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The Effects of Entry when Monopolistic Competition and Oligopoly Coexist 垄断竞争与寡头垄断并存时的进入效应
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2020-02-01 DOI: 10.1515/bejte-2017-0161
K. Fujiwara
Abstract This paper proposes a model of a continuum of industries in which some industries are monopolistically competitive, the others are oligopolistic, and they interact in a labor market. We use this model to examine the effects of entry of oligopolistic firms. We show that this raises the equilibrium wage and induces exit of monopolistically competitive firms. Then, we find that the profits of each oligopolistic firm and the whole oligopolistic industry decrease. Finally, we establish that if the elasticity of substitution is the same in all industries, welfare improves as a result of an increase in the oligopolistic firms.
摘要本文提出了一个行业连续体的模型,其中一些行业是垄断竞争的,另一些行业是寡头垄断的,它们在劳动力市场中相互作用。我们使用这个模型来检验寡头垄断企业进入的影响。我们证明,这提高了均衡工资,并导致垄断竞争企业退出。然后,我们发现每个寡头垄断企业和整个寡头垄断行业的利润都在下降。最后,我们确定,如果所有行业的替代弹性都相同,那么寡头垄断企业的增加会提高福利。
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引用次数: 0
Product R&D Coopetition and Firm Performance 产品研发合作与企业绩效
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2020-02-01 DOI: 10.1515/bejte-2018-0141
M. Ramsza, A. Karbowski
Abstract We investigate firms’ behavior in demand-enhancing product R&D. We consider and compare a cooperative and non-cooperative R&D investment setting by firms. In a non-cooperative scenario (R&D competition), firms decide on their R&D investments and outputs unilaterally. In a cooperative scenario (R&D coopetition), firms engage in a bargaining process to reach a binding R&D agreement. Firms through bargaining can reach an R&D agreement which specifies their R&D investment levels. The investment levels under R&D coopetition are higher compared with the investment levels under R&D competition. Firms’ profits are also higher under R&D coopetition compared with R&D competition. We conclude that R&D coopetition can alleviate the individual R&D investment disincentive and work as a strategic instrument that enhances product innovation and firms’ profits.
摘要本文研究了企业在需求增长型产品研发中的行为。我们考虑并比较了企业合作与非合作的研发投资环境。在非合作情景(研发竞争)中,企业单方面决定其研发投资和产出。在合作情景(研发合作)中,企业参与议价过程以达成具有约束力的研发协议。企业通过讨价还价可以达成研发协议,明确各自的研发投资水平。研发合作下的投资水平高于研发竞争下的投资水平。研发合作下的企业利润也高于研发竞争下的企业利润。研究发现,研发合作可以缓解个体研发投入的抑制作用,并作为促进产品创新和企业利润的战略工具。
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引用次数: 2
Pretrial Beliefs and Verdict Accuracy: Costly Juror Effort and Free Riding 审前信念与判决准确性:昂贵的陪审员努力和搭便车
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2020-02-01 DOI: 10.1515/BEJTE-2018-0020
Guha Brishti
If jurors care about reaching the correct verdict, but also experience costs to paying attention during the trial, even a small effort cost generates interesting interactions between pretrial beliefs and verdict accuracy. I demonstrate the existence of a strong free riding effect; jurors respond to a more informative prior by reducing their probabilities of paying attention, to the extent that over a non-empty range, a more informative prior will be associated with poorer verdicts. Pretrial beliefs can depend on several factors: I consider two – the extent of discovery during the pre-plea bargaining process, and the efficiency of the police. My results imply that more liberal discovery rules, which result in a less noisy plea bargaining process, will actually be complemented by greater juror effort over a range, resulting in more accurate verdicts. In contrast, greater police efficiency will, over a range, elicit a sufficient drop in juror effort such that verdicts are less accurate. Thus, improving discovery has added benefits over a range, while the benefits of exogenous improvements in policing may be dampened. I briefly extend the model to cases where attentive jurors receive an imperfect public signal instead of a perfect one, and to cases where jurors’ utilities from convicting a guilty defendant differ from their utilities from acquitting an innocent one.
如果陪审员关心做出正确的判决,但也经历了在审判过程中注意的成本,即使是很小的努力成本,也会在审前信念和判决准确性之间产生有趣的相互作用。我证明了搭便车效应的存在;陪审员对信息更丰富的先验的反应是减少他们注意的概率,在一定程度上,在非空范围内,信息更丰富的先验将与较差的判决相关联。审前信念可能取决于几个因素:我认为有两个因素——在辩诉前交易过程中发现证据的程度,以及警方的效率。我的研究结果表明,更自由的证据开示规则,会减少辩诉交易过程的嘈杂,实际上会得到陪审团在一定范围内更大努力的补充,从而产生更准确的判决。相比之下,在一定范围内,更高的警察效率将导致陪审员努力的充分减少,从而使判决不那么准确。因此,改进发现在一定范围内增加了好处,而警务方面的外生改进的好处可能会受到抑制。我简要地将该模型扩展到以下情况:细心的陪审员接收到的是不完美的公共信号,而不是完美的公共信号;以及陪审员判定有罪被告的效用与宣告无罪被告的效用不同的情况。
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引用次数: 1
Flourishing as Productive Tension: Theory and Model 作为生产张力的繁荣:理论与模型
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-11-20 DOI: 10.1515/bejte-2018-0162
A. Heifetz, E. Minelli
Abstract Based on insights from psychology, we argue that individual well-being has two incommensurable dimensions, gratification and flourishing. We provide a model in which the individual’s vitality is tunneled to different goals/practices. Gratification is represented by discharge of flow, while flourishing corresponds to generated power. The two dimensions are correlated in a way that depends on the characteristics of the available goals/practices, and their relationship need not be monotonic. We also extend the model to social interaction, prove existence of an equilibrium and show the possibility of ‘flourishing traps’.
摘要基于心理学的观点,我们认为个体幸福感有两个不可通约的维度,即满足和繁荣。我们提供了一个模型,在这个模型中,个人的活力被挖掘到不同的目标/实践中。感恩代表流量的释放,而繁荣则代表发电量。这两个维度的关联方式取决于可用目标/实践的特征,它们的关系不必是单调的。我们还将模型扩展到社会互动,证明了平衡的存在,并展示了“繁荣陷阱”的可能性。
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引用次数: 0
Behavioral Theory of Repeated Prisoner’s Dilemma: Generous Tit-For-Tat Strategy 屡犯困境的行为理论:慷慨的以牙还牙策略
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-11-20 DOI: 10.1515/BEJTE-2019-0030
Hitoshi Matsushima
Abstract This study investigates infinitely repeated games of a prisoner’s dilemma with additive separability in which the monitoring technology is imperfect and private. Behavioral incentives indicate that a player is not only motivated by pure self-interest but also by social preference such as reciprocity, and that a player often becomes naïve and selects an action randomly due to her cognitive limitation and uncertain psychological mood as well as the strategic complexity caused by monitoring imperfection and private observation. By focusing on generous tit-for-tat strategies, we characterize a behavioral version of Nash equilibrium termed behavioral equilibrium in an accuracy-contingent manner. By eliminating the gap between theory and evidence, we show that not pure self-interest but reciprocity plays a substantial role in motivating a player to make decisions in a sophisticated manner.
摘要本研究调查了具有附加可分性的囚犯困境的无限重复博弈,其中监控技术是不完善的和私有的。行为激励表明,玩家不仅受到纯粹的自身利益的激励,还受到互惠等社会偏好的激励,而且由于认知限制和不确定的心理情绪,以及监控不完善和私人观察造成的战略复杂性,玩家往往变得幼稚,随机选择行动。通过关注慷慨的针锋相对策略,我们以准确性为条件的方式描述了纳什均衡的行为版本,称为行为均衡。通过消除理论和证据之间的差距,我们表明,在激励参与者以复杂的方式做出决策方面,不仅仅是出于自身利益,而是互惠发挥着重要作用。
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引用次数: 3
Capital Concentration and Wage Inequality 资本集中与工资不平等
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-11-15 DOI: 10.1515/BEJTE-2019-0049
Jiancai Pi, Yanwei Fan
This paper analyzes how capital concentration affects wage inequality through the channel of financial frictions. Higher capital concentration can be regarded as the Piketty effect. Under the framework of cost constrained financial frictions, we find that higher capital concentration will expand (resp. narrow down) wage inequality if the asset-liability ratio in the skilled sector is high (resp. low) enough relative to that in the unskilled sector.
本文通过金融摩擦的渠道,分析了资本集中对工资不平等的影响。更高的资本集中度可以看作是皮凯蒂效应。在成本约束的金融摩擦框架下,我们发现资本集中度越高,资本集中度越高。如果技术部门的资产负债率高(如:),则缩小工资不平等。相对于非熟练行业,这一比例足够低。
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引用次数: 2
Synergistic Genetic Interactions between Pkhd1 and Pkd1 Result in an ARPKD-Like Phenotype in Murine Models. Pkhd1 和 Pkd1 之间的协同遗传相互作用导致小鼠模型出现类似 ARPKD 的表型。
IF 13.6 4区 经济学 Q4 ECONOMICS Pub Date : 2019-11-01 Epub Date: 2019-08-19 DOI: 10.1681/ASN.2019020150
Rory J Olson, Katharina Hopp, Harrison Wells, Jessica M Smith, Jessica Furtado, Megan M Constans, Diana L Escobar, Aron M Geurts, Vicente E Torres, Peter C Harris

Background: Autosomal recessive polycystic kidney disease (ARPKD) and autosomal dominant polycystic kidney disease (ADPKD) are genetically distinct, with ADPKD usually caused by the genes PKD1 or PKD2 (encoding polycystin-1 and polycystin-2, respectively) and ARPKD caused by PKHD1 (encoding fibrocystin/polyductin [FPC]). Primary cilia have been considered central to PKD pathogenesis due to protein localization and common cystic phenotypes in syndromic ciliopathies, but their relevance is questioned in the simple PKDs. ARPKD's mild phenotype in murine models versus in humans has hampered investigating its pathogenesis.

Methods: To study the interaction between Pkhd1 and Pkd1, including dosage effects on the phenotype, we generated digenic mouse and rat models and characterized and compared digenic, monogenic, and wild-type phenotypes.

Results: The genetic interaction was synergistic in both species, with digenic animals exhibiting phenotypes of rapidly progressive PKD and early lethality resembling classic ARPKD. Genetic interaction between Pkhd1 and Pkd1 depended on dosage in the digenic murine models, with no significant enhancement of the monogenic phenotype until a threshold of reduced expression at the second locus was breached. Pkhd1 loss did not alter expression, maturation, or localization of the ADPKD polycystin proteins, with no interaction detected between the ARPKD FPC protein and polycystins. RNA-seq analysis in the digenic and monogenic mouse models highlighted the ciliary compartment as a common dysregulated target, with enhanced ciliary expression and length changes in the digenic models.

Conclusions: These data indicate that FPC and the polycystins work independently, with separate disease-causing thresholds; however, a combined protein threshold triggers the synergistic, cystogenic response because of enhanced dysregulation of primary cilia. These insights into pathogenesis highlight possible common therapeutic targets.

背景:常染色体隐性多囊肾病(ARPKD)和常染色体显性多囊肾病(ADPKD)在遗传学上截然不同,ADPKD通常由PKD1或PKD2基因(分别编码多囊卵巢蛋白-1和多囊卵巢蛋白-2)引起,而ARPKD则由PKHD1基因(编码纤维胞浆蛋白/多导蛋白[FPC])引起。原发性纤毛症一直被认为是 PKD 发病机制的核心,因为在综合征纤毛症中,原发性纤毛症的蛋白质定位和常见的囊肿表型都是由原发性纤毛症引起的,但在单纯性 PKD 中,原发性纤毛症的相关性受到了质疑。与人类相比,ARPKD在小鼠模型中的表型较轻,这阻碍了对其发病机制的研究:为了研究 Pkhd1 和 Pkd1 之间的相互作用,包括剂量对表型的影响,我们建立了二基因小鼠和大鼠模型,并对二基因、单基因和野生型表型进行了表征和比较:结果:两种动物的基因相互作用具有协同作用,二基因动物表现出快速进展的PKD和类似典型ARPKD的早期致死表型。在二基因小鼠模型中,Pkhd1和Pkd1之间的遗传相互作用取决于剂量,在第二个基因位点表达量减少的阈值被突破之前,单基因表型不会显著增强。Pkhd1 的缺失并没有改变 ADPKD 多囊蛋白的表达、成熟或定位,也没有检测到 ARPKD FPC 蛋白与多囊蛋白之间的相互作用。在二基因小鼠和单基因小鼠模型中进行的RNA-seq分析突出表明,纤毛区是一个共同的失调靶标,在二基因模型中,纤毛表达增强,长度发生变化:这些数据表明,FPC 和多囊卵巢蛋白各自独立工作,具有不同的致病阈值;然而,由于初级纤毛的失调增强,联合蛋白阈值会触发协同的致囊反应。这些对发病机理的深入了解突显了可能的共同治疗靶点。
{"title":"Synergistic Genetic Interactions between <i>Pkhd1</i> and <i>Pkd1</i> Result in an ARPKD-Like Phenotype in Murine Models.","authors":"Rory J Olson, Katharina Hopp, Harrison Wells, Jessica M Smith, Jessica Furtado, Megan M Constans, Diana L Escobar, Aron M Geurts, Vicente E Torres, Peter C Harris","doi":"10.1681/ASN.2019020150","DOIUrl":"10.1681/ASN.2019020150","url":null,"abstract":"<p><strong>Background: </strong>Autosomal recessive polycystic kidney disease (ARPKD) and autosomal dominant polycystic kidney disease (ADPKD) are genetically distinct, with ADPKD usually caused by the genes <i>PKD1</i> or <i>PKD2</i> (encoding polycystin-1 and polycystin-2, respectively) and ARPKD caused by <i>PKHD1</i> (encoding fibrocystin/polyductin [FPC]). Primary cilia have been considered central to PKD pathogenesis due to protein localization and common cystic phenotypes in syndromic ciliopathies, but their relevance is questioned in the simple PKDs. ARPKD's mild phenotype in murine models versus in humans has hampered investigating its pathogenesis.</p><p><strong>Methods: </strong>To study the interaction between <i>Pkhd1</i> and <i>Pkd1</i>, including dosage effects on the phenotype, we generated digenic mouse and rat models and characterized and compared digenic, monogenic, and wild-type phenotypes.</p><p><strong>Results: </strong>The genetic interaction was synergistic in both species, with digenic animals exhibiting phenotypes of rapidly progressive PKD and early lethality resembling classic ARPKD. Genetic interaction between <i>Pkhd1</i> and <i>Pkd1</i> depended on dosage in the digenic murine models, with no significant enhancement of the monogenic phenotype until a threshold of reduced expression at the second locus was breached. <i>Pkhd1</i> loss did not alter expression, maturation, or localization of the ADPKD polycystin proteins, with no interaction detected between the ARPKD FPC protein and polycystins. RNA-seq analysis in the digenic and monogenic mouse models highlighted the ciliary compartment as a common dysregulated target, with enhanced ciliary expression and length changes in the digenic models.</p><p><strong>Conclusions: </strong>These data indicate that FPC and the polycystins work independently, with separate disease-causing thresholds; however, a combined protein threshold triggers the synergistic, cystogenic response because of enhanced dysregulation of primary cilia. These insights into pathogenesis highlight possible common therapeutic targets.</p>","PeriodicalId":44773,"journal":{"name":"B E Journal of Theoretical Economics","volume":"19 1","pages":"2113-2127"},"PeriodicalIF":13.6,"publicationDate":"2019-11-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6830782/pdf/","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"89310330","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":4,"RegionCategory":"经济学","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Winners and Losers of Universal Health Insurance: A Macroeconomic Analysis 全民健康保险的赢家与输家:宏观经济分析
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-10-22 DOI: 10.1515/bejte-2018-0064
W. Ng, Yin-Chi Wang
Abstract This paper studies the supply-side distortions and the consequences resulted from provider-side cost containing universal health insurance (UHI) scheme. A two-sector overlapping generations model of endogenous physicians’ specialty choice is presented. We find that the general public is possible to be benefited from the cost containing UHI if the quality of medical services does not deteriorate too much. However, physicians in the medical service sector suffer from such scheme and end up earning lower incomes, regardless of one’s specialty and talent. Inequality among physicians also increases.
摘要本文研究了供给侧扭曲和由包含全民健康保险(UHI)计划的提供者侧成本导致的后果。提出了内生医生专业选择的两部门重叠代模型。我们发现,如果医疗服务的质量不恶化太多,一般公众有可能从控制全民医保的成本中受益。然而,医疗服务部门的医生却受到这种制度的影响,最终收入较低,无论他们的专业和才能如何。医生之间的不平等也在加剧。
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引用次数: 0
Vagueness of Language: Indeterminacy under Two-Dimensional State-Uncertainty 语言的模糊性:二维状态不确定性下的不确定性
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-10-22 DOI: 10.1515/bejte-2018-0093
Saori Chiba
Abstract We study indeterminacy of indicative meanings (disagreements about meanings of messages among players), a kind of language vagueness examined in Blume and Board (2013. “Language Barriers.” Econometrica 81 (2): 781–812). They, using a cheap talk model in which the state-distribution and the players’ language competence were ex-ante uncertain, demonstrated that this vagueness occurs as an equilibrium language. We expand the work of Blume and Board by using a model between an uninformed decision maker and an informed agent in which the state-distribution and the state are both ex-ante uncertain. We show that this two-dimensional uncertainty also leads to indeterminacy of indicative meanings, that is, to a set of conditions in which an agent with different perceptions of the state-distribution intentionally uses the same symbol for the different extents of information on the state. The vagueness, contrary to common expectations, can actually lead to welfare improvement.
摘要我们研究了指示意义的不确定性(玩家之间对信息含义的分歧),这是Blume和Board(2013)研究的一种语言模糊性。“语言障碍”,《计量经济学》81(2):781–812)。他们使用了一个廉价的谈话模型,其中状态分布和参与者的语言能力事先是不确定的,证明了这种模糊性是作为一种平衡语言出现的。我们通过使用不知情决策者和知情代理人之间的模型来扩展Blume和Board的工作,在该模型中,州分布和州都是预先不确定的。我们表明,这种二维的不确定性也导致了指示意义的不确定性,也就是说,在一组条件下,对状态分布有不同感知的主体有意使用相同的符号来表示不同程度的状态信息。这种模糊性与普遍预期相反,实际上可以改善福利。
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引用次数: 0
Optimal Incentives Under Gift Exchange 礼品交换下的最优激励
IF 0.4 4区 经济学 Q4 ECONOMICS Pub Date : 2019-10-11 DOI: 10.1515/BEJTE-2018-0041
Khansa Hayat
This paper studies optimal incentives in the presence of an agent’s preference for generosity, where the principal optimally chooses the level of generosity (rent) as well as the performance-based pay. We show that some minimum level of reciprocity is required for generous contracts to become attractive. More reciprocal agents exert greater effort, but they may not necessarily receive more generous offers, as our model predicts a hump-shaped relationship between generosity and the reciprocity parameter. This means that moderately reciprocal agents tend to receive a premium relative to weakly or strongly reciprocal agents. Generosity and performance-based pay are likely to behave as complements when risk-sharing motives (determined by noise in the environment and the agent’s degree of risk-aversion) are strong, and they are likely to behave as substitutes when risk-sharing motives are relatively moderate. Moreover, base-wage gifts are always optimal, whereas piece-rate or composite gifts are only optimal in a low-noise environment.
本文研究了在代理人有慷慨偏好的情况下,委托人最优选择慷慨水平(租金)和绩效薪酬的最优激励。我们表明,慷慨的合同要变得有吸引力,就需要某种最低程度的互惠。更多的互惠行为者会付出更大的努力,但他们不一定会得到更慷慨的报价,因为我们的模型预测了慷慨和互惠参数之间的驼峰形关系。这意味着相对于弱或强互惠的药物,适度互惠的药物往往会获得溢价。当风险分担动机(由环境中的噪音和代理人的风险厌恶程度决定)较强时,慷慨和基于绩效的薪酬可能表现为互补,当风险分担动机相对温和时,它们可能表现为替代。此外,基本工资礼物总是最优的,而计件工资或复合礼物只有在低噪音环境下才是最优的。
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引用次数: 1
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B E Journal of Theoretical Economics
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