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Meet Our Editorial Board—Genesis. An Interview With Jun (Kelly) Liu, Cornell University, New York, USA 认识我们的编辑委员会--创世纪。美国纽约康奈尔大学刘俊（Kelly）访谈录
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-02-26 DOI: 10.1002/dvg.70006
Jun (Kelly) Liu, Paul Trevorrow
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With, Mark Lewandoski, National Cancer Institute, Maryland, USA 见见我们的编辑委员会——创世纪。采访马克·莱万多斯基，美国马里兰州国家癌症研究所
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-02-26 DOI: 10.1002/dvg.70007
Mark Lewandoski, Paul Trevorrow
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引用次数: 0
Unraveling the Mechanisms That Regulate Osteoclast Differentiation: A Review of Current Advances 揭示调节破骨细胞分化的机制：综述当前进展
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-02-17 DOI: 10.1002/dvg.70012
Sai Zhang, Meng Gao, Shuzhe Song, Tongdan Zhao, Bianhua Zhou, Hongwei Wang, Weishun Tian, Wenpeng Zhao, Jing Zhao

Osteoporosis is a metabolic bone disease primarily caused by a decreased bone formation and increased bone resorption. Osteoclasts are a special class of terminally differentiated cells that play an important role in normal bone remodeling and bone loss in osteoporosis as well as in a variety of osteolytic diseases. Osteoclasts can be differentiated from monocyte–macrophage cells of the hematopoietic system; they are the key cells in bone resorption. Osteoclast formation and differentiation are regulated by various cytokines and transcription factors. In this review, we summarize recent advances in research on the regulation of osteoclast differentiation and function by factors such as M-CSF, RANKL, AP-1, NFATC1, MITF, and PU.1. Understanding these cytokines and transcription factors can not only help identify targets for osteoclast differentiation but also aid in intervening in the treatment of osteoclast-related diseases.

骨质疏松症是一种代谢性骨病，主要由骨形成减少和骨吸收增加引起。破骨细胞是一类特殊的终末分化细胞，在骨质疏松症的正常骨重塑和骨质流失以及多种溶骨疾病中起重要作用。破骨细胞可以从造血系统的单核-巨噬细胞中分化出来；它们是骨吸收的关键细胞。破骨细胞的形成和分化受多种细胞因子和转录因子的调控。本文综述了M-CSF、RANKL、AP-1、NFATC1、MITF和PU.1等因子对破骨细胞分化和功能调控的最新研究进展。了解这些细胞因子和转录因子不仅有助于确定破骨细胞分化的靶点，而且有助于干预破骨细胞相关疾病的治疗。
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引用次数: 0
A New Targeted Transgenic Mouse Line for the Study of Protocadherin γC4 研究原钙粘蛋白γ - c4的新靶向转基因小鼠系
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-02-09 DOI: 10.1002/dvg.70010
Cathy M. McLeod, Camille M. Hanes, Leah C. Fuller, Samjhana Bhandari, Hannah G. Lanthier, Robert W. Burgess, Joshua A. Weiner, Andrew M. Garrett

The γ-protocadherins (γ-Pcdhs) comprise 22 homophilic cell adhesion molecule isoforms, expressed from the Pcdhg gene cluster via promoter choice mechanisms that serve many crucial functions during neural development. Emerging evidence supports the hypothesis that distinct isoforms have unique functions. The γC4 isoform, which is expressed from the Pcdhgc4 promoter and includes its unique variable exon, is the sole γ-Pcdh isoform essential for the postnatal survival in mice. Here we describe a new mouse line (C4-GFP) in which Pcdhgc4 with a C-terminal GFP tag is expressed from the Rosa26 locus following excision of a lox-Stop-lox cassette by Cre recombinase. We report that restricted expression of this transgene in the nervous system using Nestin-Cre is sufficient to rescue the neonatal lethality of mice mutant for Pcdhgc4. This new line will be a vital tool for dissecting mechanisms underlying the functions of this essential cell adhesion molecule gene, mutations in which have been associated with neurodevelopmental disorders in humans.

γ-原钙粘蛋白（γ-Pcdhs）由22种亲同型细胞粘附分子组成，由Pcdhg基因簇通过启动子选择机制表达，在神经发育过程中起着许多重要作用。新出现的证据支持不同同种异构体具有独特功能的假设。γ- c4异构体由Pcdhgc4启动子表达，包括其独特的可变外显子，是小鼠出生后生存所必需的唯一γ-Pcdh异构体。在这里，我们描述了一个新的小鼠系（C4-GFP），在Cre重组酶切除lox-Stop-lox盒后，带有c端GFP标签的Pcdhgc4从Rosa26位点表达。我们报道，使用nesting - cre在神经系统中限制性表达该转基因足以挽救Pcdhgc4突变体小鼠的新生儿死亡。这一新产品线将成为解剖这种基本细胞粘附分子基因的功能机制的重要工具，这种基因的突变与人类神经发育障碍有关。
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With, Susan Mackem, National Cancer Institute, Maryland, USA 见见我们的编辑委员会——创世纪。采访美国马里兰州国家癌症研究所的苏珊·麦克姆。
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-01-29 DOI: 10.1002/dvg.70011
Paul Trevorrow, Susan Mackem
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With Yevgenya Grinblat, University of Wisconsin-Madison, Wisconsin, USA 见见我们的编辑委员会——创世纪。美国威斯康辛大学麦迪逊分校Yevgenya Grinblat访谈
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-01-27 DOI: 10.1002/dvg.70005
Paul Trevorrow, Yevgenya Grinblat
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With Thomas Schimmang, Institute for Biomedicine and Molecular Genetics, Valladolid, Spain 见见我们的编辑委员会——创世纪。采访托马斯·希曼，西班牙巴利亚多利德生物医学和分子遗传学研究所。
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-01-27 DOI: 10.1002/dvg.70004
Paul Trevorrow, Thomas Schimmang
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With Paolo E. Forni, University at Albany, State University of New York, New York, USA 见见我们的编辑委员会——创世纪。采访美国纽约州立大学奥尔巴尼分校的保罗·e·福尼
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-01-24 DOI: 10.1002/dvg.70002
Paul Trevorrow, Paolo E. Forni
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引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With Yoh-suke Mukouyama, National Institutes of Health, Maryland, USA 见见我们的编辑委员会——创世纪。访美国马里兰州国立卫生研究院的佑介·慕山
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2025-01-24 DOI: 10.1002/dvg.70008
Yoh-suke Mukouyama, Paul Trevorrow
{"title":"Meet Our Editorial Board—Genesis. An Interview With Yoh-suke Mukouyama, National Institutes of Health, Maryland, USA","authors":"Yoh-suke Mukouyama,&nbsp;Paul Trevorrow","doi":"10.1002/dvg.70008","DOIUrl":"10.1002/dvg.70008","url":null,"abstract":"","PeriodicalId":12718,"journal":{"name":"genesis","volume":"62 6","pages":""},"PeriodicalIF":2.4,"publicationDate":"2025-01-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"143034682","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"生物学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Meet Our Editorial Board—Genesis. An Interview With, Sally Moody, The George Washington University School of Medicine and Health Sciences, USA 认识我们的编辑委员会--《创世纪》。专访美国乔治-华盛顿大学医学与健康科学学院莎莉-穆迪。
IF 2.4 4区 生物学 Q2 DEVELOPMENTAL BIOLOGY
genesis
Pub Date : 2024-12-31 DOI: 10.1002/dvg.70000
Paul Trevorrow, Sally A. Moody
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引用次数: 0
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