{"title":"Incidence of depression and suicide rate in Mexico: An observational analysis of databases from the past years","authors":"J. C. González-Orozco, Brenda M. López-López","doi":"10.24875/rmn.22000084","DOIUrl":"https://doi.org/10.24875/rmn.22000084","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-07-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46701976","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Raul Medina-Rioja, S. Saldívar-Dávila, I. Reyes-Cortés, M. García‐Grimshaw
{"title":"Beyond the hammer: reflections on neurology residency in Mexico","authors":"Raul Medina-Rioja, S. Saldívar-Dávila, I. Reyes-Cortés, M. García‐Grimshaw","doi":"10.24875/rmn.23000029","DOIUrl":"https://doi.org/10.24875/rmn.23000029","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-07-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46147112","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Katherine S. Hernandez-Cortés, Adrián A. Mesa-Pujals, María Sagaró del Campo-Nelsa, Pedrón Montoya-Arquímedes
{"title":"Tables of percentiles of the third ventricle according to age and sex associated with brain aging","authors":"Katherine S. Hernandez-Cortés, Adrián A. Mesa-Pujals, María Sagaró del Campo-Nelsa, Pedrón Montoya-Arquímedes","doi":"10.24875/rmn.23000009","DOIUrl":"https://doi.org/10.24875/rmn.23000009","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-06-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46936573","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Antifragility, rest, and low back pain","authors":"P. Guraieb-Chahín, Adriana Cabrera-Perdomo","doi":"10.24875/rmn.23000004","DOIUrl":"https://doi.org/10.24875/rmn.23000004","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-06-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46038900","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
J. A. Violante-Villanueva, Moisés E. Lázaro-Jarquín, J. Ramírez-Bermúdez, Victoria Martínez-Ángeles, J. Galnares-Olalde, K. Carrillo-Loza, Arturo Martínez-Piña, Xiomara García Salazar, J. C. López-Hernández
Background: Delirium is an important cause of morbidity and mortality in the general hospitalized population. Few information exists on risk factors in patients with neurological disease. Objective: This study was to analyze risk factors for delirium in patients presenting to a neurological emergency department. Methods: This study was observational prospective cohort study. We included all patients with neurological disease, admitted to the emergency department with a stay of more than 8 h, in the period from January to April 2022. The confusion assessment method-intensive care unit delirium detection instrument and DSM-5 criteria were applied to all patients. We obtained information on epidemiological, clinical, and paraclinical variables at the time of admission. Through a univariate and multivariate logistic regression model, we analyzed risk factors for delirium. Results: Of 3661 patients treated in the emergency department, 189 patients were included, 60.8% male gender, age 48 ± 19 years; 44 (23.2%) met criteria for delirium, 81% were diagnosed on admission; delirium scale score/index 12 (interquartile range [IQR] 10-12), and duration of delirium 5 (IQR 2-11) days. Clinical history for risk of delirium: previous delirium (odds ratio [OR] 3.9, 95% CI 2.4-6.1, p = 0.003), stroke (OR 2.2, 95% CI 1.4-3.9, p = 0.009), neurosurgery (OR 2.1, 95% CI 1.2-3.8, p = 0.024), and use of psychoactive drugs (OR 2.5, CI9 5% 1.3-4.6, p = 0.021). The following variables were independent risk factors for delirium through the multivariate logistic regression model:uncontrolled crisis [OR 5.4 (95% CI 1.2-22.9), p = 0.023], supratentorial structural brain lesion [OR 6.1 (95% CI 1.7-21.2), p = 0.004], and neuroinfection OR 9.6 (95% CI 2.9-31.4), p ≤ 0.001]. Conclusions: The frequency of delirium is 23.2% in patients presenting to the neurological emergency department; uncontrolled epilepsy, neuroinfection, and supratentorial brain lesions are independent risk factors for delirium.
{"title":"Frequency of delirium and associated factors in patients from a neurological emergency department","authors":"J. A. Violante-Villanueva, Moisés E. Lázaro-Jarquín, J. Ramírez-Bermúdez, Victoria Martínez-Ángeles, J. Galnares-Olalde, K. Carrillo-Loza, Arturo Martínez-Piña, Xiomara García Salazar, J. C. López-Hernández","doi":"10.24875/rmn.22000083","DOIUrl":"https://doi.org/10.24875/rmn.22000083","url":null,"abstract":"Background: Delirium is an important cause of morbidity and mortality in the general hospitalized population. Few information exists on risk factors in patients with neurological disease. Objective: This study was to analyze risk factors for delirium in patients presenting to a neurological emergency department. Methods: This study was observational prospective cohort study. We included all patients with neurological disease, admitted to the emergency department with a stay of more than 8 h, in the period from January to April 2022. The confusion assessment method-intensive care unit delirium detection instrument and DSM-5 criteria were applied to all patients. We obtained information on epidemiological, clinical, and paraclinical variables at the time of admission. Through a univariate and multivariate logistic regression model, we analyzed risk factors for delirium. Results: Of 3661 patients treated in the emergency department, 189 patients were included, 60.8% male gender, age 48 ± 19 years; 44 (23.2%) met criteria for delirium, 81% were diagnosed on admission; delirium scale score/index 12 (interquartile range [IQR] 10-12), and duration of delirium 5 (IQR 2-11) days. Clinical history for risk of delirium: previous delirium (odds ratio [OR] 3.9, 95% CI 2.4-6.1, p = 0.003), stroke (OR 2.2, 95% CI 1.4-3.9, p = 0.009), neurosurgery (OR 2.1, 95% CI 1.2-3.8, p = 0.024), and use of psychoactive drugs (OR 2.5, CI9 5% 1.3-4.6, p = 0.021). The following variables were independent risk factors for delirium through the multivariate logistic regression model:uncontrolled crisis [OR 5.4 (95% CI 1.2-22.9), p = 0.023], supratentorial structural brain lesion [OR 6.1 (95% CI 1.7-21.2), p = 0.004], and neuroinfection OR 9.6 (95% CI 2.9-31.4), p ≤ 0.001]. Conclusions: The frequency of delirium is 23.2% in patients presenting to the neurological emergency department; uncontrolled epilepsy, neuroinfection, and supratentorial brain lesions are independent risk factors for delirium.","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46031180","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Aldair Ochoa-Rojas, A. Calderón-Garcidueñas, César I. PenÓa-Ruelas, R. Ruiz-Ramos, Noé López-Amador, E. Villarreal-Ríos, E. Castelán-Maldonado
Objective: To investigate the relationship between mast cells (MCs) and different central nervous system (CNS) tumors. Methods : It was a comparative immunohistochemical study to investigate the presence, number, and location of MCs in pilocytic astrocytomas, glioblastoma, medulloblastoma, ependymoma, meningioma, and immature and mature terato-mas. Means, medians, and standard deviation were obtained. Comparation in the number of MCs in the different tumors was carried out using the Mann–Whitney test (p < 0.05). Results: There was a significant difference in the number of MCs between pilocytic astrocytoma ( X ̅ = 0), and mature teratoma ( X ̅ = 65), in relation to the other neoplasms ( X ̅ = 3-11.4). MCs were identified in meningeal lining in meningiomas, in the perivascular space in ependymomas and in the tumor stroma and perivascular space in the rest of the tumors analyzed. Conclusions: MCs appear to play different roles in CNS tumors. Its absence in pilocytic astrocytomas and its presence in glioblastomas suggests a role in the latter, probably related to angiogenesis. The maximum number of MCs was observed in mature teratomas, specifically in relation to stratified squamous epithelium, possibly in relation to trophic factors that contribute to epithelial renewal and maintenance. These findings call for future research to determine if MCs can be a therapeutic target or are important as a prognostic factor.
{"title":"Mast cells in central nervous system neoplasms: an immunohistochemical study","authors":"Aldair Ochoa-Rojas, A. Calderón-Garcidueñas, César I. PenÓa-Ruelas, R. Ruiz-Ramos, Noé López-Amador, E. Villarreal-Ríos, E. Castelán-Maldonado","doi":"10.24875/rmn.22000085","DOIUrl":"https://doi.org/10.24875/rmn.22000085","url":null,"abstract":"Objective: To investigate the relationship between mast cells (MCs) and different central nervous system (CNS) tumors. Methods : It was a comparative immunohistochemical study to investigate the presence, number, and location of MCs in pilocytic astrocytomas, glioblastoma, medulloblastoma, ependymoma, meningioma, and immature and mature terato-mas. Means, medians, and standard deviation were obtained. Comparation in the number of MCs in the different tumors was carried out using the Mann–Whitney test (p < 0.05). Results: There was a significant difference in the number of MCs between pilocytic astrocytoma ( X ̅ = 0), and mature teratoma ( X ̅ = 65), in relation to the other neoplasms ( X ̅ = 3-11.4). MCs were identified in meningeal lining in meningiomas, in the perivascular space in ependymomas and in the tumor stroma and perivascular space in the rest of the tumors analyzed. Conclusions: MCs appear to play different roles in CNS tumors. Its absence in pilocytic astrocytomas and its presence in glioblastomas suggests a role in the latter, probably related to angiogenesis. The maximum number of MCs was observed in mature teratomas, specifically in relation to stratified squamous epithelium, possibly in relation to trophic factors that contribute to epithelial renewal and maintenance. These findings call for future research to determine if MCs can be a therapeutic target or are important as a prognostic factor.","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"46210935","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
C. F. Aguilar-Gamas, E. Martínez-Abundis, N. Gómez-Crisóstomo, E. de la cruz-Hernandez
Neurodegenerative diseases (ND) are undoubtedly part of the more important health issues worldwide. The loss of independence and decreased quality of life are characteristics of these patients that translate into an economic load on relatives and health services. Although myriad factors or metabolic alterations are blamed as causatives, there is a common defect in almost every ND, mitochondrial dysfunction. Mitochondria are the cell’s leading energy supplier; therefore, altering its function will affect the cellular function with the energy demand. Defects in mitochondrial mechanisms such as ATP synthesis by the mitochondrial respiratory chain, mitochondrial dynamics, apoptosis regulation, and oxidative stress are described as part of the pathological characteristics of Alzheimer’s, Parkinson’s, Huntington’s diseases, and Amyotrophic Lateral Sclerosis. In this review, we will briefly describe the mitochondrial roles in cell life before analyzing the recently published bibliography that addresses the mitochondrial dysfunction and mitochondrial mechanisms implicated in ND and will discuss a relevant question in the field: is mitochondrial dysfunction a cause or consequence for these diseases?
{"title":"Relevance of mitochondrial alterations in the neurodegenerative diseases, an update and discussion of evidence","authors":"C. F. Aguilar-Gamas, E. Martínez-Abundis, N. Gómez-Crisóstomo, E. de la cruz-Hernandez","doi":"10.24875/rmn.23000006","DOIUrl":"https://doi.org/10.24875/rmn.23000006","url":null,"abstract":"Neurodegenerative diseases (ND) are undoubtedly part of the more important health issues worldwide. The loss of independence and decreased quality of life are characteristics of these patients that translate into an economic load on relatives and health services. Although myriad factors or metabolic alterations are blamed as causatives, there is a common defect in almost every ND, mitochondrial dysfunction. Mitochondria are the cell’s leading energy supplier; therefore, altering its function will affect the cellular function with the energy demand. Defects in mitochondrial mechanisms such as ATP synthesis by the mitochondrial respiratory chain, mitochondrial dynamics, apoptosis regulation, and oxidative stress are described as part of the pathological characteristics of Alzheimer’s, Parkinson’s, Huntington’s diseases, and Amyotrophic Lateral Sclerosis. In this review, we will briefly describe the mitochondrial roles in cell life before analyzing the recently published bibliography that addresses the mitochondrial dysfunction and mitochondrial mechanisms implicated in ND and will discuss a relevant question in the field: is mitochondrial dysfunction a cause or consequence for these diseases?","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"44187586","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Angelica M. Palacio-Delgado, Miguel B. Cervera-Sánchez, Anna C. Selvas-Cortinas, Samuel A. Romo-Márquez, J. M. Dueñas-Jiménez
The pandemic caused by the SARS-COV-2 virus in the years 2020-2022 altered the emotional behavior of humans. The restrictive feeling caused by the isolation and the change of life habits that demanded a social separation promoted anxiety, distress, apathy, domestic violence, educational problems, and economic instability, among other aspects. Erroneous statistics, and social media information about the number of people infected by the coronavirus SARS-COV-2, aggravated human anxiety and depression. This review compares the main psychological effects caused by pandemic isolation compared to other isolated social contexts. We studied the primary central nervous system areas involved in human reactive aggressiveness behavior. We examine this behavior in relationship with catecholamines and hormones during social isolation. We do not measure or analyze any hormone in our laboratory and only describe the circuits involved in the neuroendocrine response to the aggressive behavior. (English) [ FROM AUTHOR] La pandemia causada por el virus Sars-Cov-2 durante los años 2020 a 2022, alteró la conducta emocional de los humanos. El sentimiento de restricción causado por el aislamiento y el cambio de hábitos de vida que demandaron una separación social promovieron: ansiedad, estrés, apatía, violencia doméstica, problemas educativos e inestabilidad económica, entre otros aspectos. Estadísticas erróneas y de los medios de información acerca del número de personas infectadas por el Coronavirus Sars-Cov-2 agravaron la ansiedad y la depresión humana. Esta revisión compara los principales efectos psicológicos causados por el aislamiento durante la pandemia comparado con otros contextos de aislamiento social. Nosotros estudiamos las áreas primarias involucradas en la conducta de agresión reactiva en los humanos y analizamos esta conducta en relación con las catecolaminas y hormonas durante el aislamiento social. No medimos ni analizamos ninguna hormona en nuestro laboratorio solo describimos los circuitos involucrados en la respuesta neuroendocrina a la conducta agresiva. (Spanish) [ FROM AUTHOR] Copyright of Revista Mexicana de Neurociencia is the property of Academia Mexicana de Neurologia and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full . (Copyright applies to all s.)
2020-2022年由SARS-COV-2病毒引起的大流行改变了人类的情感行为。由于孤立和生活习惯的改变而导致的限制感导致了焦虑、痛苦、冷漠、家庭暴力、教育问题和经济不稳定，以及其他方面。关于冠状病毒SARS-COV-2感染人数的错误统计数据和社交媒体信息加剧了人们的焦虑和抑郁。本综述比较了大流行隔离与其他孤立社会环境造成的主要心理影响。我们研究了涉及人类反应性攻击行为的初级中枢神经系统区域。我们研究了这种行为在社会隔离期间与儿茶酚胺和激素的关系。在我们的实验室里，我们不测量或分析任何激素，只描述与神经内分泌反应有关的回路。(中文)[来自作者]大流行致病病毒Sars-Cov-2 durante los años 2020 a 2022, alteró La conduta emotional de los humanos。《社会促进者》、《社会促进者》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》、《社会问题与社会发展》等。Estadísticas erróneas y de los medios de información acerca del número de personas influectadas el冠状病毒Sars-Cov-2 agravaron由la depresión humana分析。Esta revisión比较的原则影响psicológicos原因贫穷的伊斯兰教和持续的大流行比较的背景下伊斯兰教和社会。Nosotros estudiamos las áreas primarias involucradas en la conductde agresión反应性的unactivactias en los人类通过分析性的unactivactias conducten relación将catecolaminas通过激素持续的el - islamiito social。在神经实验室中，没有任何媒介可以分析神经性激素，单独描述神经性神经回路、神经内分泌和神经传导的变化。(西班牙语)[来自作者]Revista Mexicana de Neurociencia版权归墨西哥神经科学院所有，未经版权所有者明确书面许可，不得将其内容复制或通过电子邮件发送到多个网站或发布到listserv。但是，用户可以打印、下载或通过电子邮件发送文章供个人使用。这可以删节。对副本的准确性不作任何保证。用户应参阅原始出版版本的材料的完整。(版权适用于所有人。)
{"title":"The psychoneuroendocrine response of aggression due to COVID-19 social isolation","authors":"Angelica M. Palacio-Delgado, Miguel B. Cervera-Sánchez, Anna C. Selvas-Cortinas, Samuel A. Romo-Márquez, J. M. Dueñas-Jiménez","doi":"10.24875/rmn.22000066","DOIUrl":"https://doi.org/10.24875/rmn.22000066","url":null,"abstract":"The pandemic caused by the SARS-COV-2 virus in the years 2020-2022 altered the emotional behavior of humans. The restrictive feeling caused by the isolation and the change of life habits that demanded a social separation promoted anxiety, distress, apathy, domestic violence, educational problems, and economic instability, among other aspects. Erroneous statistics, and social media information about the number of people infected by the coronavirus SARS-COV-2, aggravated human anxiety and depression. This review compares the main psychological effects caused by pandemic isolation compared to other isolated social contexts. We studied the primary central nervous system areas involved in human reactive aggressiveness behavior. We examine this behavior in relationship with catecholamines and hormones during social isolation. We do not measure or analyze any hormone in our laboratory and only describe the circuits involved in the neuroendocrine response to the aggressive behavior. (English) [ FROM AUTHOR] La pandemia causada por el virus Sars-Cov-2 durante los años 2020 a 2022, alteró la conducta emocional de los humanos. El sentimiento de restricción causado por el aislamiento y el cambio de hábitos de vida que demandaron una separación social promovieron: ansiedad, estrés, apatía, violencia doméstica, problemas educativos e inestabilidad económica, entre otros aspectos. Estadísticas erróneas y de los medios de información acerca del número de personas infectadas por el Coronavirus Sars-Cov-2 agravaron la ansiedad y la depresión humana. Esta revisión compara los principales efectos psicológicos causados por el aislamiento durante la pandemia comparado con otros contextos de aislamiento social. Nosotros estudiamos las áreas primarias involucradas en la conducta de agresión reactiva en los humanos y analizamos esta conducta en relación con las catecolaminas y hormonas durante el aislamiento social. No medimos ni analizamos ninguna hormona en nuestro laboratorio solo describimos los circuitos involucrados en la respuesta neuroendocrina a la conducta agresiva. (Spanish) [ FROM AUTHOR] Copyright of Revista Mexicana de Neurociencia is the property of Academia Mexicana de Neurologia and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full . (Copyright applies to all s.)","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"45958208","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
D. A. Martínez-Piña, Victor Medina-Gómez, José F. García-Hernández, E. S. Vargas-Cañas, Arturo Violante-Villanueva, J. C. López-Hernández
{"title":"Guillain-Barre syndrome with hyponatremia, more than a simple finding","authors":"D. A. Martínez-Piña, Victor Medina-Gómez, José F. García-Hernández, E. S. Vargas-Cañas, Arturo Violante-Villanueva, J. C. López-Hernández","doi":"10.24875/rmn.22000081","DOIUrl":"https://doi.org/10.24875/rmn.22000081","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"41516709","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
{"title":"Clinical markers of functional outcomes in cognition","authors":"M. Barboza","doi":"10.24875/rmn.m23000095","DOIUrl":"https://doi.org/10.24875/rmn.m23000095","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"48406144","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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