Pub Date : 2020-05-05DOI: 10.31829/2641-7456/ahs2020-4(1)-113
Aamir Jalal Al-Mosawi
Background: Autistic disorders have become increasingly known as pervasive developmental disorders since the 1980s. They have been recently called autism spectrum disorder mostly by the American Psychiatric Association. They include five chronic disorders marked by early impairment in socialization, communication, and behavior. There is no curative therapy or therapies for autistic disorders, and they continue to be regarded as life long disorders. The aim of this paper is to describe our extensive experiences with treatment of autistic disorders with emphasis on the possibility of curing these disorders with a new therapeutic approach. Marked improvement or disappearance of autistic features in these disorders has not been reported with any therapy before.
{"title":"Cure of Autistic Disorders: Mission Impossible is Possible in an Illustrated Pioneering Experience","authors":"Aamir Jalal Al-Mosawi","doi":"10.31829/2641-7456/ahs2020-4(1)-113","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-113","url":null,"abstract":"Background: Autistic disorders have become increasingly known as pervasive developmental disorders since the 1980s. They have been recently called autism spectrum disorder mostly by the American Psychiatric Association. They include five chronic disorders marked by early impairment in socialization, communication, and behavior. There is no curative therapy or therapies for autistic disorders, and they continue to be regarded as life long disorders. The aim of this paper is to describe our extensive experiences with treatment of autistic disorders with emphasis on the possibility of curing these disorders with a new therapeutic approach. Marked improvement or disappearance of autistic features in these disorders has not been reported with any therapy before.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"11 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-05-05","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"114829313","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-27DOI: 10.31829/2641-7456/ahs2020-4(1)-112
Rationale: Coronavirus disease is a current worldwide pandemic serious infection. Septic shock is a very common cause of death due to COVID-19 infection. There is a strong relationship between the severity of COVID-19 infection and death. COVID-19 infection is a well-known trigger for severe inflammatory changes and sepsis. Up till now, there is no well-known treatment for COVID-19. Patient concerns: A young girl COVID-19 patient admitted to the critical care unit with severe shock and developing pneumonia. Diagnosis: COVID-19 patient admitted to the critical care unit with COVID-19 young girl patient with septic shock and developing pneumonia. Interventions: Electrocardiography, arterial blood gas test, chest CT scan, central venous catheterization, and intravenous vasopressors infusion. Outcomes: Progressive dramatic clinical electrocardiography and radiological improvement had happened. Lessons: It denotes the role of anticoagulants, vasopressors, steroids, and antibiotics in the management of COVID-19 pneumonic infection and septic shock. C- reactive protein, lymphocytic count, liver enzymes, and CT chest may be used as a good laboratory guide for follow up for the inflammatory activity of COVID-19 patient. Blood pressure, respiratory rate, and O2 saturation are another strong guide for clinical for follow up in COVID-19 patients.
{"title":"A COVID-19 Young Girl Patient with Developing Pneumonia and Septic Shock Passing the Death","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-112","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-112","url":null,"abstract":"Rationale: Coronavirus disease is a current worldwide pandemic serious infection. Septic shock is a very common cause of death due to COVID-19 infection. There is a strong relationship between the severity of COVID-19 infection and death. COVID-19 infection is a well-known trigger for severe inflammatory changes and sepsis. Up till now, there is no well-known treatment for COVID-19. Patient concerns: A young girl COVID-19 patient admitted to the critical care unit with severe shock and developing pneumonia. Diagnosis: COVID-19 patient admitted to the critical care unit with COVID-19 young girl patient with septic shock and developing pneumonia. Interventions: Electrocardiography, arterial blood gas test, chest CT scan, central venous catheterization, and intravenous vasopressors infusion. Outcomes: Progressive dramatic clinical electrocardiography and radiological improvement had happened. Lessons: It denotes the role of anticoagulants, vasopressors, steroids, and antibiotics in the management of COVID-19 pneumonic infection and septic shock. C- reactive protein, lymphocytic count, liver enzymes, and CT chest may be used as a good laboratory guide for follow up for the inflammatory activity of COVID-19 patient. Blood pressure, respiratory rate, and O2 saturation are another strong guide for clinical for follow up in COVID-19 patients.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"84 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-27","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"132412466","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-20DOI: 10.31829/2641-7456/ahs2020-4(1)-111
A problem, that is while noticed by neither science nor public, stands before humanity. This problem carries the name «Biological degradation of type of Homo Sapiens». She is characterized by the decline of viability of population, epidemic chronic uninfectious of diseases, by the speed-up rate of aging of and other In basis of this phenomenon is a decline of stability of the thermodynamics system - organism of man. The ways of decision of problem are offered.
{"title":"Social Evolution and Health of Man","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-111","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-111","url":null,"abstract":"A problem, that is while noticed by neither science nor public, stands before humanity. This problem carries the name «Biological degradation of type of Homo Sapiens». She is characterized by the decline of viability of population, epidemic chronic uninfectious of diseases, by the speed-up rate of aging of and other In basis of this phenomenon is a decline of stability of the thermodynamics system - organism of man. The ways of decision of problem are offered.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"11 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-20","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"122228339","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-20DOI: 10.31829/2641-7456/ahs2020-4(1)-107
This conceptual study focuses on the evolving field of potential and the mentality of genius, inspiration and milestone breakthroughs alongside embedding several stories of longevity, aging-related diseases and beyond. It aims to help promote government strategy, fostering apprenticeships, and renewing ideas about the role of family, hardship, war, paradigm shifts and religion in developing genius and inspiration that will help make more groundbreaking discoveries in future
{"title":"Potential and Mentality of Genius and Inspiration and Milestone Breakthroughs: Longevity, Aging-Related Diseases and Beyond","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-107","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-107","url":null,"abstract":"This conceptual study focuses on the evolving field of potential and the mentality of genius, inspiration and milestone breakthroughs alongside embedding several stories of longevity, aging-related diseases and beyond. It aims to help promote government strategy, fostering apprenticeships, and renewing ideas about the role of family, hardship, war, paradigm shifts and religion in developing genius and inspiration that will help make more groundbreaking discoveries in future","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"49 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-20","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121735294","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-15DOI: 10.31829/2641-7456/ahs2020-4(1)-110
Abstract: Antibacterial mechanism of Ag+ ion solution against S. aureus had been found that Ag+-induced S. aureus may inactivate PGN synthesis transglycosylase TG and transpeptidase TP. Bacteriolysis of S. aureus PGN cell wall, in which wall teichoic acids control PGN synthesis crosslinking TP, is due to the inhibition of PGN elongation by enhancing the activities of PGN autolysins; amidase AmiA and AmiE, and PGN hydrolase Lysostaphin-like endopeptidase (Glycine-Glycine bond cleavage). Against E. coli, the antibacterial mechanism of Ag+ ion solution had been found that bacteriolysis and destruction of E.coli cell wall by silver ions are caused by the destruction of outer membrane structure owing to the activation of endopeptidase of lipoprotein at C-, and N-terminals, and inhibition of PGN elongation due to the damage of PGN synthetic enzyme of silver-protein Amidase in periplasmic space, and PGN autolysins of Amidase, Peptidase, and Carboxypeptidase. Bacteriolysis and destruction of E.coli cell wall are due to the damage of LPS synthesis, destructing of outer membrane structure by degrading of lipoprotein at C-, N-terminals, owing to inhibition of PGN formations by inactivation of carboxypeptidase and TP-endopeptidase, and activities of PGN autolysins of amidase, peptidase and carboxypeptidase. Bacteriolysis of S.aureus PGN cell wall by Cu2+ ions is thought to be due to inhibition of PGN elongation owing to the damages of PGN synthetic TG/TP and the activations of PGN autolysin, AmiA. Bacteriolysis of E.coli cell wall by Cu2+ ions occurs by destruction of outer membrane structure due to degradation of lipoprotein at N-, C-terminals, damage of TP enzyme and activations of PGN autolysins. Furthermore, deletion of PGN autolysin also becomes bacteriolytic factor. It is thought that the activations of these PGN autolysins by Zn2+ ions could be enhanced the inhibitions of PGN elongation simultaneously, with bacteriolysis of S. aureus PGN cell wall. Bacteriolysis of E. coli cell wall by Zn2+ ions are due to destruction of outer membrane structure by degrading of lipoprotein at C-, Nterminals, owing to PGN formation inhibition by activities of PGN autolysins of amidase and carboxypeptidase-transpeptidase Ag+,Cu2+,Zn2+ ions-induced ROS generation of O2 - and H2O2 and ROS-mediated oxidative stress in bacterial cell lead to killing by stress damage for silver ions, cell membrane damages due to high reactive •OH and OH－are formed by Haber-Weiss and Fenton reactions for Cu2+ ions, and DNA molecular damage for Zn2+ ions.
{"title":"Anti-Bacterial Mechanisms for Ag+ , Cu2+, and Zn2+ Ion Solutions against Staphylococcus Aureus and Escherichia Coli","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-110","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-110","url":null,"abstract":"Abstract:\u0000Antibacterial mechanism of Ag+ ion solution against S. aureus had been found that Ag+-induced S. aureus may inactivate PGN synthesis transglycosylase TG and transpeptidase TP. Bacteriolysis of S. aureus PGN cell wall, in which wall teichoic acids control PGN synthesis crosslinking TP, is due to the inhibition of PGN elongation by enhancing the activities of PGN autolysins; amidase AmiA and AmiE, and PGN hydrolase Lysostaphin-like endopeptidase (Glycine-Glycine bond cleavage). Against E. coli, the antibacterial mechanism of Ag+ ion solution had been found that bacteriolysis and destruction of E.coli cell wall by silver ions are caused by the destruction of outer membrane structure owing to the activation of endopeptidase of lipoprotein at C-, and N-terminals, and inhibition of PGN elongation due to the damage of PGN synthetic enzyme of silver-protein Amidase in periplasmic space, and PGN autolysins of Amidase, Peptidase, and Carboxypeptidase. Bacteriolysis and destruction of E.coli cell wall are due to the damage of LPS synthesis, destructing of outer membrane structure by degrading of lipoprotein at C-, N-terminals, owing to inhibition of PGN formations by inactivation of carboxypeptidase and TP-endopeptidase, and activities of PGN autolysins of amidase, peptidase and carboxypeptidase. \u0000\u0000Bacteriolysis of S.aureus PGN cell wall by Cu2+ ions is thought to be due to inhibition of PGN elongation owing to the damages of PGN synthetic TG/TP and the activations of PGN autolysin, AmiA. Bacteriolysis of E.coli cell wall by Cu2+ ions occurs by destruction of outer membrane structure due to degradation of lipoprotein at N-, C-terminals, damage of TP enzyme and activations of PGN autolysins. Furthermore, deletion of PGN autolysin also becomes bacteriolytic factor. \u0000\u0000It is thought that the activations of these PGN autolysins by Zn2+ ions could be enhanced the inhibitions of PGN elongation simultaneously, with bacteriolysis of S. aureus PGN cell wall. Bacteriolysis of E. coli cell wall by Zn2+ ions are due to destruction of outer membrane structure by degrading of lipoprotein at C-, Nterminals, owing to PGN formation inhibition by activities of PGN autolysins of amidase and carboxypeptidase-transpeptidase \u0000\u0000Ag+,Cu2+,Zn2+ ions-induced ROS generation of O2 - and H2O2 and ROS-mediated oxidative stress in bacterial cell lead to killing by stress damage for silver ions, cell membrane damages due to high reactive •OH and OH－are formed by Haber-Weiss and Fenton reactions for Cu2+ ions, and DNA molecular damage for Zn2+ ions.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"99 ","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"121204617","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-11DOI: 10.31829/2641-7456/ahs2020-4(1)-109
Introduction Many researchers don’t believe in our works: they speak we are wrong when we evoke the Invertebrate Antibody...BUT: Discussion We have discovered, in the past, the sea star lymphocytes ( B and T sea star lymphocytes : cf Fig 1,2) : these cells of 4-5µ in diameter are smaller than Vertebrate lymphocytes And ...We discovered recently the IPA (Invertebrate Primitive Antibody) with the sea star IGKAPPA gene with IG sites (Meta Gene 2013). Therefore genomic data assert the evidence of primitive antibody in Echinodermata. Furthermore, we find MHC genes class I and class II in 2019 and Fab gene, Fc receptor gene in these Invertebrates
{"title":"The Invertebrate Antibody","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-109","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-109","url":null,"abstract":"Introduction \u0000Many researchers don’t believe in our works: they speak we are wrong when we evoke the Invertebrate Antibody...BUT: \u0000\u0000Discussion \u0000We have discovered, in the past, the sea star lymphocytes ( B and T sea star lymphocytes : cf Fig 1,2) : these cells of 4-5µ in diameter are smaller than Vertebrate lymphocytes And ...We discovered recently the IPA (Invertebrate Primitive Antibody) with the sea star IGKAPPA gene with IG sites (Meta Gene 2013). \u0000\u0000Therefore genomic data assert the evidence of primitive antibody in Echinodermata. \u0000\u0000Furthermore, we find MHC genes class I and class II in 2019 and Fab gene, Fc receptor gene in these Invertebrates","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"19 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-11","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"114886631","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-04-07DOI: 10.31829/2641-7456/ahs2020-4(1)-108
Background: Hypertension is a chronic condition due to the high level of blood pressure. There are 2 types of hypertension. Hypertension patients are usually consumed anti-hypertension medication to prevent the high blood pressure which can affect the damage of blood vessel in the body. The used of anti-hypertension drugs can cause the oral lesion because antihypertension drugs have several effect on oral mucosa until the dysfunction of salivary glands. Objective: to know the oral lesions that associated with hypertension. Problem Statement: The used of anti-hypertension drug in hypertension patients have side effect. The side effect can cause the oral lesion and tissue damage in oral cavity. Discussion: Cardiovascular drugs such as anti-hypertension can cause oral ulcers such as agranulocytosis and thrompositopenia with hemorrhagic bullae, peteki, ecchymosis with bleeding. Terasozin and prasozin which are anti-hypertensive drugs can inhibit the salivary glands of alpha-adrenoreceptors, causing dry mouth due to reduced saliva production. Conclusion: Hypertension has a specific role as a cause of oral lesion. The use of antihypertension drug has shown the associated with oral mucosa damage.
{"title":"Oral Lesion Associated with Hypertension: A Mini Review","authors":"","doi":"10.31829/2641-7456/ahs2020-4(1)-108","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-108","url":null,"abstract":"Background: Hypertension is a chronic condition due to the high level of blood pressure. There are 2 types of hypertension. Hypertension patients are usually consumed anti-hypertension medication to prevent the high blood pressure which can affect the damage of blood vessel in the body. The used of anti-hypertension drugs can cause the oral lesion because antihypertension drugs have several effect on oral mucosa until the dysfunction of salivary glands. \u0000\u0000Objective: to know the oral lesions that associated with hypertension. \u0000\u0000Problem Statement: The used of anti-hypertension drug in hypertension patients have side effect. The side effect can cause the oral lesion and tissue damage in oral cavity. \u0000\u0000Discussion: Cardiovascular drugs such as anti-hypertension can cause oral ulcers such as agranulocytosis and thrompositopenia with hemorrhagic bullae, peteki, ecchymosis with bleeding. Terasozin and prasozin which are anti-hypertensive drugs can inhibit the salivary glands of alpha-adrenoreceptors, causing dry mouth due to reduced saliva production. \u0000\u0000Conclusion: Hypertension has a specific role as a cause of oral lesion. The use of antihypertension drug has shown the associated with oral mucosa damage.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"33 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-04-07","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"133346988","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-03-03DOI: 10.31829/2641-7456/ahs2020-4(1)-121
T. Field, S. Poling, Shantay Mines, D. Bendell, Connie H. Veazey
Isolation and loneliness have been associated with negative mood states, sleep disturbances and health problems. People experiencing pandemic lockdowns are susceptible to feelings of isolation and loneliness. In this Survey Monkey study conducted during a COVID-19 lockdown (N= 260 respondents), 81% reported feeling isolated and 68% feeling lonely. Correlation analyses suggested that feeling isolated and lonely were related to each other and were, in turn, negatively related to health practices scale scores and positively related to scores on scales measuring COVID-related stress, negative mood states including anxiety and depression, fatigue, sleep disturbances, and posttraumatic stress symptoms. Analyses of variance revealed significant differences between isolated and non-isolated groups as well as between lonely and non-lonely groups on these measures. Only a weak correlation was noted between living alone and loneliness, suggesting that feelings of isolation and loneliness extended to those living with others as well. Positive correlations between both feeling isolated and lonely and time on Facebook and gaming suggested that these activities did not compensate for their isolated and lonely feelings. Feelings of isolation and loneliness and their associated problems also increased across the lockdown survey period. The lack of touch and exercise suggested that those activities might have alleviated the negative feelings and associated problems. The results of this survey are limited by the self-reported data from a non-representative sample that is crosssectional. Nonetheless, they highlight the negative effects of isolation and loneliness during a COVID-19 lockdown
{"title":"Feeling Isolated and Lonely During Covid-19 Lockdown","authors":"T. Field, S. Poling, Shantay Mines, D. Bendell, Connie H. Veazey","doi":"10.31829/2641-7456/ahs2020-4(1)-121","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-121","url":null,"abstract":"Isolation and loneliness have been associated with negative mood states, sleep disturbances and health problems. People experiencing pandemic lockdowns are susceptible to feelings of isolation and loneliness. In this Survey Monkey study conducted during a COVID-19 lockdown (N= 260 respondents), 81% reported feeling isolated and 68% feeling lonely. Correlation analyses suggested that feeling isolated and lonely were related to each other and were, in turn, negatively related to health practices scale scores and positively related to scores on scales measuring COVID-related stress, negative mood states including anxiety and depression, fatigue, sleep disturbances, and posttraumatic stress symptoms. Analyses of variance revealed significant differences between isolated and non-isolated groups as well as between lonely and non-lonely groups on these measures. Only a weak correlation was noted between living alone and loneliness, suggesting that feelings of isolation and loneliness extended to those living with others as well. Positive correlations between both feeling isolated and lonely and time on Facebook and gaming suggested that these activities did not compensate for their isolated and lonely feelings. Feelings of isolation and loneliness and their associated problems also increased across the lockdown survey period. The lack of touch and exercise suggested that those activities might have alleviated the negative feelings and associated problems. The results of this survey are limited by the self-reported data from a non-representative sample that is crosssectional. Nonetheless, they highlight the negative effects of isolation and loneliness during a COVID-19 lockdown","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"25 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-03-03","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"130280531","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-01-30DOI: 10.31829/2641-7456/ahs2020-4(1)-133
C. Pritchard, S. Porter, Richard Williams
Introduction- Health inequalities occur in every society, until a society understands the extent of social injustice drive for change is undermined. Hence this examination of mortality of America’s Ethnic groups. Method: This study’s novel approach analyses differences of 33 mortalities of Asian, Black, Hispanic, Indian/Alaskan and White American people’s Age-Standardised-Death-Rates per million (pm). Based upon National Vital Statistics and comparative ratios are calculated for each ethnicity. Results – Black American had highest death rate 8761pm, Asian Americans lowest 3962pm. Black and Indian/Alaskan Americans had highest rates for most categories, White Americans were highest for nine. Asian and Hispanic Americans substantially lower than other groups. Black Americans had 11 higher and 10 substantially lower than White American deaths. Indian/Alaskan Americans had 10 higher and 7 substantially lower than White Americans. If Black Americans had matched the average mortality of the other groups there would have been 94,422 fewer deaths. Unexpected findings included Black Americans having lower mortalities in specific categories such as neurological disease and some cancer sites. Conclusions: Patterns of mortality strongly suggest links to relative poverty, which are barriers to social justice. While such disparities between the ethnicities remain, they are significant barriers to the pursuit of social justice.
{"title":"A Population-Based Study of 33 Causes of Death amongst America’s Five Ethnic Populations 2015. In Pursuit of Social Justice","authors":"C. Pritchard, S. Porter, Richard Williams","doi":"10.31829/2641-7456/ahs2020-4(1)-133","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-133","url":null,"abstract":"Introduction- Health inequalities occur in every society, until a society understands the extent of social injustice drive for change is undermined. Hence this examination of mortality of America’s Ethnic groups. \u0000\u0000Method: This study’s novel approach analyses differences of 33 mortalities of Asian, Black, Hispanic, Indian/Alaskan and White American people’s Age-Standardised-Death-Rates per million (pm). Based upon National Vital Statistics and comparative ratios are calculated for each ethnicity. \u0000\u0000Results – Black American had highest death rate 8761pm, Asian Americans lowest 3962pm. Black and Indian/Alaskan Americans had highest rates for most categories, White Americans were highest for nine. Asian and Hispanic Americans substantially lower than other groups. Black Americans had 11 higher and 10 substantially lower than White American deaths. Indian/Alaskan Americans had 10 higher and 7 substantially lower than White Americans. If Black Americans had matched the average mortality of the other groups there would have been 94,422 fewer deaths. Unexpected findings included Black Americans having lower mortalities in specific categories such as neurological disease and some cancer sites. \u0000\u0000Conclusions: Patterns of mortality strongly suggest links to relative poverty, which are barriers to social justice. While such disparities between the ethnicities remain, they are significant barriers to the pursuit of social justice.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"94 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-01-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"128382458","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2020-01-30DOI: 10.31829/2641-7456/ahs2020-4(1)-119
Aamir Jalal Al-Mosawi
Background: Academic medical leadership is closely related to scientific research productivity and publication. A researcher’s h-index is based on his/her most cited papers and the number of citations that they have received in other’s publications. It is generally determined by knowing the number of articles written by the author indexed in citation databases. An H-index will be 10 if 10 articles have received at least 10 citations. Ignoring the order and authorship role of an individual researcher may lead to rather a misleading H-index that is totally not relevant to academic leadership determination. The publishing of research conducted by a large collaborative research group made many collaborators with minor role in research creation, development and leadership obtain a high misleading H-index and is not correlated with their academic and research prowess. The use of methods that increase the reliability of the H-index has been increasingly recommended. The aim of this paper is to describe the determination of a more accurate, non-misleading H-index that is more relevant to academic leadership determination. Materials and Methods: An author was found to have an extremely misleading H-index of 28 at Google Scholar citation that is not relevant to academic leadership deterioration. The papers’ citations in his profile were assessed and a corrected rational non-misleading H-index was determined. Results: The author name was not among the first five authors for the first 20 papers listed by Google Scholar Citation, and in most of these papers, his name was not present among the first ten authors. The author name appears among the first three authors in 9 papers (Number 27, 28,29, 36, 41, 43, 45, 47, 50) as listed by Google Scholar Citation. These papers has 34, 30, 21, 10, 5, 5,4,4,3 citations respectively. The author real H-index is 5; because he has at least 5 papers having five citations (Number 27, 28, 29, 36, 41, 43). Conclusion: The corrected H-index should be calculated while considering the papers really authored by an individual author who should be among the first three authors. Many authors who join a large collaborative research group will generally have a minor contribution to research development and publication, but they may achieve a rather misleading high H-index. It is recommended that Google Scholar Citation adopt the corrected H-index to guarantee the reliability and usefulness of the H-index.
{"title":"A Corrected H-Index for Academic Leadership Determination: A Bibliographic Research","authors":"Aamir Jalal Al-Mosawi","doi":"10.31829/2641-7456/ahs2020-4(1)-119","DOIUrl":"https://doi.org/10.31829/2641-7456/ahs2020-4(1)-119","url":null,"abstract":"Background: Academic medical leadership is closely related to scientific research productivity and publication. A researcher’s h-index is based on his/her most cited papers and the number of citations that they have received in other’s publications. It is generally determined by knowing the number of articles written by the author indexed in citation databases. An H-index will be 10 if 10 articles have received at least 10 citations. Ignoring the order and authorship role of an individual researcher may lead to rather a misleading H-index that is totally not relevant to academic leadership determination. The publishing of research conducted by a large collaborative research group made many collaborators with minor role in research creation, development and leadership obtain a high misleading H-index and is not correlated with their academic and research prowess. The use of methods that increase the reliability of the H-index has been increasingly recommended. The aim of this paper is to describe the determination of a more accurate, non-misleading H-index that is more relevant to academic leadership determination. \u0000Materials and Methods: An author was found to have an extremely misleading H-index of 28 at Google Scholar citation that is not relevant to academic leadership deterioration. The papers’ citations in his profile were assessed and a corrected rational non-misleading H-index was determined. \u0000Results: The author name was not among the first five authors for the first 20 papers listed by Google Scholar Citation, and in most of these papers, his name was not present among the first ten authors. The author name appears among the first three authors in 9 papers (Number 27, 28,29, 36, 41, 43, 45, 47, 50) as listed by Google Scholar Citation. These papers has 34, 30, 21, 10, 5, 5,4,4,3 citations respectively. The author real H-index is 5; because he has at least 5 papers having five citations (Number 27, 28, 29, 36, 41, 43). \u0000Conclusion: The corrected H-index should be calculated while considering the papers really authored by an individual author who should be among the first three authors. Many authors who join a large collaborative research group will generally have a minor contribution to research development and publication, but they may achieve a rather misleading high H-index. It is recommended that Google Scholar Citation adopt the corrected H-index to guarantee the reliability and usefulness of the H-index.","PeriodicalId":127914,"journal":{"name":"Archives of Health Science","volume":"120 1","pages":"0"},"PeriodicalIF":0.0,"publicationDate":"2020-01-30","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"123240178","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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