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Revista Mexicana de Neurociencia最新文献

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Cognitive reserve to modify the trajectory of dementia: what does it represent for Oaxaca, Mexico? 认知储备改变痴呆症的发展轨迹:这对墨西哥瓦哈卡来说意味着什么?
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-05-16 DOI: 10.24875/rmn.22000032
Gemma M. Martínez-Martínez, D. C. Nance, Elizabeth Muñoz-Ortiz
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引用次数: 0
Accelerate the diagnosis of amyotrophic lateral sclerosis using the Gold Coast criteria and biomarkers 使用黄金海岸标准和生物标志物加速肌萎缩侧索硬化症的诊断
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-05-16 DOI: 10.24875/rmn.22000076
H. R. Martinez
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引用次数: 0
Volumetric of the lateral ventricles in computed tomography images in Cubans adults with normal cognitive functions 认知功能正常的古巴成年人的侧脑室体积的计算机断层图像
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-05-16 DOI: 10.24875/rmn.22000058
Katherine S. Hernandez-Cortés, Adrián A. Mesa-Pujals, Nelsa M. Sagaró del Campo, Montoya Pedrón-Arquímedes
{"title":"Volumetric of the lateral ventricles in computed tomography images in Cubans adults with normal cognitive functions","authors":"Katherine S. Hernandez-Cortés, Adrián A. Mesa-Pujals, Nelsa M. Sagaró del Campo, Montoya Pedrón-Arquímedes","doi":"10.24875/rmn.22000058","DOIUrl":"https://doi.org/10.24875/rmn.22000058","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-05-16","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"44031041","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Neuropsychiatric and young-onset as clinical determinants for a delayed HuntingtonÓ?s disease diagnosis 神经精神和年轻发病是延迟性亨廷顿舞蹈症的临床决定因素?s疾病诊断
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-17 DOI: 10.24875/rmn.22000055
A. Cervantes-Arriaga, David García-Romero, M. Muñuzuri-Camacho, O. Esquivel-Zapata, D. J. Dávila-Ortiz de Montellano, L. Martínez-Ruano, Ana J. Hernández-Medrano, María A. Ruiz-Mafud, Gloria I. Cerda-Hernández, A. Abundes-Corona, M. Rodríguez‐Violante
{"title":"Neuropsychiatric and young-onset as clinical determinants for a delayed HuntingtonÓ?s disease diagnosis","authors":"A. Cervantes-Arriaga, David García-Romero, M. Muñuzuri-Camacho, O. Esquivel-Zapata, D. J. Dávila-Ortiz de Montellano, L. Martínez-Ruano, Ana J. Hernández-Medrano, María A. Ruiz-Mafud, Gloria I. Cerda-Hernández, A. Abundes-Corona, M. Rodríguez‐Violante","doi":"10.24875/rmn.22000055","DOIUrl":"https://doi.org/10.24875/rmn.22000055","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-02-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"49490846","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Ketone bodies as treatments for neurological illnesses 酮体治疗神经系统疾病
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-17 DOI: 10.24875/rmn.22000062
David Arango-Morales, Nidia Arango-Morales
Ketone bodies such as β -hydroxybutyrate, acetoacetate, and acetone are produced mainly in the liver from fatty acids. In this way, they can be used by the body as an alternate source of energy. However, their properties go beyond an alternate source of energy. For many years, ketogenic therapies have been implemented for the treatment of certain neurological diseases, such as epilepsy. History shows us that its implementations were beneficial, minimizing the severity of seizures despite ignoring their mechanisms of action. At present, their applications and mechanisms are beginning to be understood.
{"title":"Ketone bodies as treatments for neurological illnesses","authors":"David Arango-Morales, Nidia Arango-Morales","doi":"10.24875/rmn.22000062","DOIUrl":"https://doi.org/10.24875/rmn.22000062","url":null,"abstract":"Ketone bodies such as β -hydroxybutyrate, acetoacetate, and acetone are produced mainly in the liver from fatty acids. In this way, they can be used by the body as an alternate source of energy. However, their properties go beyond an alternate source of energy. For many years, ketogenic therapies have been implemented for the treatment of certain neurological diseases, such as epilepsy. History shows us that its implementations were beneficial, minimizing the severity of seizures despite ignoring their mechanisms of action. At present, their applications and mechanisms are beginning to be understood.","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-02-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"48319504","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Clinical insights on some rare genetic neurologic diseases 一些罕见遗传性神经系统疾病的临床观察
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-17 DOI: 10.24875/rmn.m23000094
M. García‐Grimshaw
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引用次数: 0
Seroprevalence and seroconversion of JC virus antibodies in a Mexican multiple sclerosis patients cohort 墨西哥多发性硬化症患者队列中JC病毒抗体的血清流行率和血清转化率
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-17 DOI: 10.24875/rmn.22000057
B. Bertado-Cortés, A. Castro, D. Rubio
{"title":"Seroprevalence and seroconversion of JC virus antibodies in a Mexican multiple sclerosis patients cohort","authors":"B. Bertado-Cortés, A. Castro, D. Rubio","doi":"10.24875/rmn.22000057","DOIUrl":"https://doi.org/10.24875/rmn.22000057","url":null,"abstract":"","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-02-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"43187139","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
What a neurologist must know of hereditary ATTRv amyloidosis 对于遗传性ATTRv淀粉样变,神经科医生应该知道多少啊
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-17 DOI: 10.24875/rmn.22000071
M. E. Briseño-Godínez, K. Cárdenas-Soto, Rosa X. Domínguez-Vega, A. González-Duarte
Transthyretin-related hereditary amyloidosis (ATTR) is a systemic disease result of an autosomal dominant mutation in the transthyretin (TTR) gene. This mutation will cause the deposition of misfolded TTR protein in several organs, leading to its dysfunction. The disease has been described worldwide, although it is still considered rare. It has a region-dependent genotype-phenotype correlation. Cardiomyopathy and a symmetric length-dependent sensory-motor and autonomic polyneurop-athy are the hallmark manifestations. However, in non-endemic areas, the diagnosis will challenge the neurologist because some systemic manifestations are usually attributed to more common disorders. Therefore, in the approach of a patient with polyneuropathy, the presence of family history, early autonomic dysfunction (cardiovascular, genitourinary, or gastrointestinal features), cardiac involvement, and inexplicable weight loss should be considered red flags that point toward ATTR. After diagnosis, the goal will always be to have a multidisciplinary approach, including a complete neurologic, autonomic, and cardiologic assessment, to establish a short-and long-term strategy for treatment, including symptomatic and disease-modifying medications that are now available. In that way, the increased awareness among specialists will positively impact the patient quality of life and survival by accomplishing an earlier diagnosis and treatment.
{"title":"What a neurologist must know of hereditary ATTRv amyloidosis","authors":"M. E. Briseño-Godínez, K. Cárdenas-Soto, Rosa X. Domínguez-Vega, A. González-Duarte","doi":"10.24875/rmn.22000071","DOIUrl":"https://doi.org/10.24875/rmn.22000071","url":null,"abstract":"Transthyretin-related hereditary amyloidosis (ATTR) is a systemic disease result of an autosomal dominant mutation in the transthyretin (TTR) gene. This mutation will cause the deposition of misfolded TTR protein in several organs, leading to its dysfunction. The disease has been described worldwide, although it is still considered rare. It has a region-dependent genotype-phenotype correlation. Cardiomyopathy and a symmetric length-dependent sensory-motor and autonomic polyneurop-athy are the hallmark manifestations. However, in non-endemic areas, the diagnosis will challenge the neurologist because some systemic manifestations are usually attributed to more common disorders. Therefore, in the approach of a patient with polyneuropathy, the presence of family history, early autonomic dysfunction (cardiovascular, genitourinary, or gastrointestinal features), cardiac involvement, and inexplicable weight loss should be considered red flags that point toward ATTR. After diagnosis, the goal will always be to have a multidisciplinary approach, including a complete neurologic, autonomic, and cardiologic assessment, to establish a short-and long-term strategy for treatment, including symptomatic and disease-modifying medications that are now available. In that way, the increased awareness among specialists will positively impact the patient quality of life and survival by accomplishing an earlier diagnosis and treatment.","PeriodicalId":53921,"journal":{"name":"Revista Mexicana de Neurociencia","volume":" ","pages":""},"PeriodicalIF":0.2,"publicationDate":"2023-02-17","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"42884383","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
引用次数: 0
Guía clínica. Diagnóstico y tratamiento de crisis febriles 临床指南。发烧危机的诊断和治疗
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-07 DOI: 10.24875/rmn.m19000057
Carlos G. Aguirre-Velázquez, Alma M. Huerta Hurtado, H. Ceja-Moreno, Karina Salgado-Hernández, Roberto San Román-Tovar, Martha A. Ortiz-Villalpando, Avril Molina-García, Guadalupe Vargas-Ramírez, Jaime López-Rivera, Rosana Huerta-Albarrán
Febrile seizures (FeS) are the most common problem in pediatric neurological practice. They are convulsive episodes during the course of febrile illness in the absence of epilepsy, severe hydroelectrolytic imbalance or neuroinfection. Its diagnosis is clinical and classified as simple and complex. Febrile status epilepticus occurs in approximately 5% of cases. It is convenient to teach parents how to act in a seizure and clarify that a FeS is not epilepsy, it is a benign process that usually does not leave neurological sequelae, and in which mortality is zero. In this clinical guide, we indicate risk factors for recurrence, management instructions for the first FeS, as well as criteria for hospital admission and treatment for prolonged seizures.
热性惊厥(FeS)是儿科神经学实践中最常见的问题。它们是在没有癫痫、严重的水电解质失衡或神经感染的发热性疾病过程中的惊厥发作。它的诊断是临床的,分为简单和复杂。大约5%的病例发生发热性癫痫持续状态。教导家长在癫痫发作时如何采取行动,并澄清FeS不是癫痫,这是一种良性过程,通常不会留下神经系统后遗症,死亡率为零,这样做很方便。在本临床指南中,我们指出了复发的危险因素,第一次FeS的管理说明,以及住院标准和长期癫痫发作的治疗。
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引用次数: 0
Guía clínica. Fármacos antiepilépticos de elección para crisis focales y generalizadas en adultos 临床指南。成人局灶性和全身性癫痫发作的抗癫痫药物选择
IF 0.2 Q4 CLINICAL NEUROLOGY Pub Date : 2023-02-07 DOI: 10.24875/rmn.m19000055
Leopoldo Rivera-Castaño, H. Sentíes-Madrid, Jesús Berumen-Jaik, I. Martínez-Juárez
The mainstay of treatment in patients with epilepsy is the use of antiepileptic drugs (AEDs). Currently, there are a significant number of AEDs in Mexico. For the pharmacological management of the patient with epilepsy, it is important to know the pharmacokinetics, dosage, mechanism of action, and formulations of the AEDs. 70-80% of patients with either focal or generalized seizures are completely seizure free on AED monotherapy. When, despite the use of AED in monotherapy, seizure freedom is not achieved, a second AED should be used; AEDs with different mechanisms of action are empirically combined el 100% de control; sin embargo, es importante considerar que de un 20 a un 30% no logra esta meta 3 .
治疗癫痫患者的主要方法是使用抗癫痫药物(AEDs)。目前,墨西哥有相当数量的aed。了解抗癫痫药的药代动力学、剂量、作用机制和处方对癫痫患者的药物管理非常重要。70-80%的局灶性或全面性癫痫患者在AED单药治疗下完全无癫痫发作。当在单药治疗中使用AED后,癫痫发作仍未得到缓解时,应使用第二台AED;不同作用机制的抗癫痫药经经验组合,达到100%脱毒;在禁运中,重要的是要考虑到联合国20年的规定,联合国30年的规定没有任何意义。
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引用次数: 0
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Revista Mexicana de Neurociencia
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