Pub Date : 2018-05-03DOI: 10.15406/JLPRR.2018.05.00166
AR 2018Babaeva, EV 1Kalinina, KS Solodenkova
Systemic connective tissue disorders still remain one of the great diagnostic challenges in real practice because they often begin with nonspecific symptoms and signs and progresses slowly over months and even years. Vasculitisrefers to a heterogeneous group of disorders that is characterized by inflammatory lesion of blood vessels. Depending on size, distribution, and severity of the affected vessels, vasculitis can result in clinical syndromes that vary in severity from a minor self-limited rush to a life-threatening multisystem disorder. Current clinical classification of primary vasculitides is based on clinicopathologic features: they can be separated by the size of affected vessels (large, medium, small).1 Small-vessel vasculitides are associated with antineutrophil cytoplasmic antibodies (ANCA), which can be detected in plasma.2,3 One of the common clinical types of ANCA-associated vasculitis is Churg-Strauss syndrome (CSS) reported at first time by Churg and Strauss in 1951. Asthma, eosinophilia, and systemic vasculitis are the hallmarks of the ChurgStrauss syndrome. The Chapel Hill Consensus Conference defined CSS as a disorder characterized by eosinophil-rich, granulomatous inflammation of the respiratory tract and necrotizing vascilitis of smallto medium vessels with asthma and eosinophilia.1,4 Since lung involvement is a frequent clinical feature of ANCA-associated vasculitides the differential diagnosis should be made to verify systemic vasculitis in patients with pulmonary disorder.3,5 General clinical clues suggesting the presence of systemic vasculitis are constitutional symptoms, signs of inflammation, subacute onset, joint pain, multisystem disease evident. Establishing the diagnosis of vasculitis requires confirmation by laboratory test, biopsy, and/or serology. In term to demonstrate the typical natural history of ANCAassociated vasculitis and diagnostic difficulties in real clinical practice we present observed clinical case.
{"title":"Pulmonary manifestation in churg-strauss syndrome: clinical case","authors":"AR 2018Babaeva, EV 1Kalinina, KS Solodenkova","doi":"10.15406/JLPRR.2018.05.00166","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00166","url":null,"abstract":"Systemic connective tissue disorders still remain one of the great diagnostic challenges in real practice because they often begin with nonspecific symptoms and signs and progresses slowly over months and even years. Vasculitisrefers to a heterogeneous group of disorders that is characterized by inflammatory lesion of blood vessels. Depending on size, distribution, and severity of the affected vessels, vasculitis can result in clinical syndromes that vary in severity from a minor self-limited rush to a life-threatening multisystem disorder. Current clinical classification of primary vasculitides is based on clinicopathologic features: they can be separated by the size of affected vessels (large, medium, small).1 Small-vessel vasculitides are associated with antineutrophil cytoplasmic antibodies (ANCA), which can be detected in plasma.2,3 One of the common clinical types of ANCA-associated vasculitis is Churg-Strauss syndrome (CSS) reported at first time by Churg and Strauss in 1951. Asthma, eosinophilia, and systemic vasculitis are the hallmarks of the ChurgStrauss syndrome. The Chapel Hill Consensus Conference defined CSS as a disorder characterized by eosinophil-rich, granulomatous inflammation of the respiratory tract and necrotizing vascilitis of smallto medium vessels with asthma and eosinophilia.1,4 Since lung involvement is a frequent clinical feature of ANCA-associated vasculitides the differential diagnosis should be made to verify systemic vasculitis in patients with pulmonary disorder.3,5 General clinical clues suggesting the presence of systemic vasculitis are constitutional symptoms, signs of inflammation, subacute onset, joint pain, multisystem disease evident. Establishing the diagnosis of vasculitis requires confirmation by laboratory test, biopsy, and/or serology. In term to demonstrate the typical natural history of ANCAassociated vasculitis and diagnostic difficulties in real clinical practice we present observed clinical case.","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"62 1","pages":"77-79"},"PeriodicalIF":0.0,"publicationDate":"2018-05-03","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77696758","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-04-24DOI: 10.15406/JLPRR.2018.05.00165
H. Daisley, A. Rampersad, D. Meyers
Deep vein thrombosis and pulmonary embolism are common events in a clinical setting. These events often lead to morbidity and mortality and although preventative measures have been instituted, these events occur regularly. In autopsy based studies pulmonary embolism accounts for 1.25% to 10.88% of all cause of death.1,2 In most of these deaths, deep vein thrombosis might have heralded the fatal event of pulmonary embolism which clinically presents with swelling and discomfort of limbs, chest discomfort and breathing difficulties. Pulmonary embolism associated with the act of defecation is seldom reported in the literature.3 This mode of presentation of pulmonary embolism is often fatal and physicians should be aware of this clinical sign of pulmonary embolism.
{"title":"Pulmonary embolism associated with the act of defecation. “The bed pan syndrome”","authors":"H. Daisley, A. Rampersad, D. Meyers","doi":"10.15406/JLPRR.2018.05.00165","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00165","url":null,"abstract":"Deep vein thrombosis and pulmonary embolism are common events in a clinical setting. These events often lead to morbidity and mortality and although preventative measures have been instituted, these events occur regularly. In autopsy based studies pulmonary embolism accounts for 1.25% to 10.88% of all cause of death.1,2 In most of these deaths, deep vein thrombosis might have heralded the fatal event of pulmonary embolism which clinically presents with swelling and discomfort of limbs, chest discomfort and breathing difficulties. Pulmonary embolism associated with the act of defecation is seldom reported in the literature.3 This mode of presentation of pulmonary embolism is often fatal and physicians should be aware of this clinical sign of pulmonary embolism.","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"6 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-04-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"84970436","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-04-23DOI: 10.15406/JLPRR.2018.05.00164
N. Hussain, Faisal FaiyazZuberi, K. Rehana, S. Ashraf, A. Jamal, ShafiKhawar, Talat Mehmood, N. Maheshwary
The chronic obstructive pulmonary disease (COPD) usually results in pulmonary hypertension (PH) and about 40% of patients with COPD suffer from pulmonary hypertension during the course of their illness, that results in worsened prognosis of the disease.1,2 Some studies have defined PH as a PAP >25mm Hg.3,4 Most patients of COPD develop mild to moderate PH with only slight degree of elevation in PAP.5 PH is an essential prognostic factor in determining the course of illness in a patient suffering from COPD.5,6 In fact, some studies suggest that the severity of elevated levels of PAP is a more reliable predictor of prognosis in COPD patients in comparison to FEV1, hypoxemia or hypercapnia.6,7 It has been found that COPD patients who have PAP of >18mm Hg, are more often admitted to hospital due to exacerbations of their condition as compared to those having a mean PAP of <18mm Hg.5 The exacerbations of COPD in patients with PH are responsible for an increase in mortality and morbidity.8
{"title":"Efficacy of bosentan in the treatment of pulmonary hypertension; a prospective study focusing on safety and efficacy in patients with COPD","authors":"N. Hussain, Faisal FaiyazZuberi, K. Rehana, S. Ashraf, A. Jamal, ShafiKhawar, Talat Mehmood, N. Maheshwary","doi":"10.15406/JLPRR.2018.05.00164","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00164","url":null,"abstract":"The chronic obstructive pulmonary disease (COPD) usually results in pulmonary hypertension (PH) and about 40% of patients with COPD suffer from pulmonary hypertension during the course of their illness, that results in worsened prognosis of the disease.1,2 Some studies have defined PH as a PAP >25mm Hg.3,4 Most patients of COPD develop mild to moderate PH with only slight degree of elevation in PAP.5 PH is an essential prognostic factor in determining the course of illness in a patient suffering from COPD.5,6 In fact, some studies suggest that the severity of elevated levels of PAP is a more reliable predictor of prognosis in COPD patients in comparison to FEV1, hypoxemia or hypercapnia.6,7 It has been found that COPD patients who have PAP of >18mm Hg, are more often admitted to hospital due to exacerbations of their condition as compared to those having a mean PAP of <18mm Hg.5 The exacerbations of COPD in patients with PH are responsible for an increase in mortality and morbidity.8","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"13 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-04-23","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"85311073","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-03-30DOI: 10.15406/JLPRR.2018.05.00163
A. Cheepsattayakorn, R. Cheepsattayakorn
{"title":"Promising shorter and more effective treatment regimens for drug-resistant tuberculosis: recent research found","authors":"A. Cheepsattayakorn, R. Cheepsattayakorn","doi":"10.15406/JLPRR.2018.05.00163","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00163","url":null,"abstract":"","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"64 1","pages":"65-65"},"PeriodicalIF":0.0,"publicationDate":"2018-03-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"86196983","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-03-09DOI: 10.15406/JLPRR.2018.05.00162
Fedoseev Gb, Trofimov Vi, Negrutsa Kv, Timchik Vg, Golubeva Vi, Aleks, rin Va, Razumovskaya Ts
Bronchial asthma (BA), chronic obstructive pulmonary disease (COPD), BA+COPD, and community-acquired pneumonia (CAP) are very common conditions. The main pathogenetic mechanism in the abovementioned conditions is inflammation, which is heterogeneous in both its etiology and pathogenesis in patients with these disorders.1 Neutrophils ensure and support body homeostasis and play a pivotal role in the defense against infections.2,3 One of the main causes of a chronic inflammatory process can be neutrophil dysfunction. Reacting to the various pathogenetic factors, neutrophils release active oxygen metabolites, proteolytic enzymes, etc. into the surrounding environment, thus ‘cleaning up’ the inflammatory site but concurrently destroying its own tissues.
{"title":"The functional status of neutrophils in patients with bronchial asthma, chronic obstructive pulmonary disease, bronchial asthma with chronic obstructive pulmonary disease, and community-acquired pneumonia","authors":"Fedoseev Gb, Trofimov Vi, Negrutsa Kv, Timchik Vg, Golubeva Vi, Aleks, rin Va, Razumovskaya Ts","doi":"10.15406/JLPRR.2018.05.00162","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00162","url":null,"abstract":"Bronchial asthma (BA), chronic obstructive pulmonary disease (COPD), BA+COPD, and community-acquired pneumonia (CAP) are very common conditions. The main pathogenetic mechanism in the abovementioned conditions is inflammation, which is heterogeneous in both its etiology and pathogenesis in patients with these disorders.1 Neutrophils ensure and support body homeostasis and play a pivotal role in the defense against infections.2,3 One of the main causes of a chronic inflammatory process can be neutrophil dysfunction. Reacting to the various pathogenetic factors, neutrophils release active oxygen metabolites, proteolytic enzymes, etc. into the surrounding environment, thus ‘cleaning up’ the inflammatory site but concurrently destroying its own tissues.","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"38 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-03-09","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"84938424","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-03-08DOI: 10.15406/JLPRR.2018.05.00161
J. Shrivastava, A. Shrivastava
Lung cancer, also known as lung carcinoma,4 is a malignant lung tumor characterized by uncontrolled cell growth in tissues of the lung.5 This growth can spread beyond the lung by the process of metastasis into nearby tissue or other parts of the body.6 Incidence and mortality attributed to lung cancer has risen steadily since the 1930s, predominantly due to the popularity of cigarette smoking.7,8 Sir Richard Doll and Austin Hill’s landmark article in 1950 described mounting evidence that lung cancer was associated with cigarette smoking.7,9 Number and duration of smoking are directly proportional to incidence of lung cancer.
{"title":"Lung cancer: the global killer","authors":"J. Shrivastava, A. Shrivastava","doi":"10.15406/JLPRR.2018.05.00161","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00161","url":null,"abstract":"Lung cancer, also known as lung carcinoma,4 is a malignant lung tumor characterized by uncontrolled cell growth in tissues of the lung.5 This growth can spread beyond the lung by the process of metastasis into nearby tissue or other parts of the body.6 Incidence and mortality attributed to lung cancer has risen steadily since the 1930s, predominantly due to the popularity of cigarette smoking.7,8 Sir Richard Doll and Austin Hill’s landmark article in 1950 described mounting evidence that lung cancer was associated with cigarette smoking.7,9 Number and duration of smoking are directly proportional to incidence of lung cancer.","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"27 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-03-08","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"83333343","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-02-27DOI: 10.15406/JLPRR.2018.05.00160
A. Abdul-Hafez, Tarek Mohamed, H. Omar, Mohamed A Shemis, B. Uhal
Liver and lung fibrosis are two main organ diseases that are of particular importance in both Egypt and the US. Hepatitis C Virus "HCV" infection and idiopathic pulmonary fibrosis (IPF) are fibrotic diseases of the liver and lung respectively. The liver and lung are reported in literature to share many immune/inflammatory responses to damage through the lung-liver axis. Most importantly, HCV was shown to enhance the development of IPF and is considered one of the risk factors for IPF. The renin angiotensin system (RAS) plays a critical role in the fibrogenesis and inflammation damage of many organs including liver and lung. The relatively recently identified component of RAS, angiotensin converting enzyme-2 (ACE-2), has shown a promising therapeutic potential in models of liver and pulmonary fibrosis. This article reviews the role of RAS in organ fibrosis with focus on role of ACE-2 in fibrotic diseases of the liver and the lung.
{"title":"The renin angiotensin system in liver and lung: impact and therapeutic potential in organ fibrosis.","authors":"A. Abdul-Hafez, Tarek Mohamed, H. Omar, Mohamed A Shemis, B. Uhal","doi":"10.15406/JLPRR.2018.05.00160","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00160","url":null,"abstract":"Liver and lung fibrosis are two main organ diseases that are of particular importance in both Egypt and the US. Hepatitis C Virus \"HCV\" infection and idiopathic pulmonary fibrosis (IPF) are fibrotic diseases of the liver and lung respectively. The liver and lung are reported in literature to share many immune/inflammatory responses to damage through the lung-liver axis. Most importantly, HCV was shown to enhance the development of IPF and is considered one of the risk factors for IPF. The renin angiotensin system (RAS) plays a critical role in the fibrogenesis and inflammation damage of many organs including liver and lung. The relatively recently identified component of RAS, angiotensin converting enzyme-2 (ACE-2), has shown a promising therapeutic potential in models of liver and pulmonary fibrosis. This article reviews the role of RAS in organ fibrosis with focus on role of ACE-2 in fibrotic diseases of the liver and the lung.","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"14 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-02-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"89742020","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-02-19DOI: 10.15406/JLPRR.2018.05.00157
Assao Neino Mm, S. Adjoh, Gagara Ima, A. Taş, Ouedraogo Ar, Okemba Okombi Fh, Fiogbe Aa, S. Alfazazi, M. Bako, M. Da, O. Tidjani
Bronchopulmonary cancers (CBP) are malignant tumors developed at the expense of bronchial and/or parenchymal structures. They may be secondary or primitive. The latter are the leading cause of cancer mortality in developed countries. Several studies show that this cancer will be the leading cause of cancer death in women in France around 2020 [1]. In Africa, primary bronchial cancer appears to be a rare disease, unlike the developed countries, where it is a real public health problem [2]. The positive diagnosis of primary bronchopulmonary cancer is based on the association of: an evocative, nonspecific clinical symptomatology often neglected by the patient, suspicious thoracic imaging and histological evidence obtained by biopsy (bronchus, puncture transthoracic, pleural and lymph node biopsy) [3]. Since 1960, bronchial fibroscopy has been practiced in industrialized countries [4]. In developing countries, the financial constraints inherent in this technique mean that few respiratory services have the necessary equipment to perform bronchial endoscopies [5]. In Togo, there are no thoracic surgeons and 52 cases of bronchopulmonary cancers were observed from 1982 to 1987 at CHU Sylvanus Olympio in Lomé on 275 bronchial fibroscopies, ie 18.9% [6]. This led us to carry out this work, the aim of which is to take stock of primary bronchopulmonary cancers at the CHU Sylvanus Olympio, in the Infectious Diseases and Pneumology Department (SMIP).
{"title":"Status of Primitive Bronchopulmonary Cancers in the Sylvanus Olympio Chu Pneumology Sevice (Togo)","authors":"Assao Neino Mm, S. Adjoh, Gagara Ima, A. Taş, Ouedraogo Ar, Okemba Okombi Fh, Fiogbe Aa, S. Alfazazi, M. Bako, M. Da, O. Tidjani","doi":"10.15406/JLPRR.2018.05.00157","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00157","url":null,"abstract":"Bronchopulmonary cancers (CBP) are malignant tumors developed at the expense of bronchial and/or parenchymal structures. They may be secondary or primitive. The latter are the leading cause of cancer mortality in developed countries. Several studies show that this cancer will be the leading cause of cancer death in women in France around 2020 [1]. In Africa, primary bronchial cancer appears to be a rare disease, unlike the developed countries, where it is a real public health problem [2]. The positive diagnosis of primary bronchopulmonary cancer is based on the association of: an evocative, nonspecific clinical symptomatology often neglected by the patient, suspicious thoracic imaging and histological evidence obtained by biopsy (bronchus, puncture transthoracic, pleural and lymph node biopsy) [3]. Since 1960, bronchial fibroscopy has been practiced in industrialized countries [4]. In developing countries, the financial constraints inherent in this technique mean that few respiratory services have the necessary equipment to perform bronchial endoscopies [5]. In Togo, there are no thoracic surgeons and 52 cases of bronchopulmonary cancers were observed from 1982 to 1987 at CHU Sylvanus Olympio in Lomé on 275 bronchial fibroscopies, ie 18.9% [6]. This led us to carry out this work, the aim of which is to take stock of primary bronchopulmonary cancers at the CHU Sylvanus Olympio, in the Infectious Diseases and Pneumology Department (SMIP).","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"534 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-02-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"85668914","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2018-02-01DOI: 10.15406/JLPRR.2018.05.00158
Fatma Irem Yesiler, M. A. Izgi, Akif Demirel, S. Ozdemir
The acute respiratory distress syndrome ARDS is characterized by a non cardiogenic pulmonary edema with bilateral chest radiograph opacities and hypoxemia refractory to oxygen therapy Adenovirus is a frequent cause of mild self limiting upper respiratory tract infection gastroenteritis and conjunctivitis in infants and young children Fatal infections severe pneumonia progressing to respiratory failure septic shock and or encephalitis are rare among immunocompetent adults We report a case of twenty two years old pregnant woman who had severe adenovirus pneumonia She presented with sudden onset of respiratory distress and progressed rapidly to respiratory failure She was successfully recovered with supportive measures
{"title":"A Case of ARDS Due to Adenoviral Pneumonia","authors":"Fatma Irem Yesiler, M. A. Izgi, Akif Demirel, S. Ozdemir","doi":"10.15406/JLPRR.2018.05.00158","DOIUrl":"https://doi.org/10.15406/JLPRR.2018.05.00158","url":null,"abstract":"The acute respiratory distress syndrome ARDS is characterized by a non cardiogenic pulmonary edema with bilateral chest radiograph opacities and hypoxemia refractory to oxygen therapy Adenovirus is a frequent cause of mild self limiting upper respiratory tract infection gastroenteritis and conjunctivitis in infants and young children Fatal infections severe pneumonia progressing to respiratory failure septic shock and or encephalitis are rare among immunocompetent adults We report a case of twenty two years old pregnant woman who had severe adenovirus pneumonia She presented with sudden onset of respiratory distress and progressed rapidly to respiratory failure She was successfully recovered with supportive measures","PeriodicalId":91750,"journal":{"name":"Journal of lung, pulmonary & respiratory research","volume":"78 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2018-02-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74874139","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
京公网安备 11010802042870号
京ICP备2023020795号-1
扫码关注我们
求助内容：
应助结果提醒方式：