Environmental Chemistry and Ecotoxicology最新文献

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IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01

引用次数: 0

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01

引用次数: 0

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01

引用次数: 0

An integrated approach unveils novel mechanisms of nicotine-induced neurotoxicity in zebrafish larvae 一种综合的方法揭示了尼古丁诱导斑马鱼幼虫神经毒性的新机制

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01 DOI: 10.1016/j.enceco.2025.12.014

Yu Kou , Changsheng Guo , Yan Zhang, Yanghui Deng, Xingxing Yin, Jian Xu

Nicotine is a ubiquitous environmental pollutant which may exert neurotoxicity to aquatic organisms. However, the mechanisms at environmentally relevant concentrations remain inadequately understood. This study employed an integrated approach combining in silico predictions, in vivo zebrafish assays, transcriptomics and targeted metabolomics to decipher the mechanistic framework of nicotine-driven developmental neurotoxicity to zebrafish. Network toxicology and molecular docking identified six core targets (CXCR4, CHRNB1, CHRNA1, CHRNA3, STAT3, HIF1A) and predicted key pathways, including neuroactive ligand-receptor interaction and calcium signaling. Experimental validation in zebrafish larvae revealed that nicotine exposure, even at low environmental levels (0.03–3 μg/L), significantly induced developmental delays, heart rate imbalances, and neurobehavioral deficits. Targeted metabolomics demonstrated that nicotine caused a pronounced neurotransmitter imbalance, characterized by a sharp increase in acetylcholine and disrupted levels of dopamine, norepinephrine, and serotonin. Transcriptomics further confirmed the dysregulation of key pathways, including calcium signaling, MAPK inflammation cascade, and neuroactive ligand-receptor interaction. An integrated multi-omics analysis delineated a cohesive adverse outcome pathway: nicotine initially disrupts neuroactive ligand-receptor interactions, leading to intracellular calcium overload, which subsequently triggers MAPK-mediated inflammatory apoptosis and FoxO/p53-related oxidative stress, ultimately resulting in neuronal damage and behavioral dysfunction. Our findings provide novel and comprehensive insights into the mechanistic basis of nicotine-induced neurotoxicity, highlighting significant ecological risks at environmental concentrations and offering a robust framework for assessing the neurotoxic potential of environmental contaminants.

尼古丁是一种普遍存在的环境污染物，对水生生物具有神经毒性。然而，在与环境有关的浓度下的机制仍然没有得到充分了解。本研究采用集成的方法，结合计算机预测、斑马鱼体内实验、转录组学和靶向代谢组学，来破译尼古丁驱动斑马鱼发育神经毒性的机制框架。网络毒理学和分子对接确定了6个核心靶点（CXCR4、CHRNB1、CHRNA1、CHRNA3、STAT3、HIF1A），并预测了关键通路，包括神经活性配体-受体相互作用和钙信号转导。斑马鱼幼鱼的实验验证表明，即使在低环境水平（0.03-3 μg/L）下，尼古丁暴露也会显著诱导发育迟缓、心率失衡和神经行为缺陷。目标代谢组学表明，尼古丁引起明显的神经递质失衡，其特征是乙酰胆碱急剧增加，多巴胺、去甲肾上腺素和血清素水平被破坏。转录组学进一步证实了关键通路的失调，包括钙信号、MAPK炎症级联和神经活性配体-受体相互作用。一项综合多组学分析描绘了一个紧密相关的不良后果途径：尼古丁最初破坏神经活性配体与受体的相互作用，导致细胞内钙超载，随后触发mapk介导的炎症细胞凋亡和FoxO/p53相关的氧化应激，最终导致神经元损伤和行为功能障碍。我们的研究结果为尼古丁诱导神经毒性的机制基础提供了新颖而全面的见解，突出了环境浓度下的重大生态风险，并为评估环境污染物的神经毒性潜力提供了一个强有力的框架。

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引用次数: 0

Male-biased sensitivity to wastewater effluent exposure in Harpagifer antarcticus from Fildes Bay, King George Island 乔治国王岛菲尔德斯湾南极Harpagifer对废水暴露的男性偏敏感性

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01 DOI: 10.1016/j.enceco.2025.12.023

Karen Fabres , Elvira Vergara , Ivan Sola , María José Díaz , Fabio Blanco-Murillo , Fernando Valenzuela , Diana M. Cárdenas , Mark R. Servos , Mario Sanhueza , Gustavo Chiang , Paulina Bahamonde

Antarctica has experienced a growing human presence in recent decades, with year-round scientific and military stations concentrated along the coast. Wastewater effluents from these settlements represent a growing concern, as they contain pharmaceuticals and personal care products (PPCPs) capable of disrupt endocrine functions in aquatic organisms at environmentally relevant concentrations. This study evaluated the impact of wastewater exposure on the gonadal transcriptome of the Antarctic fish Harpagifer antarcticus, collected nearby human settlements and research stations in Fildes Bay, King George Island. Chemical analysis revealed bioaccumulation of diclofenac, carbamazepine, and fluoxetine in fish muscle. Morphological endpoints indicate significant decreases in male gonadosomatic index (GSI) at impacted sites. Molecular analysis suggested disruption of genes related to reproduction. Females displayed significant overexpression of zp at impacted sites, while males from contaminated areas showed increased expression of both zp and esr1.Transcriptomic analysis revealed a male-biased response, with enriched GO terms associated to sperm motility, nervous system function, and immune and inflammatory regulation, as well as chemical homeostasis and lipid metabolism. Females were less impacted, exhibiting transcriptomic alterations mainly linked to metabolic homeostasis. Together, these findings suggest that male H. antarcticus are particularly sensitive to wastewater-derived contaminants, with transcriptional shifts serving as early warning signals of impaired reproduction, physiology, and population resilience. This study highlights the vulnerability of Antarctic coastal ecosystems to anthropogenic pressures and underscores the urgent need for improved wastewater management in this fragile environment.

近几十年来，人类在南极洲的活动越来越多，全年的科学和军事站都集中在沿海地区。这些定居点的废水日益引起人们的关注，因为它们含有能够以环境相关浓度破坏水生生物内分泌功能的药品和个人护理产品（PPCPs）。本研究评估了污水暴露对南极Harpagifer antarcticus鱼性腺转录组的影响，该鱼收集于乔治国王岛菲尔德斯湾附近的人类住区和研究站。化学分析显示双氯芬酸、卡马西平和氟西汀在鱼类肌肉中的生物蓄积。形态学终点显示受影响部位的男性性腺指数（GSI）显著降低。分子分析表明与繁殖有关的基因被破坏。雌性在受影响部位表现出明显的zp过表达，而来自污染区域的雄性则表现出zp和esr1的高表达。转录组学分析揭示了一种男性偏向的反应，与精子活力、神经系统功能、免疫和炎症调节、化学稳态和脂质代谢相关的氧化石墨烯富集。雌性受影响较小，表现出主要与代谢稳态相关的转录组改变。在一起,这些发现表明,男性h . antarcticus strain wastewater-derived污染物尤为敏感,与转录变化作为预警信号受损生殖,生理学以及人口弹性。这项研究强调了南极沿海生态系统对人为压力的脆弱性，并强调了在这一脆弱环境中改善废水管理的迫切需要。

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引用次数: 0

Microplastics in agricultural soils: Impacts on soil microbial communities, nutrient cycling, earthworm health, and soil ecosystem functions 农业土壤中的微塑料：对土壤微生物群落、养分循环、蚯蚓健康和土壤生态系统功能的影响

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01 DOI: 10.1016/j.enceco.2026.01.025

Iqra Aslam , Nabi Bux , Xiaohong Yu , Mengfei Bi , He Guo , Yali Niu , Qiuling Ma , Tiecheng Wang

Global plastic production has increased exponentially since the mid-20th century, leading to the widespread accumulation of microplastics (MPs) in terrestrial ecosystems. Agricultural soils have become major sinks for MPs due to inputs from plastic mulching, sewage sludge, compost, irrigation with reclaimed water, and atmospheric deposition. MPs alter soil physicochemical characteristics, affect nutrient cycling, and disrupt microbial diversity, structure, and enzymatic activities. They also interact with co-occurring pollutants, facilitating the transport and bioavailability of heavy metals and organic contaminants. Earthworms, as key soil engineers, are particularly vulnerable to MP exposure, exhibiting growth inhibition, oxidative stress, intestinal damage, and alterations in gut microbiota composition. Moreover, MPs enhance greenhouse gas emissions by influencing microbial metabolism and soil redox conditions, further impacting ecosystem stability. Human exposure through ingestion, inhalation, and dermal contact raises additional health concerns. This review demonstrates that MPs function as integrated stressors in terrestrial ecosystems, compromising soil health through physicochemical degradation of soil structure, destabilization of microbial communities, and direct toxicity to keystone soil fauna. Impact severity is highly context-dependent, governed by MP properties (size, polymer type), soil characteristics, and exposure duration. Without immediate source control and remediation strategies, continued MP accumulation will irreversibly impair critical soil ecosystem services, threatening agricultural productivity and food security.

自20世纪中叶以来，全球塑料产量呈指数级增长，导致陆地生态系统中微塑料（MPs）的广泛积累。由于塑料覆盖、污水污泥、堆肥、再生水灌溉和大气沉降的投入，农业土壤已成为MPs的主要汇。MPs改变土壤理化特性，影响养分循环，破坏微生物多样性、结构和酶活性。它们还与共存的污染物相互作用，促进重金属和有机污染物的运输和生物利用度。蚯蚓作为关键的土壤工程师，特别容易受到MP的影响，表现出生长抑制、氧化应激、肠道损伤和肠道微生物群组成的改变。此外，MPs通过影响微生物代谢和土壤氧化还原条件增加温室气体排放，进一步影响生态系统的稳定性。人体通过摄入、吸入和皮肤接触接触引起了额外的健康问题。这一综述表明，MPs在陆地生态系统中作为综合应激源，通过土壤结构的理化退化、微生物群落的不稳定和对关键土壤动物的直接毒性来损害土壤健康。影响严重程度与环境高度相关，受MP属性（大小、聚合物类型）、土壤特征和暴露时间的影响。如果没有立即的源头控制和补救策略，持续的MP积累将不可逆转地损害关键的土壤生态系统服务，威胁农业生产力和粮食安全。

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Exploring the cleaning effect of a typhoon on polycyclic aromatic hydrocarbons in the upper-ocean of the Northern South China Sea 探讨台风对南海北部上层海洋多环芳烃的净化作用

IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

Pub Date : 2026-01-01 DOI: 10.1016/j.enceco.2026.01.027

Yicheng Li , Yali Li , Wenbo Wang , Qiong Xia , Ying Yang , Shichun Zou , Weidong Zhai

Typhoons can largely alter hydrodynamic conditions and air-sea exchange processes in the upper ocean, thereby influencing the environmental behavior of organic pollutants, including polycyclic aromatic hydrocarbons (PAHs). Nonetheless, the comprehensive magnitude and mechanisms of these influences remain inadequately characterized. The passage of Typhoon Chaba through the South China Sea (SCS) in 2022 provided a unique opportunity to investigate PAH dynamics in response to typhoons. Field sampling was conducted one week pre-typhoon and one-week post-typhoon. In surface seawater, the total concentration of 15 US EPA priority PAHs (TPAHs) decreased markedly by 45%, from 220 ± 160 ng/L in the pre-typhoon region to 120 ± 42.5 ng/L in the post-typhoon region. Conversely, the water-particle partition coefficient (K_d) increased by 73%. Source apportionment revealed consistent PAHs origins between the two regions, primarily derived from fossil fuel combustion (51%), followed by petroleum leakage (38%), and biomass burning (11%). At a continuous 24-h monitoring site within the post-typhoon region, TPAH concentrations were higher in the surface layer than at the bottom of the upper mixed layer, while the K_d values showed the inverse trend. The observed decline in PAH levels confirms that typhoons promote the removal of these pollutants from the mixed layer. A proposed mechanism involves typhoon-enhanced partitioning of dissolved PAHs (particularly 4-ring compounds) to particulate phase, subsequently removed by particle sinking. The total mass of PAHs removed by the typhoon was estimated to be 703 kg within the upper mixed layer across the 2.65 × 10⁴ km² study area over 24 h. This study provides the first fine-scale elucidation of the cleansing effect of typhoons on organic pollutants, underscoring their critical role in regulating the fate of PAHs in upper ocean.

台风可以在很大程度上改变海洋上层的水动力条件和海气交换过程，从而影响有机污染物的环境行为，包括多环芳烃（PAHs）。尽管如此，这些影响的综合规模和机制仍然没有得到充分的描述。2022年台风“查巴”通过南海为研究台风对多环芳烃的响应提供了一个独特的机会。在台风前一周和台风后一周分别进行了实地抽样。在表层海水中，15种US EPA优先多环芳烃（TPAHs）的总浓度从台风前的220±160 ng/L下降到台风后的120±42.5 ng/L，显著下降了45%。相反，水颗粒分配系数（Kd）增加了73%。多环芳烃的来源一致，主要来源于化石燃料燃烧（51%），其次是石油泄漏（38%）和生物质燃烧（11%）。在台风后区域内连续24 h监测点，表层TPAH浓度高于上层混合层底部，而Kd值呈相反趋势。观测到的多环芳烃水平下降证实，台风促进了这些污染物从混合层中去除。提出的机制涉及台风增强溶解的多环芳烃（特别是4环化合物）到颗粒相的分配，随后通过颗粒沉降去除。在2.65 × 104 km2的研究区域内，台风在24 h内清除上层混合层中多环芳烃的总质量估计为703 kg。该研究首次在精细尺度上阐明了台风对有机污染物的净化作用，强调了台风在调节上层海洋中多环芳烃的作用。

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IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

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IF 8.2 Q1 ENVIRONMENTAL SCIENCES

Environmental Chemistry and Ecotoxicology

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Environmental Chemistry and Ecotoxicology

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