Pub Date : 1936-01-01DOI: 10.1136/jnnp.s1-16.63.274
evidently as a result of disturbed renal secretion; also diabetes mellitus was present together with a moderate degree of spondylarthritis deformans and a slight degree of neuritis or radiculitis. The cramps ceased when the radiculitis became worse; they returned when the symptoms of radiculitis improved. An increase in the blood uric acid and a slight degree of radiculitis evidently were of great importance in producing painful spasms. On the other hand, metabolic changes resulting from diabetes were of no importance in the production of cramp. The spasms were diminished by a meatless diet and by decreasing the amount of uric acid in the body. The patient usually felt better in the spring and still more so in the summer than during the winter. By electrical examination the so-called myospastic reaction was demonstrated. The signs of the reaction were as follows: With a prolonged faradic current cramp in the abdominal muscles arose; this never resulted from a galvanic current. In the lower limbs irritability to galvanism was diminished for direct as well as indirect excitation but to faradism the excitability was normal in the lower limbs for direct as well as indirect excitation. The quality of the contraction was everywhere normal. AI; PROGNOSIS AND TREATMENT [117] Treatment of epilepsy with antirabic vaccine (Behandlung der Epilepsie mit antirabischen Vakzinen).-M. NIKOLIC. Munch. med. Woch., 1935, 82, 1493. A MAN, age 48, had suffered from typical idiopathic epilepsy for 38 years. For the last two years one or two epileptic fits had occurred daily. He was bitten by a dog suspected of suffering from rabies. The patient received an injection of antirabic Markvaccine Type B (Hempt) for two days and of Hirnvaccine Type A on each of the following six days. He had no fits and was well and healthy when seen three months after the first injection. It would be of interest to know if other cases of genuine epilepsy can be relieved in the same manner. M. [118] Trends in the outcome of general paresis.-HORATIO M. POLLOCK. Psychiatric Quarterly, 1935, 9, 194. THIS study comprises 10,240 first admissions, of which 8,186 were males and 2,054 females. It was found that the trend in rate of first admissions with general paresis is slowly declining, but the trend in female first admissions is rising. No change is found in trend in age-distribution of first admissions. Trends in recovery and improvement are upward and in death rates down274
显然是由于肾脏分泌紊乱所致;此外，糖尿病患者还伴有中度的脊柱畸形和轻度的神经炎或神经根炎。当神经根炎加重时，痉挛停止;当神经根炎的症状好转时，他们又回来了。血尿酸升高和轻微的神经根炎显然是引起疼痛性痉挛的重要因素。另一方面，由糖尿病引起的代谢变化在抽筋的产生中并不重要。通过无肉饮食和减少体内尿酸的量，痉挛得以减轻。病人通常在春天感觉好些，夏天比冬天感觉好些。通过电检查证实了所谓的肌痉挛反应。反应的表现如下:随着时间的延长，腹部肌肉出现痉挛;这绝不是电流造成的。在直接和间接刺激下，下肢对电激的兴奋性减弱，而对法拉第的直接和间接刺激下，下肢的兴奋性正常。所有地方的收缩都是正常的。人工智能;[117]黄志军。抗结核疫苗治疗癫痫的临床研究[j]。尼克里奇。蒙克。地中海,Woch。， 1935, 82, 1493。男性，48岁，患有典型的特发性癫痫38年。在过去的两年里，每天都有一两次癫痫发作。他被一只疑似患狂犬病的狗咬了。患者连续两天注射抗rabic Markvaccine B (Hempt)，随后6天每天注射Hirnvaccine A。第一次注射后三个月，他没有痉挛，身体健康。我们很想知道其他真正的癫痫病例是否也能以同样的方式得到缓解。[118]李建平。——霍雷肖·波洛克精神病学季刊，1935,9,194。这项研究包括10,240名首次入学的学生，其中8,186名男性和2,054名女性。研究发现，全面性脑卒中患者首次入院率呈缓慢下降趋势，而女性首次入院率呈上升趋势。首次入学的年龄分布趋势没有变化。恢复和改善的趋势是上升的，死亡率是下降的
{"title":"PROGNOSIS AND TREATMENT","authors":"","doi":"10.1136/jnnp.s1-16.63.274","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.63.274","url":null,"abstract":"evidently as a result of disturbed renal secretion; also diabetes mellitus was present together with a moderate degree of spondylarthritis deformans and a slight degree of neuritis or radiculitis. The cramps ceased when the radiculitis became worse; they returned when the symptoms of radiculitis improved. An increase in the blood uric acid and a slight degree of radiculitis evidently were of great importance in producing painful spasms. On the other hand, metabolic changes resulting from diabetes were of no importance in the production of cramp. The spasms were diminished by a meatless diet and by decreasing the amount of uric acid in the body. The patient usually felt better in the spring and still more so in the summer than during the winter. By electrical examination the so-called myospastic reaction was demonstrated. The signs of the reaction were as follows: With a prolonged faradic current cramp in the abdominal muscles arose; this never resulted from a galvanic current. In the lower limbs irritability to galvanism was diminished for direct as well as indirect excitation but to faradism the excitability was normal in the lower limbs for direct as well as indirect excitation. The quality of the contraction was everywhere normal. AI; PROGNOSIS AND TREATMENT [117] Treatment of epilepsy with antirabic vaccine (Behandlung der Epilepsie mit antirabischen Vakzinen).-M. NIKOLIC. Munch. med. Woch., 1935, 82, 1493. A MAN, age 48, had suffered from typical idiopathic epilepsy for 38 years. For the last two years one or two epileptic fits had occurred daily. He was bitten by a dog suspected of suffering from rabies. The patient received an injection of antirabic Markvaccine Type B (Hempt) for two days and of Hirnvaccine Type A on each of the following six days. He had no fits and was well and healthy when seen three months after the first injection. It would be of interest to know if other cases of genuine epilepsy can be relieved in the same manner. M. [118] Trends in the outcome of general paresis.-HORATIO M. POLLOCK. Psychiatric Quarterly, 1935, 9, 194. THIS study comprises 10,240 first admissions, of which 8,186 were males and 2,054 females. It was found that the trend in rate of first admissions with general paresis is slowly declining, but the trend in female first admissions is rising. No change is found in trend in age-distribution of first admissions. Trends in recovery and improvement are upward and in death rates down274","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1936-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.63.274","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913659","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1936-01-01DOI: 10.1136/jnnp.s1-16.63.275
[121] A note -on the persistence of moods.-E. N. KENDREW. Brit. Jour. Psychol., 1935, 26, 165. TiE experiment here dealt with was devised to test the possibility of achieving a technique for measuring the degree of persistence of moods experimentally aroused in young children. The 20 cases fell into three groups. In nine disappointment appeared to produce persistent effects of a diminishing order of magnitude; in eight cases the persistent effect was of an increasing order of magnitude; in three cases no noticeable effects could be observed. Ji several cases a persisting conative activity showed itself in general behaviour, but the emotional disturbance had a greater effect on the natural rate of working. The experimental results are shown to fit well with general observations. Interest and fatigue influenced the results in a few cases, while two children, at least, delayed the effect of disturbance by a determined effort.
{"title":"Psychopathology","authors":"","doi":"10.1136/jnnp.s1-16.63.275","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.63.275","url":null,"abstract":"[121] A note -on the persistence of moods.-E. N. KENDREW. Brit. Jour. Psychol., 1935, 26, 165. TiE experiment here dealt with was devised to test the possibility of achieving a technique for measuring the degree of persistence of moods experimentally aroused in young children. The 20 cases fell into three groups. In nine disappointment appeared to produce persistent effects of a diminishing order of magnitude; in eight cases the persistent effect was of an increasing order of magnitude; in three cases no noticeable effects could be observed. Ji several cases a persisting conative activity showed itself in general behaviour, but the emotional disturbance had a greater effect on the natural rate of working. The experimental results are shown to fit well with general observations. Interest and fatigue influenced the results in a few cases, while two children, at least, delayed the effect of disturbance by a determined effort.","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1936-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.63.275","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913699","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1936-01-01DOI: 10.1136/jnnp.s1-16.63.270
enlargement was observed clinically in seven patients suffering from chroniic encephalitis treated by atropine for a prolonged period. A disturbance in tone of the musculature of the bowel appeared to be the cause of the enlarge-
临床观察到7例长期使用阿托品治疗的慢性脑炎患者脑膜肿大。肠内肌肉组织的张力紊乱似乎是肠肿大的原因
{"title":"SENSORIMOTOR NEUROLOGY","authors":"","doi":"10.1136/jnnp.s1-16.63.270","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.63.270","url":null,"abstract":"enlargement was observed clinically in seven patients suffering from chroniic encephalitis treated by atropine for a prolonged period. A disturbance in tone of the musculature of the bowel appeared to be the cause of the enlarge-","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1936-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.63.270","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913619","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.157
THE question is discussed whether the products of disintegration arising in the body, which act on the bloodvessels, may have an influence upon the permeability of the bloodvessels in the brain. Experiments performed on dogs showed that the pharmacological representative of such substances of disintegration, viz., histamine, enables fuchsin S to penetrate into the cerebrospinal fluid. Other experiments were performed in order to ascertain the regions in which this permeability is increased by histamine. M.
{"title":"SENSORIMOTOR NEUROLOGY","authors":"","doi":"10.1136/jnnp.s1-16.62.157","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.157","url":null,"abstract":"THE question is discussed whether the products of disintegration arising in the body, which act on the bloodvessels, may have an influence upon the permeability of the bloodvessels in the brain. Experiments performed on dogs showed that the pharmacological representative of such substances of disintegration, viz., histamine, enables fuchsin S to penetrate into the cerebrospinal fluid. Other experiments were performed in order to ascertain the regions in which this permeability is increased by histamine. M.","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.157","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913019","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.179
chromatolysis. The number of neurons was reduced, but focal necroses were seldom present. In two other cases which showed Korsakow's syndrome, areas of arteriosclerotic softening were found, but there was no constant localization of the pathological changes. It does not seem justifiable to state that there is a selective degeneration present in chronic alcoholism, or any correspondence between severity of the clinical symptoms and the degree of corresponding brain lesions. R. G. G.
{"title":"PROGNOSIS AND TREATMENT","authors":"","doi":"10.1136/jnnp.s1-16.62.179","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.179","url":null,"abstract":"chromatolysis. The number of neurons was reduced, but focal necroses were seldom present. In two other cases which showed Korsakow's syndrome, areas of arteriosclerotic softening were found, but there was no constant localization of the pathological changes. It does not seem justifiable to state that there is a selective degeneration present in chronic alcoholism, or any correspondence between severity of the clinical symptoms and the degree of corresponding brain lesions. R. G. G.","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.179","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913517","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.144
F P Weber
{"title":"Regarding the `Stuff' of which Dreams are Made-The Psychophysiological Basis of Dreams.","authors":"F P Weber","doi":"10.1136/jnnp.s1-16.62.144","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.144","url":null,"abstract":"","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.144","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40108359","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.111
K Zeiner-Henriksen
{"title":"Crystal-Formations in the Spinal Fluid and their Diagnostic Significance.","authors":"K Zeiner-Henriksen","doi":"10.1136/jnnp.s1-16.62.111","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.111","url":null,"abstract":"","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.111","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"40108355","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"OA","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.167
A. Long
THE investigations of the writer started with the major anxiety attack. This somatic syndrome is composed of intense vasoconstriction of the skin (paresthesias, sensation of cold, pallor), tachycardia up to 150, inhibition of salivation, cold sweating, mydriasis, arterial hypertonus up to 150 mm. (of mercury), and relaxation of the voluntary muscle system. This indicates a stormy excitation of the sympathetic system, which may be combined at the end with parasympathetic phenomena. It was found that this syndrome could be removed by choline preparations, which by their stimulating effect on the parasympathetic nerve produced an effect exactly the opposite to the anxiety syndrome: by an intramuscular injection of 0-1 c.cm. acetylcholine the anxiety attack can be stopped also in its psychical effects, and even chronic anxiety attacks can disappear after a few days by oral administration of the choline preparations pacyl or hypotan. We can therefore suppose that the fear of an anxiety neurosis arises primarily in a somatic way. The vasoconstriction here plays an important part, and the character of the anxiety syndrome corresponding to the adrenaline effect may be traced back to a primary action of the adrenal glands. Since some sexual damage is found, such as the inhibition of normal relief, and since the anxiety could be removed both by the prevention of this and by choline preparations, this confirms Freud's theory of the damming up of libido, and Reich's theory of the origin of anxiety in a sympathetic-toxic action of the sexual hormone. The somatic genesis of neurotic anxiety can be designed in the following way. There is always a disturbance of the course of sexual irritation to be found. This results either from sexual abstinence caused by external or neurotic reasons, or from neurotic sexual hypaesthesia while relatively great libido is present, and leads, with a' certain sympatheticotonic disposition, to a strong excitation of the sympathetic system. At this point the mechanism can be interrupted by choline medication. According to the working through of the state of sympathetic excitation there results either the syndrome phrenocardia, or, with corresponding psychic preparedness, the anxiety neurosis. If this continues for some time it can become fixated and psychically worked through so that anxiety can then also arise from the psyche. The somatic neurosis only lasts for a certain time and then becomes built over in a psychoneurotic manner so that then it may only be treated by a longcontinued psychotherapy.
{"title":"NEUROSES AND PSYCHONEUROSES","authors":"A. Long","doi":"10.1136/jnnp.s1-16.62.167","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.167","url":null,"abstract":"THE investigations of the writer started with the major anxiety attack. This somatic syndrome is composed of intense vasoconstriction of the skin (paresthesias, sensation of cold, pallor), tachycardia up to 150, inhibition of salivation, cold sweating, mydriasis, arterial hypertonus up to 150 mm. (of mercury), and relaxation of the voluntary muscle system. This indicates a stormy excitation of the sympathetic system, which may be combined at the end with parasympathetic phenomena. It was found that this syndrome could be removed by choline preparations, which by their stimulating effect on the parasympathetic nerve produced an effect exactly the opposite to the anxiety syndrome: by an intramuscular injection of 0-1 c.cm. acetylcholine the anxiety attack can be stopped also in its psychical effects, and even chronic anxiety attacks can disappear after a few days by oral administration of the choline preparations pacyl or hypotan. We can therefore suppose that the fear of an anxiety neurosis arises primarily in a somatic way. The vasoconstriction here plays an important part, and the character of the anxiety syndrome corresponding to the adrenaline effect may be traced back to a primary action of the adrenal glands. Since some sexual damage is found, such as the inhibition of normal relief, and since the anxiety could be removed both by the prevention of this and by choline preparations, this confirms Freud's theory of the damming up of libido, and Reich's theory of the origin of anxiety in a sympathetic-toxic action of the sexual hormone. The somatic genesis of neurotic anxiety can be designed in the following way. There is always a disturbance of the course of sexual irritation to be found. This results either from sexual abstinence caused by external or neurotic reasons, or from neurotic sexual hypaesthesia while relatively great libido is present, and leads, with a' certain sympatheticotonic disposition, to a strong excitation of the sympathetic system. At this point the mechanism can be interrupted by choline medication. According to the working through of the state of sympathetic excitation there results either the syndrome phrenocardia, or, with corresponding psychic preparedness, the anxiety neurosis. If this continues for some time it can become fixated and psychically worked through so that anxiety can then also arise from the psyche. The somatic neurosis only lasts for a certain time and then becomes built over in a psychoneurotic manner so that then it may only be treated by a longcontinued psychotherapy.","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.167","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913410","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.149
G. Reat
{"title":"The Pathogenesis and Treatment of Acute Anterior Poliomyelitis","authors":"G. Reat","doi":"10.1136/jnnp.s1-16.62.149","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.149","url":null,"abstract":"","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.149","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63912965","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 1935-10-01DOI: 10.1136/jnnp.s1-16.62.185
Les Enciphalites Psychosiques. By Dr. L. Marchand and Dr. A. Courtois. With a Preface by Dr. E. Toulouse, and 31 microphotographic illustrations. Paris: Librairie E. Le Frangois. 1935. Pp. 144. Price 25 fr. BY the term 'psychotic encephalitis' is meant an inflammation of the brain that reveals itself solely by mental symptoms. These generally take the form of acute confusion, delirium, hallucinosis, and other features of a toxic psychosis; neurological symptoms are lacking. Pathologically, the lesions are partly degenerative, partly inflammatory; they are in no way specific, yet they are quite definite. There is no perivascular demyelination, and little glial reaction. The mesoderm, however, reacts, while the parenchyma is damaged. On the clinical side the authors draw particular attention to the blood urea, which in acute cases always rises and is regarded by them as constant and characteristic-so much so that they use the term ' encephalite psychosique aigue azotemique.' Subacute and chronic cases are also known; their syndromes can be schizoid, paranoid, manic-depressive, hebephrenocatatonic, etc. The authors rely on pathological anatomy to show the foundation of the symptoms, but since they admit causation is multiple it is evident that any correlation of syndromes and lesions is impossible; the various mental symptoms they describe as occurring in subacute and chronic cases can also develop in cases where the lesions specified are not found. They have done well, however, in directing attention to the fact that some forms of encephalitis are accompanied by symptoms of the psychical series alone. The explanation of this interesting peculiarity is still obscure.
{"title":"Reviews and Notices of Books","authors":"","doi":"10.1136/jnnp.s1-16.62.185","DOIUrl":"https://doi.org/10.1136/jnnp.s1-16.62.185","url":null,"abstract":"Les Enciphalites Psychosiques. By Dr. L. Marchand and Dr. A. Courtois. With a Preface by Dr. E. Toulouse, and 31 microphotographic illustrations. Paris: Librairie E. Le Frangois. 1935. Pp. 144. Price 25 fr. BY the term 'psychotic encephalitis' is meant an inflammation of the brain that reveals itself solely by mental symptoms. These generally take the form of acute confusion, delirium, hallucinosis, and other features of a toxic psychosis; neurological symptoms are lacking. Pathologically, the lesions are partly degenerative, partly inflammatory; they are in no way specific, yet they are quite definite. There is no perivascular demyelination, and little glial reaction. The mesoderm, however, reacts, while the parenchyma is damaged. On the clinical side the authors draw particular attention to the blood urea, which in acute cases always rises and is regarded by them as constant and characteristic-so much so that they use the term ' encephalite psychosique aigue azotemique.' Subacute and chronic cases are also known; their syndromes can be schizoid, paranoid, manic-depressive, hebephrenocatatonic, etc. The authors rely on pathological anatomy to show the foundation of the symptoms, but since they admit causation is multiple it is evident that any correlation of syndromes and lesions is impossible; the various mental symptoms they describe as occurring in subacute and chronic cases can also develop in cases where the lesions specified are not found. They have done well, however, in directing attention to the fact that some forms of encephalitis are accompanied by symptoms of the psychical series alone. The explanation of this interesting peculiarity is still obscure.","PeriodicalId":50117,"journal":{"name":"Journal of Neurology and Psychopathology","volume":null,"pages":null},"PeriodicalIF":0.0,"publicationDate":"1935-10-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1136/jnnp.s1-16.62.185","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"63913550","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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