Psychoneuroendocrinology最新文献

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When and why does EMA go MIA?: A mixed-method exploration of participant engagement with app-supported data collection

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107356

S. Dockray , L. Maye

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Women's domain-specific affiliation across the menstrual cycle

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107372

K.E. Phimmasene, A. Makhanova

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Presentation 3: Habitual caffeine use moderates relationships of sleep duration and depression symptoms to cortisol awakening response

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107297

S.M. Vrshek-Schallhorn, E.L. Cole, C.B. Crayton

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Software tools to advance open and reproducible Science in PNE research

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107292

R. Richer , B.M. Eskofier, N. Rohleder

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Salivary cortisol, protein concentrations, and total salivation following the socially evaluative cold pressor test

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107341

J. Hamza , K. Jansakova , J. Rajcani

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Salivary IL-6 moderates the effects of ethnic social rejection and exclusion on symptoms of depression and social anxiety among LGBTQ and Straight Latinx emerging adults

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107342

L.A. Parra , A.N. Bell , P.D. Hastings

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Hatha yoga reduces momentary stress but does not impact diurnal profiles of salivary cortisol and alpha-amylase: a randomized controlled trial

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107327

B. Szaszkó , H. Tschenett , U. Ansorge , U.M. Nater

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Presentation 2: Analyzing temperature effects on mitochondrial bioenergetics in peripheral blood mononuclear cells from individuals with and without major depressive disorder: an empirical contribution to the debate on mitochondrial dysfunction

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2025.107300

A. Karabatsiakis , K. de Punder , F. Scholkmann , L. Toscano , E. Guenova , L. Holper

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Memory under pressure: The impact of acute stress across different memory tasks 压力下的记忆：急性压力对不同记忆任务的影响。

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2024.107246

Selen Soylu , Robert Miller , Maximilian Pilhatsch , Tanja Endrass , Lisa Weckesser

In the present study, we aimed to investigate how acute stress exerts its heterogeneous effects. Based on biophysical network models, we hypothesized that acute stress would improve occipital-mediated ultra-short-term and to a lesser degree affect occipital- and frontal-mediated short-term and working, and impairs hippocampal-mediated long-term memory processes and their respective behavioral measures. To test this, 111 healthy individuals (57 female) underwent both the Trier Social Stress Test (TSST) and a control test. Immediately afterward, participants’ performance was measured in four memory tasks (Rapid Serial Visual Presentation, RSVP, Match-to-Sample, MTS, N-Back, NB and Free-Recall, FR tasks). TSST exposure seems to impair long-term memory (AC_FR; β = −1.50 ± 0.62; when free recall was tested approx. 80 minutes after initial encoding, immediately after the TSST), and working memory (AC_NB; β= −0.42 ± 0.20 %) but did not affect ultra-short-term (AC_RSVP; β = −0.03 ± 0.31 %) and short-term (AC_MTS; β=-0.18 ± 0.31 %) memory accuracies (ACs). Interestingly, TSST exposure increased the exploratory included measure of response times in MTS (RT_MTS; β =16.42 ± 7.18 msec) and impaired T1 detection in the RSVP (AC_T1; β=-0.48 ± 0.22 %) tasks. Contrary to the hypothesis, TSST exposure did not have the hypothesized effects on the memory processes. Instead, TSST exposure appeared to affect secondary behavioral indicators of motivation or task instruction adherence.

在本研究中，我们旨在探讨急性应激如何发挥其异质性效应。基于生物物理网络模型，我们假设急性应激会改善枕部介导的超短期记忆，并在较小程度上影响枕部和额部介导的短期和工作记忆，并损害海马介导的长期记忆过程及其相应的行为测量。为了验证这一点，111名健康个体（57名女性）接受了特里尔社会压力测试（TSST）和对照测试。紧接着，参与者在四个记忆任务（快速连续视觉呈现，RSVP，匹配样本，MTS, N-Back， NB和自由回忆，FR任务）中表现被测量。TSST暴露似乎损害长期记忆(ACFR；β = -1.50±0.62；当自由回忆测试大约。80 初始编码后分钟，紧接TSST)，工作记忆(ACNB；β= -0.42±0.20 %)，但不影响超短期(ACRSVP；β = -0.03±0.31 %)和短期(ACMTS；β=-0.18±0.31 %)记忆精度（ACs）。有趣的是，TSST暴露增加了MTS的探索性反应时间测量(RTMTS；β =16.42±7.18 msec)， RSVP中T1检测受损(ACT1；β=-0.48±0.22 %)任务。与假设相反，TSST暴露对记忆过程没有假设的影响。相反，TSST暴露似乎会影响动机或任务指示依从性的次要行为指标。

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引用次数: 0

Salivary cortisol and affective responses to acute psychosocial stress among adolescents 唾液皮质醇和青少年对急性社会心理压力的情感反应。

IF 3.4 2区医学 Q2 ENDOCRINOLOGY & METABOLISM

Psychoneuroendocrinology

Pub Date : 2025-02-01 DOI: 10.1016/j.psyneuen.2024.107265

Haley Dveirin , Victoria Acuna , Mai-Lan Tran , Elizabeth E. Antici , Kate Ryan Kuhlman

Background

Acute psychosocial stress activates the hypothalamic-pituitary-adrenal (HPA) axis and triggers the release of cortisol, a commonly used biomarker of stress reactivity. Yet only 25 % of studies have reported a correlation between cortisol and affective responses to stress. This study aimed to examine whether cortisol reactivity following an acute psychosocial stressor in the laboratory correlated with concurrent positive and negative affect in adolescents, and whether early life adversity (ELA) moderated this relationship.

Methods

The current study examined the salivary cortisol response of 89 adolescents (46.1 % female) following administration of the Trier Social Stress Test for Children (TSST-C). Using 7 simultaneous measurements, changes in cortisol were compared to changes in concurrent affect using the Positive and Negative Affect Schedule (PANAS). Parents reported their child’s exposure to ELA.

Results

Within-person variability in cortisol was associated with higher negative affect at baseline, b = 1.43 (SE =.41), p < .001, as well as more rapid negative affective recovery following stress, b = -0.003 (SE =.002), p = .04. ELA-exposed participants were not more sensitive to this effect. Within-person variability in cortisol was not associated with positive affect at baseline or following the TSST-C on average, all ps > .27. However, within-person increases in cortisol were associated with decrements in positive affect among ELA-exposed individuals, all ps < .01, suggesting differential sensitivity to anhedonic effects of glucocorticoids.

Conclusions

Stress impacts affective states, in part through acute increases in HPA axis activity. The present data show that negative affect is more vulnerable to this than positive affect among adolescents. Further, adolescents with high ELA may be more vulnerable to decrements in positive affect in the acute aftermath of HPA axis activation. Whether this is a modifiable source of vulnerability to stress-related disease in this high-risk population remains to be understood.

背景：急性社会心理压力会激活下丘脑-垂体-肾上腺（HPA）轴，并引发皮质醇的释放，而皮质醇是压力反应性的常用生物标志物。然而，仅有 25% 的研究报告称皮质醇与对压力的情感反应之间存在相关性。本研究旨在探讨在实验室中受到急性社会心理压力后皮质醇的反应性是否与青少年同时出现的积极和消极情绪相关，以及早期生活逆境（ELA）是否会调节这种关系：本研究检测了89名青少年（46.1%为女性）在接受特里尔儿童社会压力测试（TSST-C）后的唾液皮质醇反应。通过 7 次同步测量，皮质醇的变化与使用积极和消极情绪表 (PANAS) 的并发情绪变化进行了比较。家长报告了他们孩子接触英语语言学习的情况：结果：皮质醇在人体内的变化与基线时较高的负面情绪有关，b = 1.43 (SE =.41), p .27。然而，皮质醇在人体内的增加与暴露于 ELA 的人的积极情绪下降有关，所有 ps 结论：压力会影响情绪状态，部分原因是 HPA 轴活动的急性增加。目前的数据显示，在青少年中，消极情绪比积极情绪更容易受到影响。此外，高 ELA 青少年可能更容易在 HPA 轴激活后的急性期出现积极情绪的下降。在这一高风险人群中，这是否是易患压力相关疾病的一个可改变的来源，还有待进一步了解。

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