Pub Date : 2019-01-16DOI: 10.19080/jocct.2019.12.555848
María de la Paz Scribano Parada
Introduction: Relationship between Cardiovascular Risk Factors (CVRF) and Neurocognitive Disorders (ND) is important, since the first are modifiable. It was studied the influence of cardiovascular risk factors in the neurocognitive profile. Patients and Methods: Transversal analytic prospective study. Patients attended by cognitive symptoms were included. CVRF were recorded. According to Montreal Cognitive Assesment (MoCA) (cutoff of 26 points) two groups were made: Without cognitive disorder (NoCD) and cognitive disorder (CD). Cognitive Domains (CD) were evaluated. Risk groups were established (No CVRF, 1 CVRF, 2 CVRF, 3 or more CVRF). The relation between risk groups with invidual cd and with CD and noCD groups, was established. Chi Square for attributes. ANOVA and Kruskal-Wallis for differences between groups, significance level p<0.05. Results: 66 patients (female 57/86%). Age 63.7±16 years. Most prevalent CVRF: physical inactivity, hypertension, smoking. MoCA average of 20.91±5.78. The CD group (n=48) received lower score in MoCA and worst performers in every cd. Groups 2 and 3CVRF able worst performance, and visuospatial/executive function were the most affected. Conclusion: People over 65 years with increased cardiovascular risk, have poorer cognitive performance. The factors with the greatest implication would be female, age, years of schooling and summation of cardiovascular risk factors. The cd most affected are visuospatial and attention.
{"title":"Relationship Between Neurocognitive Profile and Cardiovascular Risk Factors","authors":"María de la Paz Scribano Parada","doi":"10.19080/jocct.2019.12.555848","DOIUrl":"https://doi.org/10.19080/jocct.2019.12.555848","url":null,"abstract":"Introduction: Relationship between Cardiovascular Risk Factors (CVRF) and Neurocognitive Disorders (ND) is important, since the first are modifiable. It was studied the influence of cardiovascular risk factors in the neurocognitive profile. Patients and Methods: Transversal analytic prospective study. Patients attended by cognitive symptoms were included. CVRF were recorded. According to Montreal Cognitive Assesment (MoCA) (cutoff of 26 points) two groups were made: Without cognitive disorder (NoCD) and cognitive disorder (CD). Cognitive Domains (CD) were evaluated. Risk groups were established (No CVRF, 1 CVRF, 2 CVRF, 3 or more CVRF). The relation between risk groups with invidual cd and with CD and noCD groups, was established. Chi Square for attributes. ANOVA and Kruskal-Wallis for differences between groups, significance level p<0.05. Results: 66 patients (female 57/86%). Age 63.7±16 years. Most prevalent CVRF: physical inactivity, hypertension, smoking. MoCA average of 20.91±5.78. The CD group (n=48) received lower score in MoCA and worst performers in every cd. Groups 2 and 3CVRF able worst performance, and visuospatial/executive function were the most affected. Conclusion: People over 65 years with increased cardiovascular risk, have poorer cognitive performance. The factors with the greatest implication would be female, age, years of schooling and summation of cardiovascular risk factors. The cd most affected are visuospatial and attention.","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2019-01-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"48423405","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-12-14DOI: 10.19080/JOCCT.2018.12.555847
Stewart D Rose
This prompted research on using a vegetarian diet as a treatment for coronary artery disease. For over 45 years, evidence from interventional studies has strongly indicated that a low-fat plant-based diet is both safe and efficacious in the treatment of coronary artery disease (CAD). It’s particularly effective in the treatment angina pectoris. Interventional studies have shown that a low-fat plant-based diet is a safe and efficacious alternative to other treatments. This treatment can be used alone or in combination with standard treatment regimens, including medication, stenting and CABG. Treatment with the plant-based diet has the distinct advantages of having no adverse reactions or contraindications, is affordable, effectively treats common comorbidities and has been shown to have a high patient compliance.
{"title":"A Comprehensive Review of the Prevention and Treatment of Heart Disease with a Plant-Based Diet","authors":"Stewart D Rose","doi":"10.19080/JOCCT.2018.12.555847","DOIUrl":"https://doi.org/10.19080/JOCCT.2018.12.555847","url":null,"abstract":"This prompted research on using a vegetarian diet as a treatment for coronary artery disease. For over 45 years, evidence from interventional studies has strongly indicated that a low-fat plant-based diet is both safe and efficacious in the treatment of coronary artery disease (CAD). It’s particularly effective in the treatment angina pectoris. Interventional studies have shown that a low-fat plant-based diet is a safe and efficacious alternative to other treatments. This treatment can be used alone or in combination with standard treatment regimens, including medication, stenting and CABG. Treatment with the plant-based diet has the distinct advantages of having no adverse reactions or contraindications, is affordable, effectively treats common comorbidities and has been shown to have a high patient compliance.","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-12-14","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43474934","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-12-12DOI: 10.19080/jocct.2018.12.555846
S. Mukherjee
At the most severe end of the spectrum of acute coronary syndromes is ST-Segment Elevation Myocardial Infarction (STEMI), which usually occurs when a fibrin-rich thrombus completely occludes an epicardial coronary artery. The diagnosis of STEMI is based on clinical characteristics and persistent ST-segment elevation as demonstrated by 12-lead electrocardiography. Patients with STEMI should undergo rapid assessment for reperfusion therapy, and a reperfusion strategy should be implemented promptly after the patient’s contact with the health care system. Two methods are currently available for establishing timely coronary reperfusion: primary percutaneous coronary intervention and fibrinolytic therapy [1]. Percutaneous coronary intervention is the preferred method but is not always available. ST-segment elevation myocardial infarction (STEMI) is characterized by total occlusion of the infarct-related artery in contrast to Unstable Angina or Non-ST elevate d Myocardial Infarction (UA/NSTEMI) [2]. Evidence from several randomized clinical trials during the past two decades has established the importance of the open artery theory, which states that prompt and complete restoration of flow in the occluded artery decreases infarct size, preserves Left Ventricular (LV) function, and improves survival rates. The role of Percutaneous Coronary Interventions (PCIs) in the early hours of an STEMI can be divided into primary PCI, Pharmacoinvasive PCI, and Delayed PCI [3].
{"title":"Evaluation and Outcome of Patients of Stemi with Acute Total Occlution of Coronary Artery in The Setting of Primary PCI, Pharmaco Invasive PCI and Delayed PCI","authors":"S. Mukherjee","doi":"10.19080/jocct.2018.12.555846","DOIUrl":"https://doi.org/10.19080/jocct.2018.12.555846","url":null,"abstract":"At the most severe end of the spectrum of acute coronary syndromes is ST-Segment Elevation Myocardial Infarction (STEMI), which usually occurs when a fibrin-rich thrombus completely occludes an epicardial coronary artery. The diagnosis of STEMI is based on clinical characteristics and persistent ST-segment elevation as demonstrated by 12-lead electrocardiography. Patients with STEMI should undergo rapid assessment for reperfusion therapy, and a reperfusion strategy should be implemented promptly after the patient’s contact with the health care system. Two methods are currently available for establishing timely coronary reperfusion: primary percutaneous coronary intervention and fibrinolytic therapy [1]. Percutaneous coronary intervention is the preferred method but is not always available. ST-segment elevation myocardial infarction (STEMI) is characterized by total occlusion of the infarct-related artery in contrast to Unstable Angina or Non-ST elevate d Myocardial Infarction (UA/NSTEMI) [2]. Evidence from several randomized clinical trials during the past two decades has established the importance of the open artery theory, which states that prompt and complete restoration of flow in the occluded artery decreases infarct size, preserves Left Ventricular (LV) function, and improves survival rates. The role of Percutaneous Coronary Interventions (PCIs) in the early hours of an STEMI can be divided into primary PCI, Pharmacoinvasive PCI, and Delayed PCI [3].","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-12-12","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45069440","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-12-10DOI: 10.19080/JOCCT.2018.12.555845
J. Mingou
{"title":"Non-Valvular Atrial Fibrillation: Diagnostic and Therapeutic Aspects About 45 Cases in a Cardiology Department of Sikasso Hospital in Mali","authors":"J. Mingou","doi":"10.19080/JOCCT.2018.12.555845","DOIUrl":"https://doi.org/10.19080/JOCCT.2018.12.555845","url":null,"abstract":"","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-12-10","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43980173","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-11-28DOI: 10.19080/JOCCT.2018.12.555842
Z. Saad
Chest pain is one of the most common symptoms in patients presented to Emergency Department (ED). Various differential diagnoses have to be considered, some of them are potentially lifethreatening. Some of these patients were having high risk factors of coronary artery diseases, and are presented with typical chest pain, along with Electrocardiogram (ECG) changes suggestive Acute Coronary Syndrome (ACS). However, many others are present with a typical chest pain with some ECG changes which cannot confirm or exclude myocardial ischemia. Some other diseases can present with clinical picture which can mimic Ischemic Heart Disease (IHD) such as Pulmonary Embolism (PE) aortic dissection [1-3] as well as Pulmonary, Pleural or osseous lesions, that must be taken into account [4]. The diagnosis of the cause of chest pain is a true challenge for ED physicians. The uncertainty of the diagnosis of these cases results in the practice of defensive medicine and consequent un-necessary admissions which costs too much [5-8].
{"title":"“Triple-Rule-Out” in Investigating Cases of Acute Chest Pain (Saudi Experience)","authors":"Z. Saad","doi":"10.19080/JOCCT.2018.12.555842","DOIUrl":"https://doi.org/10.19080/JOCCT.2018.12.555842","url":null,"abstract":"Chest pain is one of the most common symptoms in patients presented to Emergency Department (ED). Various differential diagnoses have to be considered, some of them are potentially lifethreatening. Some of these patients were having high risk factors of coronary artery diseases, and are presented with typical chest pain, along with Electrocardiogram (ECG) changes suggestive Acute Coronary Syndrome (ACS). However, many others are present with a typical chest pain with some ECG changes which cannot confirm or exclude myocardial ischemia. Some other diseases can present with clinical picture which can mimic Ischemic Heart Disease (IHD) such as Pulmonary Embolism (PE) aortic dissection [1-3] as well as Pulmonary, Pleural or osseous lesions, that must be taken into account [4]. The diagnosis of the cause of chest pain is a true challenge for ED physicians. The uncertainty of the diagnosis of these cases results in the practice of defensive medicine and consequent un-necessary admissions which costs too much [5-8].","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-11-28","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45886634","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-10-31DOI: 10.19080/JOCCT.2018.12.555841
S. Mukherjee
{"title":"Correlation between severity of Coronary Artery Disease and severity of Peripheral Artery Disease, a detail analytical study from Eastern India","authors":"S. Mukherjee","doi":"10.19080/JOCCT.2018.12.555841","DOIUrl":"https://doi.org/10.19080/JOCCT.2018.12.555841","url":null,"abstract":"","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-10-31","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"41525375","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-10-09DOI: 10.19080/JOCCT.2018.12.555839
M. Noble
If one accepts that arterial glycocalyx dysfunction is the first step in the atherothrombotic process, screening of asymptomatic subjects could detect this change. Measurement of the arterial dilatation in response to increased arterial flow (FMD) is widely used in research and would seem to be a possible general non-invasive screening test in humans. Once there has been a break-down of this first line of protection, the second step in the atherothrombotic process might be effectively detected by measurement of blood platelet-platelet and platelet-leukocyte aggregates. While there is no clear treatment to counteract the first step other than exercise programmes, there is evidence to suggest that effective treatment of the second step could be achieved using 5HT2A antagonism.
{"title":"Protection of the Vascular Wall","authors":"M. Noble","doi":"10.19080/JOCCT.2018.12.555839","DOIUrl":"https://doi.org/10.19080/JOCCT.2018.12.555839","url":null,"abstract":"If one accepts that arterial glycocalyx dysfunction is the first step in the atherothrombotic process, screening of asymptomatic subjects could detect this change. Measurement of the arterial dilatation in response to increased arterial flow (FMD) is widely used in research and would seem to be a possible general non-invasive screening test in humans. Once there has been a break-down of this first line of protection, the second step in the atherothrombotic process might be effectively detected by measurement of blood platelet-platelet and platelet-leukocyte aggregates. While there is no clear treatment to counteract the first step other than exercise programmes, there is evidence to suggest that effective treatment of the second step could be achieved using 5HT2A antagonism.","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-10-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46819757","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-10-09DOI: 10.19080/jocct.2018.12.555837
J. Ajit
*Correspondence: Jadhav Ajit, Department of Cardiology, Baroda Heart Institute and Research Centre, 102, Wingsville appt. 41 Arunodaya Society, Alkapuri, Vadodara, Gujarat, 390007, India, Tel: 919028898494; E-mail: ajit92698@gmail.com Received Date: 21 Jun 2018 Accepted Date: 03 Aug 2018 Published Date: 10 Aug 2018 Citation: Ajit J, Suhas H. Adenosine: A Cause of Angina. Ann Cardiol Cardiovasc Med. 2018; 2(2): 1017.
{"title":"Case Report - Adenosine: A Cause of Angina","authors":"J. Ajit","doi":"10.19080/jocct.2018.12.555837","DOIUrl":"https://doi.org/10.19080/jocct.2018.12.555837","url":null,"abstract":"*Correspondence: Jadhav Ajit, Department of Cardiology, Baroda Heart Institute and Research Centre, 102, Wingsville appt. 41 Arunodaya Society, Alkapuri, Vadodara, Gujarat, 390007, India, Tel: 919028898494; E-mail: ajit92698@gmail.com Received Date: 21 Jun 2018 Accepted Date: 03 Aug 2018 Published Date: 10 Aug 2018 Citation: Ajit J, Suhas H. Adenosine: A Cause of Angina. Ann Cardiol Cardiovasc Med. 2018; 2(2): 1017.","PeriodicalId":73635,"journal":{"name":"Journal of cardiology & cardiovascular therapy","volume":" ","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-10-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"44563432","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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