Excessive foot eversion and abnormal tibial rotation have been associated with knee injuries. Why and whether or not overpronation in athletes leads to overuse injuries at the knee joint, however, is still not understood. It could be that this is related to the mechanical coupling at the ankle joint complex. The purpose of this study was to quantify the movement transferred from calcaneal eversion-inversion into tibial rotation, and vice-versa from tibial rotation into calcaneal eversion-inversion. Fourteen foot-leg specimens were used for investigation. A holding device with six degrees of freedom was constructed which allowed to rotate the calcaneus and to determine the resulting tibial rotation, and vice-versa. The movement transfer between calcaneus and tibia varied substantially, and it was not the same for both input modes. In the case of applied internal tibial rotation no calcaneal eversion was found. Apparently, the movement transfer depends from the individual mechanical coupling at the ankle joint complex. Excessive calcaneal eversion does only result in excessive rotational loading of the knee joint when coupled with a high movement transfer at the ankle joint complex. Finally, the results imply that, during gait, pronation results from ground reaction forces acting on the calcaneus, since internal tibial rotation is not transferred into calcaneal eversion.
Most divers and diving medicine specialists know that application of normobaric oxygen as first aid after a bubble disease incident is highly effective. However, as yet technical difficulties acted as a deterrent to using normobaric oxygen at the diving site. This can now be overcome by a newer technique. To be efficient, any therapy of bubble disease should follow three main principles: maximal partial pressure of inhaled oxygen (i.e. 100 kpa in normobaric, and 280 kpa in hyperbaric conditions); minimal partial pressure of inhaled nitrogen, which should ideally be near zero; immediate start of therapy, if possible at the diving site, but not later than 2 hours after the onset of the first symptoms. However, it has to be borne in mind that for an efficient normobaric oxygenation (100%), the standard apparatus design without oxygen reservoir is obsolete, for it offers at most 40% oxygen to the lungs. Currently the following technical approaches for an efficient normobaric oxygenation are available: open one-way systems with tightly fitting mask and oxygen reservoir bag (type Ambu or Leardal, etc.); open systems with on-demand regulation and tightly fitting mouth piece (type SCUBA, or Bird-respirator); closed systems with CO2 absorber (type oxygen rebreathing diving gear). The closed system is a genuine technical advance, because it needs 15 times less oxygen than open systems (about 90 liters oxygen for a 3-hours oxygenation run). Such an apparatus is thus of light weight, far less cumbersome, and nevertheless highly efficient. The therapy should start immediately at the site of the mishap and be maintained during the transport to the next HBO-unit (usually 3 to 6 hours).(ABSTRACT TRUNCATED AT 250 WORDS)
A diving accident may occur during working and leisure time, scientific or archaeological investigations, even during police duty. It is therefore necessary to carry out several preventive measures. The therapeutic treatment based on oxyhyperbaric recompression, is a medical therapy that needs a precise diagnosis and an evaluation of contingent associated pathologies. The following elements have to be checked on the diving place: the anamnestic data, the diving outline and the symptomatology, together with the usual clinical tests. The clinical survey will be completed in hospital, including electrocardiogram, thorax x-ray and laboratory tests. CT-scan, NMR and Evoked Potentials may be considered complementary actions to be taken, according to perceived needs.
An estimation of the electrolytic losses through the skin was evaluated in a run over a distance of 10 km on a 400 m track. Na, K, Ca and Mg excreted by the skin were collected in standardised clothes and thereafter washed out. The remaining electrolytes on the skin were collected by washing the body with deionized water. In addition the concentrations of hormones and metabolites in blood before and after the race were measured. Mean ambient temperature and relative humidity amounted to 21 degrees C and 35%, respectively. The mean performance was 40.5 min. and the average body weight loss was 1.45 kg (1.95% of body weight). The only significant changes in the serum concentrations were the increases of free fatty acids and glycerol. This can be explained, together with a slight increase of glucose and a decrease of insulin, by a higher sympathoadrenergic activity. In the mean 20 mg calcium, 5 mg magnesium, 200 mg potassium and 800 mg sodium were lost by the skin per kg body weight loss. These values compared well with corresponding data found in literature. The described method can therefore be proposed for further experiments.
Although doping in leisure sports may potentially be of relevance for medical emergency situations, it has attracted much less attention than doping in elite athletes. The aim of our study was to evaluate the prevalence of amphetamine consumption in medium altitude mountaineering. Urine samples were taken from 253 males after a successful ascent. Analysis for amphetamines proved positive for 7.1% of mountaineers climbing above 3300 m. On peaks between 2500 to 3300 meters above sea level, 2.7% of the mountaineers we examined had amphetamines residues in their urine. Below 2500 meters, no positive sample was detected. For tourists living outside of the Alpine range, we noticed a significantly higher proportion of positive analyses. We conclude that attempts to induce a higher performance level by pharmacological means are not overly uncommon in leisure mountaineering. Such a behaviour may be of medical relevance in emergency situations.
Between spring and autumn 1990 a study was performed with the goal of recording and classifying overstrain injuries due to rock-climbing and to define their causes. Of the 332 climbers participating in the study, 114 (34.4%) had suffered from at least one overstrain injury. The degree of climbing skill proved to be the main risk factor; with increasing climbing skills of the observed persons the percentage of injuries increased very substantially. The degree of climbing skill also was the only significant difference between injured and non-injured persons--injured persons had a climbing skill which was 1.3 degrees (UIAA) higher. Warming up was unable to prevent most overstrain injuries. A total of 237 injuries were described. 34.6% of these were long-term defects such as foot deformations and nail dystrophies of the toes. 65.4% were overstrain injuries; 90.3% of these cases concerned the upper part of the body and the upper extremities including the thoracic girdle, areas which are particularly strained in climbs of high degrees of difficulty. The areas affected were almost exclusively tendons, joint capsules and ligaments. By far the most frequent injury of the upper extremity was the proximal interphalangeal joint injury, followed by injuries to the proximal phalanx, the flexor tendons of the forearm and the distal interphalangeal joint. With regard to training injuries, finger injuries occurred most frequently in addition to elbow injuries. 51% of the overstrain injuries were severe, with healing times of months to years. Only 30% of the injured persons consulted a physician.