Scandinavian journal of work, environment & health最新文献

Objectives: Sleep supports cognitive performance and recovery, shaping human capital development through education and workplace knowledge application. This study investigates how polygenic indices (PGI) for insomnia (IPGI), short sleep (SSPGI), long sleep (LSPGI), and sleep duration (SDPGI) are associated with educational attainment, occupational group, and income in the Finnish general population.

Methods: Genetic and socioeconomic registry data were merged with pooled data from six pentennial (1992-2017) cohorts representative of Finnish regional populations aged 25-64 (N=20 121). Regression models assessed associations between sleep trait PGI and human capital outcomes. In extended regression models, phenotypic sleep traits were treated as endogenous variables-potentially influenced by unobserved confounders-and instrumented with their respective PGI to isolate variation attributable to genetic predisposition.

Results: IPGI, SSPGI, and LSPGI were substantially negatively associated with educational attainment (P<0.001) and selection into knowledge work occupational group (P≤0.005). Their negative association with income (P<0.005) primarily operated through pathways involving education and occupational group. Extended regression models confirmed that these PGI validly predicted their respective phenotypic sleep traits, which, when instrumented, were significantly negatively associated with education and belonging to the knowledge work occupational group, supporting causal pathways linking genetic sleep predispositions to human capital outcomes via phenotypic sleep traits. In contrast, SDPGI-an aggregate proxy for genetically distinct short and long sleep traits-was not significantly associated with any human capital outcome.

Conclusions: Genetic predispositions to insomnia, short sleep, and long sleep were robustly and substantially negatively associated with human capital development. These associations may help to clarify how genetic sleep traits relate to outcomes in work and health contexts.

Objective: Wearing medical personal protective equipment (PPE) substantially increases heat strain by elevating metabolic heat production while impairing heat dissipation. Cooling vests are a practical countermeasure, yet their efficiency depends on thermal conductivity and comfort. This study examined the thermoregulatory and perceptual responses to PPE use and evaluated the efficacy of a novel carbon-based cooling vest with enhanced heat transfer capacity.

Methods: A randomized crossover design was employed in which 12 participants completed 100 minutes of simulated healthcare activity in a climatic chamber (32 °C, 70% RH) under three conditions: medical scrubs (NoPPE), scrubs with PPE (PPE), and scrubs with PPE plus the cooling vest (PPE+Vest). Physiological, thermoregulatory, and perceptual variables were continuously monitored across conditions.

Results: Compared with PPE alone, PPE+Vest markedly attenuated heat strain, lowering core temperature [PPE 38.4, standard deviation (SD) 0.4, 0C versus PPE+Vest 37.5 (SD 0.4) 0C, P=0.001] and heart rate [PPE 123 (SD 11) bpm versus PPE+Vest 107 (SD 15) bpm, P<0.001], while improving thermal sensation [PPE 2.0 (SD 0.8) versus PPE+Vest 0.8 (SD 0.8), P=0.006]. These thermoregulatory benefits occurred without an increase in metabolic energy expenditure [PPE 317 (SD 50) kcal versus PPE+Vest 317 (SD 53) kcal, P=0.891].

Conclusions: The novel carbon-based cooling vest effectively suppressed heat storage by enhancing conductive heat transfer, leading to core and skin temperatures comparable to NoPPE. Importantly, despite its additional weight, the vest did not impose extra metabolic demands, offering a practical strategy to maintain thermal comfort and physiological stability during prolonged medical work in hot environments.

Objective: This study aimed to investigate the effect of a 12-week multi-component intervention on neck pain among Swiss office workers.

Methods: Between January 2020 and April 2021, we conducted a stepped-wedge cluster randomized controlled trial involving 120 office workers (18-65 years) without severe neck problems from two Swiss companies. Participants started in the control condition and sequentially transitioned to the intervention condition by their cluster. The 12-week intervention included neck exercises, health-promotion workshops, and workplace ergonomics. Neck pain was assessed by intensity [numeric rating scale (NRS) 0=no pain, 10=maximum pain], frequency (days with neck pain in the past 28 days), and disability [Neck Disability Index (NDI) 0%=no disability, 100%=maximum disability). Mixed-effects models were used to assess the intervention's effect on neck pain intensity, frequency, and disability.

Results: This analysis includes 517 observations (295 control, 222 intervention). At baseline, the mean age was 43.7 years [standard deviation years (SD) 9.8], and 71.7% were female. The average neck pain intensity was NRS 2.4 (SD 2.0), frequency 6.8 days (SD 8.0), and disability 11.8% (SD 9.9). A statistically significant effect favoring the multi-component intervention was found for neck pain frequency, with a marginal predicted mean reduction of 1.55 days [95% confidence interval (CI) -2.84--0.26], and neck disability, with a marginal predicted mean reduction of NDI 2.23% (95% CI -2.96--1.68).

Conclusions: This study provides evidence of the effectiveness of a 12-week multi-component intervention in reducing neck pain among office workers. Specifically, office workers experienced neck pain less frequently and with a milder impact on daily activities. Further research is needed to investigate long-term effects.

Objective: This study aimed to examine the association between job strain (combination of job demands and job control) and alcohol-related health problems among men and women and the extent to which workplace social support moderates this association.

Methods: This study used information from the register-based Swedish Work, Illness, and Labor-market Participation (SWIP) cohort, focusing on working individuals born 1945-1975, who were registered in Sweden in 2005 (N=2 822 462). Job demands, control and workplace social support were measured using job exposure matrices (JEM). Information on alcohol-related health problems (morbidity, mortality, medication prescription, sickness absence and disability pension) was obtained from multiple registers between 2006 and 2020. Cox regression models were employed to estimate associations between job strain and alcohol-related health problems, adjusting for sociodemographic background and previous health. The modifying effect of social support was assessed using relative excess risk due to interactions (RERI).

Results: High-strain and passive jobs were related to an increased risk of alcohol-related health problems among both men [hazard ratios (HR) 1.28 and 1.32] and women (HR 1.06 and 1.05), after adjusting for important individual and parental covariates. Weak social support had diverging associations with the outcome for men and women. Strong social support appeared to buffer the risk in passive jobs but only among women (RERI=0.08).

Conclusions: In Sweden, job strain seems to influence alcohol-related health problems, particularly among men. Women, however, appear to experience the protective effects of strong work-related social support measured at the occupational level.

Objectives: We investigated changes in weight, exercise frequency, and perceived health from the first to last health profile assessment (HPA) and between the number of tests within five years. We examined whether sociodemographic factors, or baseline values influenced these changes.

Methods: Data from 106 005 employees with ≥2 HPA (1990-2021) were included. Change between the first and last HPA within a five-year period was analyzed. Baseline age, sex, education, occupation, and baseline values of each outcome were included as predictors. XGBoost models assessed changes in the outcomes, and performance was evaluated via root mean squared error, mean absolute error, and R-squared. We employed Shapley Additive Explanations and forward marginal effects to interpret dose-response relationships and subgroup differences.

Results: Predictive performance was low, suggesting that the included variables only partially explained observed changes. Nonetheless, longer intervals between the first and last HPA correlated with greater weight gain, while a higher number of tests predicted slightly lower weight gain and modest improvements in perceived health and exercise frequency, compared to the average change. Younger participants had larger weight increases, whereas those with higher education showed smaller declines in exercise frequency.

Conclusions: Infrequent HPA alone did not appear to substantially influence the lifestyle-related factors studied. However, more frequent HPA, coupled with enhanced feedback and support, may yield small improvements in weight, perceived health, and exercise frequency compared to the average change.

Objective: With climate change exacerbating occupational heat stress, objective and systematic exposure assessment is essential for epidemiological studies. We developed a job exposure matrix (JEM) to assign occupational heat stress exposure across Europe.

Methods: Aligned with the International Organization for Standardization (ISO: 7243, 8996 and 9920), the heat JEM provides region- and year-specific estimates of annual heat stress hours by job title, using the International Standard Classification of Occupations 1988 for Europe [ISCO-88(COM)]. Heat stress was defined as wet bulb globe temperature effective (WBGT_eff) exceeding WBGT reference (WBGT_ref). Outdoor and indoor WBGT were determined using historical, region-specific hourly meteorological data (temperature, radiation, humidity, wind speed) across Europe, between 1970 and 2024. WBGT values were adjusted for job-specific clothing to obtain WBGT_eff. WBGT_ref was based on metabolic rate, calculated using body surface area and job-specific physical activity, and adjusted for acclimatization status. Further adjustments were made for the job title-specific presence of local heat and cooling sources, time spent indoors versus outdoors, and working schedules.

Results: The number of annual hours workers experience heat stress is highest among jobs involving local heat sources and physical demanding tasks, especially when work clothing is mandatory. Southern Europe has a higher annual heat stress burden compared to other regions. Exposure varies across calendar years and is substantially higher among unacclimatized versus acclimatized workers.

Conclusions: Incorporating job-, region-, and year-specific factors, the heat JEM provides a harmonized tool for studying occupational heat stress. Its transparent framework allows for updates with new data and extensions to other years and regions.

Objectives: Work-related musculoskeletal disorders (MSD) affect over 1.7 billion people globally with a huge economic burden. Despite decades with legislations, policies and risk-reduction interventions, we see no decreases in MSD prevalence. Current prevention models focus on eliminating workplace hazards, overlooking that physical and psychosocial work factors can also promote musculoskeletal health (MSH). We were commissioned through the Danish Working Environment Agreement to develop new approaches addressing this conceptual gap.

Methods: Through iterative stakeholder dialogue with Danish policymakers, social partners, and workplace practitioners, we developed and visualized the CoWork (Copenhagen work-related) MSH model to shift the focus from preventing MSD through risk reduction toward actively promoting work-related MSH. The model aims to bridge theory with workplace structure and implementation by addressing stakeholder requests for clear terminology, conceptual understanding in a workplace context, and actionable guidance.

Results: The CoWork MSH model provides a new definition of work-related MSH as "a state of physical, mental, and social well-being of the locomotor system in relation to work" as well as five integrated elements; (i) a health-oriented approach, (ii) a just-right work factor conceptualization, (iii) the Organizational, Management, Group, !ndividual (OMG!) workplace framework, (iv) an intervention guidance, and (v) health economics perspective. This approach recognizes that work factors can benefit rather than harm health when properly designed and implemented.

Conclusions: The CoWork MSH model represents a paradigm shift, extending from risk reduction to MSH promotion, providing researchers, policymakers, and practitioners with a framework for understanding, researching, and practice to promote workplace MSH.

Objective: The aim of this study was to explore the prospective association between compositions of accelerometry-measured occupational physical behaviors and the risk of knee pain among eldercare workers.

Methods: We performed a prospective study among 377 eldercare workers employed across 20 Danish nursing homes. Occupational physical behaviors were measured using thigh-worn accelerometers over 1-4 working days. Workers reported intensity of and days with knee pain in a questionnaire at baseline and after one year. We explored associations between compositions of occupational physical behaviors [ie, sedentary, standing, light physical activity (LPA), and moderate-to-vigorous physical activity (MVPA)] and knee pain, adjusting for potential confounders.

Results: No significant associations were found. Trends were found for increased occupational time spent in MVPA and decreased risk of days with knee pain [relative risk (RR) 0.58, 95% confidence interval (CI) 0.32-1.05, P=0.07] in main analyses, and for decreased risk of knee pain intensity among non-knee pain cases (RR 0.36, 95% CI 0.12-1.13, P=0.08) in sensitivity analyses.

Conclusions: No significant associations were found between baseline occupational physical behaviors and knee pain at one-year follow-up. However, a non-significant trend suggested that increasing occupational MVPA might be associated with reduced risk of knee pain at follow-up, though studies with larger samples are needed to confirm this finding.

Objectives: Emerging evidence suggests that low-dose ionizing radiation increases the risk of neurodegenerative diseases. Past studies have relied on death data to identify dementia, and these are prone to under-ascertainment and complicate the estimation of health risks as individuals tend to live with dementia for many years following onset. We present findings from the first occupational cohort to investigate dementia risk from low-dose radiation using incident outcomes.

Methods: This is a retrospective cohort of 60 874 Ontario Nuclear Power Plant workers from the Canadian National Dose Registry. Personal identifiers were linked to Ontario population-based administrative health data. Incident dementias between 1996 and 2022 were identified using a validated algorithm based on physician, hospital, and prescription drug data. Individual-level annual estimates of whole-body external ionizing radiation were derived from personal workplace monitoring. The incidence of dementia among these workers was compared to a random sample of Ontario residents matched by sex, age, and residential area. Internal cohort analysis using Poisson and linear excess relative risk (ERR) models, adjusted for sex, attained age, calendar period, and neighborhood income quintile, were used to characterize the shape of the exposure-response curve between low-dose cumulative radiation (lagged 10 years) and incident dementia.

Results: There were 476 incident dementias and 867 028 person-years of follow-up. The mean whole-body lifetime accumulated exposure at the end of follow-up was 11.7 millisieverts (mSv). Workers with cumulative exposure between 50-100 mSv had an increased risk of dementia [RR 1.50, 95% confidence interval (CI) 0.99-2.28] compared to those unexposed. Spline analysis suggested that the dose-response relationship was non-linear. The linear ERR per 100 mSv increase in exposure was 0.704 (95% CI 0.018-1.390).

Conclusion: Our findings suggest that low-dose exposure to ionizing radiation increases the risk of incident dementia.