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Homocysteine and immune injury 同型半胱氨酸与免疫损伤
Clinical Immunology Newsletter
Pub Date : 1999-08-01 DOI: 10.1016/S0197-1859(00)88209-6
Norm granholm (Assistant Professor)
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引用次数: 1
Phagocytic recognition of apoptotic leukocytes 凋亡白细胞的吞噬细胞识别
Clinical Immunology Newsletter
Pub Date : 1999-08-01 DOI: 10.1016/S0197-1859(00)88208-4
Krista L. Hess, Joan M. Cook-Mills
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引用次数: 0
Immune reconstitution after combination antiretroviral therapy 抗逆转录病毒联合治疗后的免疫重建
Clinical Immunology Newsletter
Pub Date : 1999-08-01 DOI: 10.1016/S0197-1859(00)88206-0
Hernan Valdez
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引用次数: 0
Flow cytometry based procedures for the measurement of apoptosis applicable to the investigation of HIV infection 基于流式细胞术的细胞凋亡测定方法适用于HIV感染的研究
Clinical Immunology Newsletter
Pub Date : 1999-08-01 DOI: 10.1016/S0197-1859(00)88207-2
Thomas W. Mc Closkey , Savita Pahwa
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引用次数: 0
Fibrin(ogen) and inflammation: Current understanding and new perspectives 纤维蛋白(原)和炎症:当前的认识和新的观点
Clinical Immunology Newsletter
Pub Date : 1999-08-01 DOI: 10.1016/S0197-1859(00)88210-2
Ashley P. DeAnglis, Gregory S. Retzinger
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引用次数: 8
Human herpesvirus 6 (hhv-6): Could it play a role in the etiopathogenesis of multiple sclerosis (ms)? 人类疱疹病毒6 (hhv-6):它是否在多发性硬化症(ms)的发病机制中起作用?
Clinical Immunology Newsletter
Pub Date : 1999-06-01 DOI: 10.1016/S0197-1859(00)89203-1
MD L.F. Kastrukoff , MD, PhD E.E. Thomas
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引用次数: 0
Abstracts from the twelfth annual meeting of the association of medical laboratory immunologists (amli) 医学实验室免疫学家协会(amli)第十二届年会摘要
Clinical Immunology Newsletter
Pub Date : 1999-06-01 DOI: 10.1016/S0197-1859(00)89204-3
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引用次数: 0
The human thymus: A new perspective on thymic function, aging, and hiv infection 人类胸腺:胸腺功能、衰老和hiv感染的新视角
Clinical Immunology Newsletter
Pub Date : 1999-06-01 DOI: 10.1016/S0197-1859(00)89202-X
Carolyn M. Steffens , Giulia Marchetti , Alan Landay , Lena Al-Harthi

Shortly after birth, the human thymus begins a life long process of involution, whereby the net size of the thymus is not altered but the organ is replaced by adipose tissue. As a result, it has long been believed that thymic involution is indicative of a nonfunctional organ. Recently, however, with the use of computed tomography analysis and innovative molecular approaches that measure T-cell receptor circles, indicative of recent thymic emigrants, doubt has been placed on that dogma. The thymus appears to be active in thymopoiesis throughout the adult life, albeit inversely correlated with age. Being faced with diseases that deplete T-cells such as the acquired immunodeficiency syndrome (AIDS), this recent finding has the potential to exploit novel approaches that enhance thymic output as a mechanism to reconstitute the immune system. In this review, we will revisit the role of T-cells in immunity, the relationship between thymic function and age, and closely examine the impact of HIV-mediated thymic dysregulation on thymopoiesis.

出生后不久，人的胸腺开始了一个漫长的衰老过程，胸腺的净大小没有改变，但器官被脂肪组织所取代。因此，长期以来人们一直认为胸腺退化是一个无功能器官的标志。然而，最近，使用计算机断层扫描分析和创新的分子方法来测量t细胞受体圈，表明最近的胸腺迁移，对这一教条产生了怀疑。尽管胸腺与年龄呈负相关，但在整个成年生活中，胸腺似乎都在积极地进行胸腺生成。面对诸如获得性免疫缺陷综合征(AIDS)等消耗t细胞的疾病，这一最近的发现有可能开发出新的方法，以增强胸腺输出作为重建免疫系统的机制。在这篇综述中，我们将重新审视t细胞在免疫中的作用，胸腺功能与年龄的关系，并仔细研究hiv介导的胸腺功能失调对胸腺生成的影响。
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引用次数: 7
Immune reconstitution in lymphoid tissue following potent antiretroviral therapy 强效抗逆转录病毒治疗后淋巴组织的免疫重建
Clinical Immunology Newsletter
Pub Date : 1999-06-01 DOI: 10.1016/S0197-1859(00)89201-8
MD, PhD Jan Andersson, PhD Thomas E. Fehniger

During untreated HIV-1 infection, a chronic state of immune activation and inflammation develops at the lymphoid tissue sites of viral replication. The early effect of potent combination drug therapy is a reduction in peripheral viral burden and a reduction in the production of inflammatory and type 1 cytokines. Further along in treatment there are trends toward normalization in the frequencies of CD88 T-cells, CD4+ CD45RA+ cells, as well as CD4+ CD45R0+ cells. Finally, the CD1a+ dendritic cell network is re-established and germinal centers are reformed. Although this restoration of the lymphoid dynamic form is coupled to a reconstitution of peripheral blood T-cell function in vitro and by skin testing, sterilizing immunity to HIV-1 does not develop. Furthermore there is no heightened development of cytotoxic CD8+ T-cell function at the site of HIV-1 latency. This is evidenced by a massive recrudescence of HIV-1 viral replication within lymphoid tissue when therapy is stopped. The development of supplemental therapies, which reconstitute anti-HIV-1 immunity, will be required. Specific defects in anti-HIV-1 activity which occur in lymphoid tissue during infection include a downregulation of perform expression by cytotoxic T-cells, the down regulation of the TCR signal transducing chain CD3ζ, and inadequate CD4+ T-cell help within the tissue compartment of immune regeneration.

在未经治疗的HIV-1感染期间，在病毒复制的淋巴组织部位出现免疫激活和炎症的慢性状态。强效联合药物治疗的早期效果是减少外周病毒负荷，减少炎症和1型细胞因子的产生。随着治疗的深入，CD88 t细胞、CD4+ CD45RA+细胞以及CD4+ CD45R0+细胞的频率有趋于正常化的趋势。最后，CD1a+树突状细胞网络被重新建立，生发中心被改造。尽管这种淋巴细胞动态形态的恢复与体外和皮肤试验中外周血t细胞功能的重建相结合，但对HIV-1的绝育免疫并未发展。此外，在HIV-1潜伏的部位，细胞毒性CD8+ t细胞功能没有增强。当治疗停止时，淋巴组织内HIV-1病毒复制的大量复发证明了这一点。需要开发补充疗法，以重建抗hiv -1免疫。感染期间淋巴组织中出现的抗hiv -1活性的特异性缺陷包括细胞毒性t细胞表达的下调，TCR信号转导链CD3ζ的下调，以及免疫再生组织室中CD4+ t细胞帮助不足。
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引用次数: 1
Immunobiology of reproduction: Role of uniquely abundant NK cells in the placenta 生殖的免疫生物学:独特丰富的NK细胞在胎盘中的作用
Clinical Immunology Newsletter
Pub Date : 1999-04-01 DOI: 10.1016/S0197-1859(00)89200-6
Daniel Rukavina, Gordana Laskarin, Natasa Strbo, Vlatka Sotosek, Tatjana Bogovic
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引用次数: 0
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