Journal of Neurorestoratology最新文献

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IF 3.4 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2025-01-01

引用次数: 0

IF 3.4 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2025-01-01

引用次数: 0

IF 3.4 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2025-01-01

引用次数: 0

Advancements in neurodegenerative diseases: Pathogenesis and novel neurorestorative interventions 神经退行性疾病的进展：发病机制和新的神经修复干预措施

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-12-22 DOI: 10.1016/j.jnrt.2024.100176

Wenyong Gao , Shiyuan Jing , Chao He , Hooshang Saberi , Hari Shanker Sharma , Fabin Han , Lin Chen

Progressive neurodegenerative diseases (NDs) that lack effective disease-modifying treatments, including Alzheimer's disease (AD), Parkinson's disease (PD), amyotrophic lateral sclerosis (ALS), and Huntington's disease (HD), represent significant global health challenges. In recent years, key research findings have included the role of neuroinflammation driven by microglia and astrocytes, the impact of genetic mutations, and the importance of autophagy and mitochondrial quality control in maintaining neuronal health. In this review, we summarize recent advancements of the pathogenesis of NDs, the cellular and animal models that have provided valuable insights into disease mechanisms, and the development of blood-based biomarkers for early diagnosis and monitoring of disease progression. We also highlight emerging neurorestorative therapeutic strategies involving stem cell therapy, antisense oligonucleotides, and induced pluripotent stem cells. Additionally, we cover recent clinical trials of promising drugs, such as lecanemab and donanemab for AD, and tavapadon for PD. Finally, we propose future research directions, emphasizing the need for combination therapies that target multiple pathways, the development of more precise animal models, and the integration of nanotechnology for improved drug delivery across the blood–brain barrier.

进行性神经退行性疾病（ndds）缺乏有效的疾病改善治疗，包括阿尔茨海默病（AD）、帕金森病（PD）、肌萎缩侧索硬化症（ALS）和亨廷顿病（HD），是全球健康面临的重大挑战。近年来，主要研究成果包括小胶质细胞和星形胶质细胞驱动的神经炎症的作用、基因突变的影响以及自噬和线粒体质量控制在维持神经元健康中的重要性。在这篇综述中，我们总结了NDs发病机制的最新进展，为疾病机制提供有价值见解的细胞和动物模型，以及用于早期诊断和监测疾病进展的基于血液的生物标志物的发展。我们还强调了新兴的神经修复治疗策略，包括干细胞治疗、反义寡核苷酸和诱导多能干细胞。此外，我们还介绍了最近有希望的药物的临床试验，如治疗AD的lecanemab和donanemab，以及治疗PD的tavapadon。最后，我们提出了未来的研究方向，强调需要针对多种途径的联合治疗，开发更精确的动物模型，以及整合纳米技术来改善血脑屏障的药物传递。

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引用次数: 0

Multimodal MRI and artificial intelligence: Shaping the future of glioma 多模态MRI和人工智能：塑造胶质瘤的未来

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-12-12 DOI: 10.1016/j.jnrt.2024.100175

Yiqin Yan , Chenxi Yang , Wensheng Chen , Zhaoxing Jia , Haiying Zhou , Zhong Di , Longbiao Xu

Gliomas are the most common malignant tumors in the central nervous system and are known for their inherent diversity and propensity to invade surrounding tissue. These features pose significant challenges in diagnosing and treating these tumors. Magnetic resonance imaging (MRI) has not only remained at the forefront of glioma management but has also evolved significantly with the advent of multimodal MRI. The rise of multimodal MRI represents a pivotal leap forward, as it seamlessly integrates diverse MRI sequences and advanced techniques to offer an unprecedented, comprehensive, and multidimensional glimpse into the complexities of glioma pathology, including encompassing structural, functional, and even molecular imaging. This holistic approach empowers clinicians with a deeper understanding of tumor characteristics, enabling more precise diagnoses, tailored treatment strategies, and enhanced monitoring capabilities, ultimately improving patient outcomes. Looking ahead, the integration of artificial intelligence (AI) with MRI data heralds a new era of unparalleled precision in glioma diagnosis and therapy. This integration holds the promise to revolutionize the field, enabling more sophisticated analyses that fully leverage all aspects of multimodal MRI. In summary, with the continuous advancement of multimodal MRI techniques and future deep integrations with artificial intelligence, glioma care is poised to evolve toward increasingly personalized, precise, and efficacious strategies.

胶质瘤是中枢神经系统中最常见的恶性肿瘤，以其固有的多样性和侵犯周围组织的倾向而闻名。这些特征对这些肿瘤的诊断和治疗提出了重大挑战。磁共振成像（MRI）不仅一直处于胶质瘤治疗的前沿，而且随着多模态MRI的出现也有了显著的发展。多模态MRI的兴起代表了一个关键的飞跃，因为它无缝地集成了不同的MRI序列和先进的技术，为胶质瘤病理的复杂性提供了前所未有的，全面的，多维的一瞥，包括包括结构，功能，甚至分子成像。这种整体方法使临床医生能够更深入地了解肿瘤特征，从而实现更精确的诊断，量身定制的治疗策略和增强的监测能力，最终改善患者的治疗效果。展望未来，人工智能（AI）与MRI数据的整合预示着胶质瘤诊断和治疗无与伦比的精确度的新时代。这种整合有望彻底改变该领域，实现更复杂的分析，充分利用多模态MRI的各个方面。综上所述，随着多模态MRI技术的不断进步以及未来与人工智能的深度融合，胶质瘤治疗将朝着越来越个性化、精确和有效的策略发展。

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引用次数: 0

Clinical association of habitual breakfast skipping with cognitive decline and neurodegeneration among older adults 老年人习惯性不吃早餐与认知能力下降和神经退行性变的临床关系

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-11-30 DOI: 10.1016/j.jnrt.2024.100173

Jun Zhang , Ya-Jun Li , Shu Yang , Bing-Hu Li , Duo-Zi Wang , Lei Liu , Jian-Hong Wang

Background

Unhealthy lifestyles have a considerable impact on the incidence of dementia. Skipping breakfast disturbs energy homeostasis and impairs brain function. In this study, we investigated the association between breakfast skipping and cognitive performance among community-dwelling adults.

Methods

We recruited 859 community-dwelling adults aged ≥60 years from January 1 to December 31, 2021. Participants’ sociodemographic information and breakfast skipping habits were self-reported. Participants were followed up for 36 months and cognitive function was assessed using the Mini-Mental State Examination (MMSE) with an interval of 18 months. Trajectories of cognitive change were compared between individuals with and without breakfast skipping. To reduce the risk of bias owing to unmatched sample sizes between the groups, we conducted 1:1 propensity score matching (PSM) based on age, sex, education level, and ApoE genotype.

Results

At baseline and 18-month follow-up, no difference was found in MMSE scores between participants with and without breakfast skipping. However, those who habitually skipped breakfast had significantly lower MMSE scores than those who did not at 36-month follow-up. Individuals with habitual breakfast skipping had a steeper rate of cognitive decline than those without habitual breakfast skipping during follow-up. Breakfast skipping was a risk factor for longitudinal cognitive decline, defined as a decrease in MMSE scores of ≥3, adjusted for age, sex, education, body mass index, ApoE ε4 carrier status, hypertension, diabetes, and hyperlipidemia. At the last follow-up, participants who habitually skipped breakfast had significantly higher levels of ptau181 and NfL than those who did not. In the PSM cohort, similar findings were obtained regarding cognitive trajectories and plasma biomarkers.

Conclusion

Breakfast skipping was linked to an increased risk of long-term cognitive decline and neurodegeneration among older adults. The link between unhealthy dietary habits and cognitive decline may be attributed to a deficiency in neurorestoration resulting from inadequate energy consumption.

健康的生活方式对痴呆症的发病率有相当大的影响。不吃早餐会扰乱能量平衡，损害大脑功能。在这项研究中，我们调查了在社区居住的成年人中不吃早餐与认知表现之间的关系。方法于2021年1月1日至12月31日招募859名年龄≥60岁的社区居民。参与者的社会人口统计信息和不吃早餐的习惯是自我报告的。参与者随访36个月，并使用间隔18个月的迷你精神状态检查（MMSE）评估认知功能。研究人员比较了不吃早餐和不吃早餐的人的认知变化轨迹。为了减少由于组间样本量不匹配而导致的偏倚风险，我们基于年龄、性别、教育水平和ApoE基因型进行了1:1的倾向评分匹配（PSM）。结果在基线和18个月的随访中，不吃早餐和不吃早餐的参与者的MMSE评分没有差异。然而，在36个月的随访中，那些习惯性不吃早餐的人的MMSE得分明显低于不吃早餐的人。在随访期间，习惯性不吃早餐的个体认知能力下降的速度比没有习惯性不吃早餐的个体要快。不吃早餐是纵向认知能力下降的危险因素，定义为MMSE评分≥3，调整年龄、性别、教育程度、体重指数、ApoE ε4携带者状态、高血压、糖尿病和高脂血症。在最后一次随访中，习惯性不吃早餐的参与者的ptau181和NfL水平明显高于不吃早餐的参与者。在PSM队列中，在认知轨迹和血浆生物标志物方面获得了类似的发现。结论：不吃早餐与老年人长期认知能力下降和神经变性的风险增加有关。不健康的饮食习惯和认知能力下降之间的联系可能归因于能量消耗不足导致的神经恢复不足。

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引用次数: 0

Analysis of causal relationship between immune cells and intracranial aneurysm: A mendelian randomization study 免疫细胞与颅内动脉瘤的因果关系分析：孟德尔随机化研究

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-11-15 DOI: 10.1016/j.jnrt.2024.100168

Yang Zhang , Sifei Wang , Yiming Huang , Miaowen Jiang , Baoying Song , Di Wu , Ming Wei , Ming Li , Xunming Ji

Background

Immune cells have been detected in intracranial aneurysms (IAs). However, the causal effect of immune cell phenotypes on IAs remains unclear and difficult to comprehensively analyze.

Methods

Instrumental variables for 731 immunophenotypes were extracted from publicly available genetic databases. The influence of these immune cell traits on IAs was evaluated using the Mendelian randomization (MR) method. Five MR analysis methods, with inverse-variance-weighted as the main method, along with a comprehensive sensitivity analysis, were used to determine reliability of the results. Multivariable MR analysis was performed to correct for interactions between different immune cell phenotypes.

Results

Overall, 27 immune cell traits exhibited significant causal effects on IAs. Among them, 13 immunophenotypes increased the risk of IA progression. Conversely, 14 immune cell characteristics might protect against IAs. Following false discovery rate correction, two hazardous and three protective immunophenotypes remained significant. Moreover, multivariate MR analysis showed that only naive CD4− CD8− T cells %T cells remained causally associated with a risk of IA, while CD19 on IgD+ CD38− naive B cells inhibited development of IAs.

Conclusions

Our study shows that immune cell traits and IAs are causally correlated, providing a new theoretical framework for understanding immune-IA crosstalk.

背景在颅内动脉瘤（IAs）中发现了免疫细胞。方法从公开的遗传数据库中提取了 731 种免疫表型的工具变量。采用孟德尔随机化（MR）方法评估了这些免疫细胞性状对IAs的影响。以反方差加权法为主要方法的五种 MR 分析方法以及全面的敏感性分析被用来确定结果的可靠性。进行了多变量 MR 分析，以校正不同免疫细胞表型之间的相互作用。其中，13种免疫表型增加了IA进展的风险。相反，有 14 种免疫细胞特征可预防原发性心肌梗死。经误诊率校正后，两种危险性免疫表型和三种保护性免疫表型仍具有显著性。此外，多变量 MR 分析表明，只有天真 CD4- CD8- T 细胞 %T 细胞仍与 IA 风险存在因果关系，而 IgD+ CD38- 天真 B 细胞上的 CD19 可抑制 IA 的发展。

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引用次数: 0

Authors’ response to correspondence regarding “Application of deep brain stimulation and transcranial magnetic stimulation in stroke neurorestoration: A review” 作者对有关 "脑深部刺激和经颅磁刺激在中风神经恢复中的应用：综述"

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-10-22 DOI: 10.1016/j.jnrt.2024.100161

Yanxi Chen, Wen Wang, Fangling Sun

引用次数: 0

Response to the Letter from Dr. Li et al. for “Two Sides of One Coin: Neurorestoratology and Neurorehabilitation” 对李博士等人就 "一枚硬币的两面：神经恢复学和神经康复"

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-10-19 DOI: 10.1016/j.jnrt.2024.100160

Hongyun Huang, Hari Shanker Sharma, Lin Chen, Ali Otom, John R. Bach, Wagih S. El Masri

引用次数: 0

Letter to Editor: Correspondence to "Two sides of one coin: Neurorestoratology and Neurorehabilitation" 致编辑的信致 "一枚硬币的两面：神经恢复学和神经康复 "的通信

IF 3.1 4区医学 Q2 CLINICAL NEUROLOGY

Journal of Neurorestoratology

Pub Date : 2024-10-12 DOI: 10.1016/j.jnrt.2024.100159

Xiumin Li, Jie Wang, Yong Liu

引用次数: 0

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