: MIBG might be a promising method for excluding a 3-month left ventricular functional recovery.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"3 1","pages":"371-376"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"75462097","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/J.ISSN.2309-6861.2015.02.77
G. Sloop, J. Weidman, L. Shecterle, J. Cyr
Normal aortic compliance allows cardiac output to be distributed throughout the cardiac cycle, resulting in lower peak blood velocity. Loss of compliance with aging, hypertension, and possibly other risk factors for atherosclerosis increases peak blood velocity, creating eddy currents in areas of changing vascular geometry, as well as creating adverse patterns of diastolic flow. In the resulting areas of low flow, increased blood viscosity creates the potential for thrombosis. Occlusive thrombi may cause death; parietal or mural thrombi may organize to form atherosclerotic plaques.
{"title":"The Interplay of Aging, Aortic Stiffness and Blood Viscosity in Atherogenesis","authors":"G. Sloop, J. Weidman, L. Shecterle, J. Cyr","doi":"10.17554/J.ISSN.2309-6861.2015.02.77","DOIUrl":"https://doi.org/10.17554/J.ISSN.2309-6861.2015.02.77","url":null,"abstract":"Normal aortic compliance allows cardiac output to be distributed throughout the cardiac cycle, resulting in lower peak blood velocity. Loss of compliance with aging, hypertension, and possibly other risk factors for atherosclerosis increases peak blood velocity, creating eddy currents in areas of changing vascular geometry, as well as creating adverse patterns of diastolic flow. In the resulting areas of low flow, increased blood viscosity creates the potential for thrombosis. Occlusive thrombi may cause death; parietal or mural thrombi may organize to form atherosclerotic plaques.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"285 1","pages":"350-354"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"76300471","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/j.issn.2309-6861.2015.02.82
M. Quezada-Feijoó, A. Mangas, Carmen Rodríguez, E. Segura, Carmen Toro-Fernandez, C. Gamero, David Gómez-Villarejo, R. Toro
AIMS: Coronary diseases are the main cause of mortality in the West. Several factors may affect successful percutaneous revascularization, such as the type of stent used or the cardiovascular risk factors associated with the patient. The systolic function of the left ventricle has usually been disregarded in restenosis scores. Our aim is to evaluate restenosis while taking into account the kind of stent used and the systolic function of the left ventricle. METHODS: A prospective, observational and population study. A total of 209 patients with percutaneous revascularization were recruited between March 2011 and January 2013 and monitored every six months. Their clinical data was collected and transthoracic echocardiograms were performed. RESULTS: The average age was 67±11 years old, 64.6% were men. Regarding cardiovascular risk factors, 55.6% were diabetics, 84.2% had hypertension and 81.3% were dyslipemic. A combined revascularization strategy was more often used among the general population than in the diabetic subgroup. The diabetic patient subgroup received a larger number of drug-coated stents. The systolic function was present among 84% of the general population compared to 80% of the diabetic patient subgroup. Both the general population as well as the diabetic patient subgroup presented a significant decreased sistolic function of the left ventricle when the restenosis rate was greater (p<0.001 respectively). Similarly, we observe that there is a significant connection between the ejection fraction and the emergence of a new symptoms. CONCLUSION: The non-combined strategy was used more frequently in the diabetic population with the use of a superior drug-eluting stent Furthermore, systolic function evaluation stratifies the restenosis risk, especially in patients with a depressed ventricular function.
{"title":"Clinical Evaluation of Different Revascularization Strategies: Unlabelled Use","authors":"M. Quezada-Feijoó, A. Mangas, Carmen Rodríguez, E. Segura, Carmen Toro-Fernandez, C. Gamero, David Gómez-Villarejo, R. Toro","doi":"10.17554/j.issn.2309-6861.2015.02.82","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2015.02.82","url":null,"abstract":"AIMS: Coronary diseases are the main cause of mortality in the West. Several factors may affect successful percutaneous revascularization, such as the type of stent used or the cardiovascular risk factors associated with the patient. The systolic function of the left ventricle has usually been disregarded in restenosis scores. Our aim is to evaluate restenosis while taking into account the kind of stent used and the systolic function of the left ventricle. METHODS: A prospective, observational and population study. A total of 209 patients with percutaneous revascularization were recruited between March 2011 and January 2013 and monitored every six months. Their clinical data was collected and transthoracic echocardiograms were performed. RESULTS: The average age was 67±11 years old, 64.6% were men. Regarding cardiovascular risk factors, 55.6% were diabetics, 84.2% had hypertension and 81.3% were dyslipemic. A combined revascularization strategy was more often used among the general population than in the diabetic subgroup. The diabetic patient subgroup received a larger number of drug-coated stents. The systolic function was present among 84% of the general population compared to 80% of the diabetic patient subgroup. Both the general population as well as the diabetic patient subgroup presented a significant decreased sistolic function of the left ventricle when the restenosis rate was greater (p<0.001 respectively). Similarly, we observe that there is a significant connection between the ejection fraction and the emergence of a new symptoms. CONCLUSION: The non-combined strategy was used more frequently in the diabetic population with the use of a superior drug-eluting stent Furthermore, systolic function evaluation stratifies the restenosis risk, especially in patients with a depressed ventricular function.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"17 1","pages":"377-382"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"78502422","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/j.issn.2309-6861.2015.02.76
A. Ogunsua, Amartya Kundu, Amir Y. Shaikh
With an increase in the survival rate and longevity of cancer patients and rapid development of novel anti neoplastic agents, there have been growing concerns about the potential long-term side effects of these drugs. Cardiotoxicity resulting from targeted chemotherapy is a common adverse effect encountered in clinical practice. Traditionally, the cardiac side effects of chemotherapeutic agents have been evaluated primarily by measurement of reduction in systolic function of the heart by assessment of left ventricular ejection fraction (LVEF). Recently, there has been increasing emphasis on earlier and more accurate prediction of cardiotoxicity using imaging modalities besides evaluation of LVEF. Such methods include myocardial strain imaging. We present a review elaborating the role of myocardial strain imaging in chemotherapy related cardiotoxicity.
{"title":"Role of Myocardial Strain Imaging in Chemotherapy Related Cardiotoxicity","authors":"A. Ogunsua, Amartya Kundu, Amir Y. Shaikh","doi":"10.17554/j.issn.2309-6861.2015.02.76","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2015.02.76","url":null,"abstract":"With an increase in the survival rate and longevity of cancer patients and rapid development of novel anti neoplastic agents, there have been growing concerns about the potential long-term side effects of these drugs. Cardiotoxicity resulting from targeted chemotherapy is a common adverse effect encountered in clinical practice. Traditionally, the cardiac side effects of chemotherapeutic agents have been evaluated primarily by measurement of reduction in systolic function of the heart by assessment of left ventricular ejection fraction (LVEF). Recently, there has been increasing emphasis on earlier and more accurate prediction of cardiotoxicity using imaging modalities besides evaluation of LVEF. Such methods include myocardial strain imaging. We present a review elaborating the role of myocardial strain imaging in chemotherapy related cardiotoxicity.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"5 1","pages":"345-349"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"82474744","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/J.ISSN.2309-6861.2015.02.78
A. Berezin
Circulating microparticles (MPs) delivered from the native cell membrane are suitable forms of cell-to-cell communication and transfer of biological active effectors. Circulating endothelial-derived MPs as a marker of endothelial dysfunction may discuss as integrated effectors interrelated target cells, endothelium, and conventional cardiovascualr risk factors. Endothelial-derived MPs play a pivotal role in vascular damage, integrity and tissue reparation. However, the role of MPs as biomarker of cardiovascular outcomes is required scrutinizes. The present review accumulates data regarding the role of circulating endothelial-derived MPs as novel marker of cardiovascular risk.
{"title":"Impaired Phenotype of Circulating Endothelial-Derived Microparticles: Novel Marker of Cardiovascular Risk","authors":"A. Berezin","doi":"10.17554/J.ISSN.2309-6861.2015.02.78","DOIUrl":"https://doi.org/10.17554/J.ISSN.2309-6861.2015.02.78","url":null,"abstract":"Circulating microparticles (MPs) delivered from the native cell membrane are suitable forms of cell-to-cell communication and transfer of biological active effectors. Circulating endothelial-derived MPs as a marker of endothelial dysfunction may discuss as integrated effectors interrelated target cells, endothelium, and conventional cardiovascualr risk factors. Endothelial-derived MPs play a pivotal role in vascular damage, integrity and tissue reparation. However, the role of MPs as biomarker of cardiovascular outcomes is required scrutinizes. The present review accumulates data regarding the role of circulating endothelial-derived MPs as novel marker of cardiovascular risk.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"4565 3 1","pages":"365-370"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"78802538","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/j.issn.2309-6861.2015.02.81
A. Leone
Two cardiovascular structures are usually involved in the harm caused by cigarette smoke: myocardium and endothelium, which are mainly affected by carbon monoxide and nicotine. The harmful effects of smoking induce functional responses that eventually lead to morphological damage. With regard to the myocardium, three main steps have been well documented: myocardial alterations related to the hypoxia caused by increased concentrations in carboxyhemoglobin, reversible degenerative alterations of the cardiac muscle and irreversible myocardial necrosis. A typical experimental pattern due to smoking effects is the smoke cardiomyopathy. Endothelial dysfunction triggers a large number of responses, mainly consisting of blood and inflammatory cell migration and adhesion at the site of altered arterial wall,rupture of the muscular and elastic cells of the arterial wall and lipid infiltrates, which lead to atherosclerosis plaque. In addition, arteriosclerosis is the result of smoking on the resistance arteries. Evidence indicates that morphological alterations of the heart and blood vessels from smoking follow a well-defined way that allow us to tell the story of the cardiovascular alterations.
{"title":"Morphological Alterations of the Heart and Blood Vessels from Tobacco Smoke: the Steps of the Damage","authors":"A. Leone","doi":"10.17554/j.issn.2309-6861.2015.02.81","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2015.02.81","url":null,"abstract":"Two cardiovascular structures are usually involved in the harm caused by cigarette smoke: myocardium and endothelium, which are mainly affected by carbon monoxide and nicotine. The harmful effects of smoking induce functional responses that eventually lead to morphological damage. With regard to the myocardium, three main steps have been well documented: myocardial alterations related to the hypoxia caused by increased concentrations in carboxyhemoglobin, reversible degenerative alterations of the cardiac muscle and irreversible myocardial necrosis. A typical experimental pattern due to smoking effects is the smoke cardiomyopathy. Endothelial dysfunction triggers a large number of responses, mainly consisting of blood and inflammatory cell migration and adhesion at the site of altered arterial wall,rupture of the muscular and elastic cells of the arterial wall and lipid infiltrates, which lead to atherosclerosis plaque. In addition, arteriosclerosis is the result of smoking on the resistance arteries. Evidence indicates that morphological alterations of the heart and blood vessels from smoking follow a well-defined way that allow us to tell the story of the cardiovascular alterations.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"4 1","pages":"355-359"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73100955","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-08-10DOI: 10.17554/j.issn.2309-6861.2015.02.75
Rb Singh, Toru Takahashi, S. Shastun, G. Elkilany, K. Hristova, A. Shehab, E. Onsaard, W. Phomkong, H. Chaves, Mukta Singh, Ratan K Srivastava, Munushi Srivastav, Rg Singh
The objectives of this communication are to make efforts which might ensure that the goals and targets for prevention set are adequate to address the many challenges of global malnutrition. These include both under-nutrition and obesity which are major risk factors of cardiovascular diseases (CVDs); hypertension, coronary artery disease (CAD, stroke and heart failure and other chronic diseases; type 2 diabetes mellitus, cancer, chronic respiratory diseases, bone and joint diseases and neurodegenerative diseases. We also find that this is an opportunity to develop an international consensus on how to approach the prevention of malnutrition, CVDs and other chronic diseases, so that there is no increase in human susceptibility to these diseases.
{"title":"The Concept of Functional Foods and Functional Farming (4 F) in the Prevention of Cardiovascular Diseases: A Review of Goals from 18th World Congress of Clinical Nutrition","authors":"Rb Singh, Toru Takahashi, S. Shastun, G. Elkilany, K. Hristova, A. Shehab, E. Onsaard, W. Phomkong, H. Chaves, Mukta Singh, Ratan K Srivastava, Munushi Srivastav, Rg Singh","doi":"10.17554/j.issn.2309-6861.2015.02.75","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2015.02.75","url":null,"abstract":"The objectives of this communication are to make efforts which might ensure that the goals and targets for prevention set are adequate to address the many challenges of global malnutrition. These include both under-nutrition and obesity which are major risk factors of cardiovascular diseases (CVDs); hypertension, coronary artery disease (CAD, stroke and heart failure and other chronic diseases; type 2 diabetes mellitus, cancer, chronic respiratory diseases, bone and joint diseases and neurodegenerative diseases. We also find that this is an opportunity to develop an international consensus on how to approach the prevention of malnutrition, CVDs and other chronic diseases, so that there is no increase in human susceptibility to these diseases.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"85 1","pages":"341-344"},"PeriodicalIF":0.0,"publicationDate":"2015-08-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77468120","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-06-10DOI: 10.17554/j.issn.2309-6861.2014.02.588
P. Moronval, Lorraine Legeleux, S. Thomann, J. Schved, J. Davy, F. Roubille
Objective: The toxicity profile of new oral anticoagulants is well established and with their increasing use the adverse events are frequently encountered in clinical practice. Here, we describe the haemostatic abnormalities after a massive ingestion of dabigatran. Case summary: A 69-year-old woman was admitted for suicidal attempt after ingestion of 9 grams of dabigatran (60 x 150mg pills), associated with 400 mg of nebivolol, 600 mg of diazepam and 1.5 grams of amitriptyline. The patient did not suffer from any bleeding events. Only few voluntary massive intakes are until now reported. Discussion: There are only few cases reporting such a massive voluntary intake of dabigatran leading to biological disorders contrasting with no clinically significant intake. This surprising absence of bleeding events in our case could be at least partly explained by the poor gastrointestinal intake of the drug, a functional gastroparesis, and a theoretically self-limiting biological effect. Conclusion: As no antidote is currently available in routine use for dabigatran, a systematic collection of cases of dabigatran toxicity provide an insight into the clinical manifestations and potential therapeutic implications.
{"title":"A Massive Intake of Dabigatran: Case-Report and Short Review","authors":"P. Moronval, Lorraine Legeleux, S. Thomann, J. Schved, J. Davy, F. Roubille","doi":"10.17554/j.issn.2309-6861.2014.02.588","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2014.02.588","url":null,"abstract":"Objective: The toxicity profile of new oral anticoagulants is well established and with their increasing use the adverse events are frequently encountered in clinical practice. Here, we describe the haemostatic abnormalities after a massive ingestion of dabigatran. Case summary: A 69-year-old woman was admitted for suicidal attempt after ingestion of 9 grams of dabigatran (60 x 150mg pills), associated with 400 mg of nebivolol, 600 mg of diazepam and 1.5 grams of amitriptyline. The patient did not suffer from any bleeding events. Only few voluntary massive intakes are until now reported. Discussion: There are only few cases reporting such a massive voluntary intake of dabigatran leading to biological disorders contrasting with no clinically significant intake. This surprising absence of bleeding events in our case could be at least partly explained by the poor gastrointestinal intake of the drug, a functional gastroparesis, and a theoretically self-limiting biological effect. Conclusion: As no antidote is currently available in routine use for dabigatran, a systematic collection of cases of dabigatran toxicity provide an insight into the clinical manifestations and potential therapeutic implications.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"81 1","pages":"338-340"},"PeriodicalIF":0.0,"publicationDate":"2015-06-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"86668156","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-06-10DOI: 10.17554/j.issn.2309-6861.2014.02.585
Mariama Akodad, T. Gandet, Y. Lounes, Corentin Curinier, F. Roubille
A 70-year-old patient was admitted in our center for acute coronary syndrome but investigations revealed a giant ascending aortic aneurysm without dissection. The patient was successfully treated with a Bentall procedure, usually challenging in the case of giant aneurysm, because of the risk of preoperative injury.
{"title":"Giant Ascending Aortic Aneurysm Mimicking Acute Coronary Syndrome","authors":"Mariama Akodad, T. Gandet, Y. Lounes, Corentin Curinier, F. Roubille","doi":"10.17554/j.issn.2309-6861.2014.02.585","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2014.02.585","url":null,"abstract":"A 70-year-old patient was admitted in our center for acute coronary syndrome but investigations revealed a giant ascending aortic aneurysm without dissection. The patient was successfully treated with a Bentall procedure, usually challenging in the case of giant aneurysm, because of the risk of preoperative injury.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"66 1","pages":"334-335"},"PeriodicalIF":0.0,"publicationDate":"2015-06-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"79536182","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2015-06-10DOI: 10.17554/j.issn.2309-6861.2014.02.586
Mariama Akodad, M. Berous, B. Lattuca, Sébastien Jugant, F. Roubille
We report a case of a young woman with systemic lupus induced cardiac tamponade. After discussion with the cardiac surgeons’ team, medical management was decided and no pericardiocentesis was performed. Bolus of Methylprednisolon intravenously were administrated with a rapid improvement of the symptom and resolution of the pericardial effusion.
{"title":"Systemic Lupus Erythematosus Presenting as Cardiac Tamponade Successfully Treated With High-Dose Steroid: A Case Report","authors":"Mariama Akodad, M. Berous, B. Lattuca, Sébastien Jugant, F. Roubille","doi":"10.17554/j.issn.2309-6861.2014.02.586","DOIUrl":"https://doi.org/10.17554/j.issn.2309-6861.2014.02.586","url":null,"abstract":"We report a case of a young woman with systemic lupus induced cardiac tamponade. After discussion with the cardiac surgeons’ team, medical management was decided and no pericardiocentesis was performed. Bolus of Methylprednisolon intravenously were administrated with a rapid improvement of the symptom and resolution of the pericardial effusion.","PeriodicalId":92802,"journal":{"name":"Journal of clinical cardiology and cardiovascular therapy","volume":"28 1","pages":"336-337"},"PeriodicalIF":0.0,"publicationDate":"2015-06-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"89157928","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}