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Hypoxia-inducible factor signaling regulates embryonic interneuron development, GRIN2B expression and adult cortical function. 缺氧诱导因子信号调节胚胎间神经元发育、GRIN2B表达和成人皮质功能。
IF 8.7 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-02-05 DOI: 10.1016/j.devcel.2026.01.007
I-Ling Lu, Mengyi Song, Cinthia Rangel-Sandoval, Juan Moriano Palacios, JaeYeon Kim, Eric J Huang, Arturo Alvarez-Buylla, Arnold Kriegstein, Mercedes F Paredes, David H Rowitch

Although hypoxia-inducible factors (HIFs) are central regulators of cellular adaptation to oxygen and metabolic fluctuations in the mammalian brain, potential roles for HIF regulation during inhibitory neuron development are poorly understood. Here, we report that Nkx2.1-cre-driven conditional deletion of Hif1/2a in the medial ganglionic eminence (MGE) leads to reduced proliferation of Lhx6-positive interneuron precursors, whereas loss of von Hippel-Lindau (vHL), required for HIF degradation, drives increased precursor proliferation. Integrating single-cell transcriptomics, we identified HIF targets regulating proliferation and synaptogenesis. We also show that HIF1A directly activates glutamate ionotropic receptor NMDA type subunit 2B (GRIN2B), encoding glutamate ionotropic receptor N-methyl-D-aspartate (NMDA) subunit 2B. In the adult HIF1 conditional knockout (cKO) cortex, we observed decreased numbers of parvalbumin (PV) interneurons and fewer GABAergic synapses and GRIN2B/Bassoon puncta on layer 2/3 excitatory neurons, resulting in attenuated long-term potentiation. These findings identify non-canonical roles for HIF signaling that are essential for PV interneuron production, GRIN2B expression, and cortical circuit maturation and function.

尽管缺氧诱导因子（HIF）是哺乳动物大脑中细胞适应氧气和代谢波动的主要调节因子，但HIF调节在抑制性神经元发育中的潜在作用尚不清楚。在这里，我们报告了nkx2.1 - cred驱动的内侧神经节隆起（MGE）中Hif1/2a的条件缺失导致lhx6阳性中间神经元前体的增殖减少，而HIF降解所需的von Hippel-Lindau （vHL）的缺失导致前体增殖增加。结合单细胞转录组学，我们确定了调节增殖和突触发生的HIF靶点。我们还发现HIF1A直接激活谷氨酸离子化受体NMDA型亚基2B (GRIN2B)，编码谷氨酸离子化受体n -甲基- d -天冬氨酸（NMDA）亚基2B。在成人HIF1条件敲除（cKO）皮层中，我们观察到2/3层兴奋性神经元上的小白蛋白（PV）中间神经元数量减少，gaba能突触和GRIN2B/巴松管点减少，导致长期增强减弱。这些发现确定了HIF信号在PV中间神经元产生、GRIN2B表达和皮质回路成熟和功能中所必需的非规范作用。
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引用次数: 0
Mitogen-activated kinase 6 facilitates autophagy initiation to confer salt tolerance in maize 丝裂原活化激酶6促进自噬起始，赋予玉米耐盐性
IF 11.8 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-02-03 DOI: 10.1016/j.devcel.2026.01.002
Wenqi Jing, Pan Yin, Changyun Liu, Tao Zhou, Fenrong Li, Fang Liu, Kaitong Du, Jörg Kudla, Caifu Jiang
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引用次数: 0
Mitigating xenogeneic barriers to chimerism through Cas13-induced host attenuation 通过cas13诱导的宿主衰减减轻异种嵌合障碍
IF 11.8 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-29 DOI: 10.1016/j.devcel.2026.01.001
Bingbing He, Shijian Lv, Leijie Li, Lin Zhang, Yingying Hu, Daiji Okamura, Jia Huang, Jun Wu
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引用次数: 0
MYRF controls mesothelium specification, signaling, and plasticity in lung development MYRF控制肺发育中的间皮规范、信号传导和可塑性
IF 11.8 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-19 DOI: 10.1016/j.devcel.2025.12.011
Gidsela Luna, Jamie M. Verheyden, Chunting Tan, Estelle Kim, Ziai Zhu, Michelle Hwa, Jugraj Sahi, Yufeng Shen, Wendy K. Chung, David J. McCulley, Xin Sun
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引用次数: 0
Sticky neutrophils, risky tumors: NETs in DICER1-associated rhabdomyosarcoma. 黏性中性粒细胞，危险肿瘤：dicer1相关横纹肌肉瘤的NETs。
IF 8.7 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.12.001
Siyue Wang, Xiang H-F Zhang

In this issue of Developmental Cell, Larsen, Hanna et al. show that germline Dicer1 haploinsufficiency promotes fusion-negative rhabdomyosarcoma (FN-RMS) through neutrophil accumulation and the production of neutrophil extracellular traps (NETs). This research uncovers a non-cell-autonomous mechanism of DICER1 tumor predisposition whereby NET-prone neutrophils transform the tumor microenvironment and promote tumor expansion.

在本期《发育细胞》中，Larsen， Hanna等人发现种系Dicer1单倍不全通过中性粒细胞积累和中性粒细胞胞外陷阱（NETs）的产生促进融合阴性横纹肌肉瘤（FN-RMS）。本研究揭示了DICER1肿瘤易感的非细胞自主机制，即net易感中性粒细胞改变肿瘤微环境，促进肿瘤扩张。
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引用次数: 0
The metabolic mood: Cholesterol homeostasis as a convergence point for depression risk 代谢情绪：胆固醇内稳态是抑郁风险的趋同点
IF 11.8 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.12.008
Mu Seog Choe, In-Hyun Park
Major depressive disorder arises from an interplay of genetic and environmental factors. In this issue of Developmental Cell, Oberst et al. reveal that chronic stress, inflammation, and SIRT1 deficiency converge on a defect in neuronal cholesterol homeostasis. Restoring cholesterol levels rescues this deficit, highlighting lipid metabolism as a driver of depression.
重度抑郁症是遗传和环境因素共同作用的结果。在本期的《发育细胞》杂志上，Oberst等人揭示了慢性应激、炎症和SIRT1缺乏汇聚在神经元胆固醇稳态缺陷上。恢复胆固醇水平挽救了这一缺陷，强调了脂质代谢是抑郁症的驱动因素。
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引用次数: 0
Integrin-mediated adhesion drives microglial entry into the developing CNS. 整合素介导的粘附驱动小胶质细胞进入发育中的中枢神经系统。
IF 8.7 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.12.006
Fanny Jaudon, Lorenzo A Cingolani

In this issue of Developmental Cell, Petry et al. show that early microglial progenitors infiltrate the embryonic CNS via an extracellular matrix (ECM)-rich pial route requiring talin-1-dependent integrin activation. This work revises long-standing vascular entry models and highlights mechanosensitive adhesion as a regulator of early neuroimmune assembly.

在这一期的《发育细胞》中，Petry等人发现早期小胶质祖细胞通过细胞外基质（ECM）丰富的脑顶通路浸润胚胎中枢神经系统，这需要talin-1依赖性整合素激活。这项工作修订了长期存在的血管进入模型，并强调机械敏感性粘附作为早期神经免疫组装的调节因子。
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引用次数: 0
TMK-PIN1 drives a short self-organizing circuit for auxin export and signaling in Arabidopsis. TMK-PIN1驱动拟南芥中生长素输出和信号传导的短自组织电路。
IF 8.7 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.11.004
Rongfeng Huang, Jiacheng Wang, Mingzeng Chang, Wenxin Tang, Yongqiang Yu, Yi Zhang, Yao Peng, Yanan Wang, Yihan Guo, Ting Lu, Yang Cao, Yewei Zhou, Qinglin Zhang, Yuehong Huang, Angxiao Wu, Luyan Ren, Michelle Gallei, Juan Dong, Haodong Chen, Jun He, Mingzhang Wen, Jiří Friml, Linfeng Sun, Yan Xiong, Zhenbiao Yang, Tongda Xu
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引用次数: 0
Lmo3-expressing peri-isthmus progenitor cells sustain renewal and repair of the mammalian intestinal telocyte niche. 表达lmo3的峡周祖细胞维持哺乳动物肠远端细胞生态位的更新和修复。
IF 8.7 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.11.007
Daxin Jiang, Guoli Zhu, Yongchao Zhang, Jiawen Wang, Nannan Qian, Zhen Jin, Qingyu Sun, Haimeng Yu, Kebei Tang, Tao Cai, Fengchao Wang, Rongwen Xi
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引用次数: 0
A high-humidity niche at the shoot meristem shapes plant adaption to terrestrial environments 茎部分生组织的高湿生态位决定了植物对陆地环境的适应
IF 11.8 1区 生物学 Q1 CELL BIOLOGY
Developmental cell
Pub Date : 2026-01-14 DOI: 10.1016/j.devcel.2025.12.004
Dani Amat, Viktoriia Voloboeva, Daan A. Weits
In this issue of Developmental Cell, Zhu et al. report that plants maintain a high-humidity niche within their meristematic zones through enclosing organs and regulating the expression of aquaporins. The epidermal hydrodynamics are conserved across land plants, likely facilitating land colonization while enabling adaptation to arid environments.
在本期的Developmental Cell中，Zhu等人报道了植物通过封闭器官和调节水通道蛋白的表达来维持其分生组织区域内的高湿生态位。在陆地植物中，表皮流体动力学是保守的，这可能促进了陆地定植，同时使其能够适应干旱环境。
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引用次数: 0
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