Revue Veterinaire Clinique最新文献

{"title":"Un cas d’obstruction duodénale lithiasique","authors":"E. Quentin, A. Franquart, C. Bibaut, C. Tilmant, M. Garcia","doi":"10.1016/j.anicom.2025.06.004","DOIUrl":"10.1016/j.anicom.2025.06.004","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 170-171"},"PeriodicalIF":0.0,"publicationDate":"2025-06-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144680036","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Stabilisation d’une instabilité atlanto-axiale associée à une dysplasie atlanto-occipitale par plaque dorsale imprimée en 3D : 3 chiens","authors":"M. Guichard, F. Sager","doi":"10.1016/j.anicom.2025.06.005","DOIUrl":"10.1016/j.anicom.2025.06.005","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 172-174"},"PeriodicalIF":0.0,"publicationDate":"2025-06-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144680037","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Rôle du microbiote intestinal dans la pathogénie de la maladie lithiasique chez le chat","authors":"P. Joubran, F. Roux, J.-Y. Deschamps","doi":"10.1016/j.anicom.2025.06.008","DOIUrl":"10.1016/j.anicom.2025.06.008","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 180-181"},"PeriodicalIF":0.0,"publicationDate":"2025-06-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144679485","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Cytological and histopathological lesions consistent with cutaneous nasal cryptococcosis in a guinea pig (Cavia porcellus)","authors":"M.A. Moraes ,&nbsp;R.S. de Moraes ,&nbsp;F.B. Carvalho de Moura ,&nbsp;L.G.D. Benevenuto ,&nbsp;T.P. Onuma ,&nbsp;N.C. Leite ,&nbsp;D.Q. Cagnini","doi":"10.1016/j.anicom.2025.05.001","DOIUrl":"10.1016/j.anicom.2025.05.001","url":null,"abstract":"<div><div>The growing trend of having non-conventional animals as pets highlights the importance of veterinarians being familiar with the diseases associated with these species. Cryptococcosis is an opportunistic disease with a zoonotic potential caused by fungi of the genus <em>Cryptococcus</em>. It affects many species, including canids, felines, humans, and rodents such as <em>Cavia porcellus</em>, popularly known in Brazil as guinea pig. The present case report highlights macro- and microscopic findings through cytology and histopathology, revealing pyogranulomatous inflammatory process on the skin of the nasal planum of a guinea pig (<em>Cavia porcellus</em>) associated with the presence of yeast-like structures morphologically compatible with <em>Cryptococcus</em> spp. The etiological diagnosis is useful for preventive measures toward owners since guinea pigs can be a source of infection for humans.</div></div><div><div>La popularité croissante des animaux non conventionnels en tant qu’animaux de compagnie met en évidence l’importance pour les vétérinaires de connaître les maladies associées à ces espèces. La cryptococcose est une maladie opportuniste et potentiellement zoonotique provoquée par des champignons du genre <em>Cryptococcus</em>. Elle affecte de nombreuses espèces : les canidés, les félidés, les humains et les rongeurs, comme <em>Cavia porcellus</em>, couramment appelé cochon d’Inde au Brésil. La description de ce cas met en évidence les résultats macro- et microscopiques de la cytologie et de l’histopathologie, révélant un processus inflammatoire pyogranulomateux sur la peau du <em>planum</em> nasal d’un cochon d’Inde (<em>Cavia porcellus)</em> associé à la présence de structures levuriformes morphologiquement compatibles avec <em>Cryptococcus</em> spp. Le diagnostic étiologique est essentiel pour la mise en place de mesures péventives afin de protéger les propriétaires car les cochons d’Inde peuvent être une source d’infection pour les humains.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 136-139"},"PeriodicalIF":0.0,"publicationDate":"2025-05-29","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144680028","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Persistance du canal artériel chez un chien Coton de Tuléar de 3 ans et demi","authors":"A. Herbreteau ,&nbsp;P. Maitre","doi":"10.1016/j.anicom.2025.04.001","DOIUrl":"10.1016/j.anicom.2025.04.001","url":null,"abstract":"<div><div>La persistance du canal artériel est la malformation cardiaque congénitale la plus fréquente chez le chien. Les chiens atteints peuvent présenter des signes cliniques généraux peu spécifiques (retard de croissance, intolérance à l’effort). Les anomalies retrouvées à l’examen clinique sont plus spécifiques, comme la présence d’un <em>thrill</em> et d’un souffle systolo-diastolique basal gauche. Les examens complémentaires tiennent une place importante dans la prise en charge. Si la radiographie thoracique permet d’étayer l’hypothèse de persistance du canal artériel et d’en apprécier les conséquences pulmonaires, le diagnostic de certitude repose sur la réalisation d’une échocardiographie, permettant d’objectiver la présence du canal, sa forme et sa topographie, ainsi que flux sanguin anormal qui le traverse et ses potentielles conséquences cardiaques. Le traitement de la persistance du canal artériel repose sur l’occlusion du canal par voie chirurgicale et doit être réalisé le plus tôt possible, les chiens malades ayant une médiane de survie inférieure à 2 ans. Ce cas clinique présente la démarche diagnostique, la prise en charge chirurgicale et la gestion des complications peropératoires d’un cas de persistance du canal artériel chez un chien Coton de Tuléar de trois ans et demi avec une expression clinique tardive.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 130-135"},"PeriodicalIF":0.0,"publicationDate":"2025-05-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144679995","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"","authors":"G. Pageot","doi":"10.1016/j.anicom.2025.03.003","DOIUrl":"10.1016/j.anicom.2025.03.003","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 104-105"},"PeriodicalIF":0.0,"publicationDate":"2025-03-26","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881339","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Pathologie comparée : une nouvelle rubrique","authors":"","doi":"10.1016/j.anicom.2025.03.001","DOIUrl":"10.1016/j.anicom.2025.03.001","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Page 63"},"PeriodicalIF":0.0,"publicationDate":"2025-03-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143881325","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Un cas d’encéphalite limbique féline","authors":"A. Forestier ,&nbsp;M. Guehl ,&nbsp;E. Lyon ,&nbsp;E. Reyes-Gomez ,&nbsp;S. Besnard ,&nbsp;C. Benzimra ,&nbsp;C. Escriou ,&nbsp;S. Blot","doi":"10.1016/j.anicom.2025.03.002","DOIUrl":"10.1016/j.anicom.2025.03.002","url":null,"abstract":"<div><div>Un chat mâle castré de 4,5 ans a été présenté pour des manifestations récurrentes évoquant des crises épileptiques focales associées à une modification du comportement. L’examen IRM de l’encéphale, l’analyse du liquide cérébrospinal, et l’examen électroencéphalographique étaient évocateurs d’une encéphalite limbique, sans pour autant permettre de l’affirmer avec certitude. L’encéphalite limbique a pu être confirmer avec certitude grâce à l’examen anatomopathologique post-mortem couplé au dosage sérique spécifique des anticorps anti-LGI1. L’encéphalite limbique féline à anticorps anti-LGI1 (EL-LGI1) est une forme d’encéphalite auto-immune, récemment reconnue chez les chats, possiblement causée par des anticorps ciblant la protéine LGI1. Similaire à sa forme humaine, l’EL-LGI1 chez les chats se manifeste par des crises épileptiques focales, souvent accompagnées d’automatismes orofaciaux et de changement de comportement comme l’agressivité. Le diagnostic repose sur la détection des anticorps anti-LGI1, bien que ce dosage ne soit pas disponible en routine en médecine vétérinaire et particulièrement en France. Ce cas illustre un cas typique d’encéphalite limbique pour lequel un diagnostic a été établi avec certitude en post-mortem et souligne la difficulté du diagnostic pré-mortem.</div></div><div><div>A 4.5-year-old neutered male cat was presented with recurrent manifestations suggesting focal seizures associated with behavioral changes. MRI of the brain, analysis of cerebrospinal fluid, and electroencephalographic examination were suggestive of limbic encephalitis, although they could not confirm it with certainty. Limbic encephalitis was definitively confirmed through post-mortem histopathological examination combined with specific serum testing for anti-LGI1 antibodies. Feline limbic encephalitis with anti-LGI1 (EL-LGI1) is a form of autoimmune encephalitis recently recognized in cats, possibly caused by antibodies targeting the LGI1 protein. Similar to its human form, EL-LGI1 in cats manifests as focal seizures, often accompanied by oro-facial automatisms and behavioral changes such as aggression. The diagnosis relies on the detection of anti-LGI1 antibodies, although this test is not routinely available in veterinary medicine, especially in France. This case illustrates a typical example of limbic encephalitis for which a definitive diagnosis was established post-mortem and highlights the challenges of pre-mortem diagnosis.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 121-129"},"PeriodicalIF":0.0,"publicationDate":"2025-03-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144679983","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"Atrophies progressives de la rétine du chien, rétinopathies pigmentaires de l’homme : similitudes et différences","authors":"G. Chaudieu","doi":"10.1016/j.anicom.2025.02.002","DOIUrl":"10.1016/j.anicom.2025.02.002","url":null,"abstract":"<div><div>Les rétinopathies pigmentaires de l’homme et les atrophies progressives de la rétine du chien, dont les classifications sont initialement rappelées, sont homologues à beaucoup d’égards. Leur phénotype est dominé par une perte progressive de vision avec une cécité terminale très fréquente. Elles présentent une triple hétérogénéité : clinique, génétique et moléculaire. Les données cliniques sont rappelées, comparativement dans les deux espèces, pour les formes isolées et les formes syndromiques. Leur transmission mendélienne est majoritairement autosomique récessive, surtout chez le chien où ce mode est quasi-exclusif. Des gènes communs sont impliqués, leurs mutations sont plus nombreuses chez l’homme, chez qui le pourcentage de cas sporadiques est élevé, alors que le suivi d’une lignée affectée offre une facilité d’étude propre à la structuration génétique de l’espèce canine en races sélectionnées. Ces facteurs contribuent à faire du chien un modèle privilégié transposable à l’homme dans la recherche en thérapie génique rétinienne.</div></div><div><div>Human pigmentary retinopathies and canine progressive retinal atrophies, whose classifications are initially recalled, are homologous in many aspects. Their phenotype is dominated by a progressive vision loss with very frequent terminal blindness. They present a triple heterogeneity: clinical, genetic and molecular. Clinical data are reviewed comparatively in these two species for isolated and syndromic forms. Their mendelian transmission is mostly autosomal recessive, especially in dogs, where this mode is almost exclusive. Common genes are involved, their mutations are most numerous in humans, in whom the percentage of sporadic cases is high, while an affected breeding line following offers an ease of study specific to the genetic structuring of the canine species into selected breeds. These factors contribute to making the dog a privileged model that can be transposed to humans in retina gene therapy research.</div></div>","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 2","pages":"Pages 64-82"},"PeriodicalIF":0.0,"publicationDate":"2025-03-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143882050","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}

{"title":"","authors":"B. Cloet-Chabre","doi":"10.1016/j.anicom.2025.02.001","DOIUrl":"10.1016/j.anicom.2025.02.001","url":null,"abstract":"","PeriodicalId":38077,"journal":{"name":"Revue Veterinaire Clinique","volume":"60 3","pages":"Pages 163-164"},"PeriodicalIF":0.0,"publicationDate":"2025-03-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"144680033","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}