Clinical and Experimental Allergy最新文献

英文中文

Investigation of the Pathomechanism of Chronic Cough Using an In Vitro Approach. 慢性咳嗽的体外病理机制研究。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-21 DOI: 10.1111/cea.14628

Umesh Singh, Jonathan A Bernstein

引用次数: 0

Correction to "T cells and eosinophils in bronchial smooth muscle cell death in asthma". 修正“哮喘支气管平滑肌细胞死亡中的T细胞和嗜酸性粒细胞”。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-21 DOI: 10.1111/cea.14627

引用次数: 0

Associations Between Self-Reported Dietary Intake and Atopic Dermatitis Risk in Young Adults From Singapore and Malaysia. 新加坡和马来西亚年轻人自我报告的饮食摄入量与特应性皮炎风险之间的关系

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-21 DOI: 10.1111/cea.14629

Jun Jie Lim, Kavita Reginald, Yee-How Say, Mei Hui Liu, Fook Tim Chew

This cross-sequential study found that frequent intake of high-fat and high-protein foods was associated with higher odds of atopic dermatitis (AD). However, occasional intake across all three macronutrients significantly lowered AD odds, suggesting that moderation-not strict avoidance-may benefit AD management in allergic populations.

这项交叉序列研究发现，频繁摄入高脂肪和高蛋白食物与特应性皮炎（AD）的几率较高有关。然而，偶尔摄入这三种常量营养素会显著降低AD的发病率，这表明适度摄入——而不是严格避免摄入——可能有利于过敏人群的AD管理。

引用次数: 0

Food Allergy and Mental Health in Children and Adolescents—The Role of Shared Familial Environment 儿童和青少年食物过敏与心理健康——共享家庭环境的作用。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-15 DOI: 10.1111/cea.14619

Hanna Karim, Cecilia Lundholm, Tong Gong, Bronwyn Brew, Michael Silverman, Catarina Almqvist

Background

Evidence suggests a link between food allergy and poor mental health, however, this may be explained by shared genetic and environmental factors. We aimed to investigate the association between food allergy of different severity and mental health in children, and the role of familial factors.

Methods

This population-based, longitudinal cohort study is based on the Child and Adolescent Twin Study in Sweden with questionnaire data reported by parents and/or children. Food allergy ‘ever’ and doctor's diagnosis were reported at age 9–12 years, and ≥ 1 recent dispensation of adrenaline was used as a marker for current severe food allergy. Outcomes were identified using validated questionnaires for anxiety; Screen for Child Anxiety Related Disorders (SCARED); Strength and Difficulties Questionnaire (SDQ) and depression; Short Mood and Feelings Questionnaire (SMFQ), Center for Epidemiological Studies Depression Scale (CES-D) and Diagnostic and Statistical manual of Mental Disorders (DSM-IV-MDE) and reported at 9–12, 15 and 18 years of age. Multivariate linear and logistic modelling was applied to the whole cohort and a co-twin control approach to remove confounding by familial factors.

Results

In total, 3039 (8.9%) children had a parent-reported food allergy. Among these, 1292 (43.5%) had non-severe food allergy without diagnosis, 1490 (49%) had non-severe food allergy with diagnosis and 257 (8.5%) had severe food allergy. Compared to children with no food allergy, non-severe food allergy with diagnosis by 9–12 years was associated with parent-reported anxiety/depression; SCARED (adjOR 2.10, 95% CI 1.48–2.98), SMFQ (adjOR 1.92, 95% CI 1.19–3.10) at 9–12 years and SDQ (adjβ 0.2, 95% CI 0.0–0.4) at 15 years. All other associations were null including for those with severe food allergy. All positive estimates in the full cohort were attenuated using co-twin controls.

Conclusion

Evidence associating paediatric food allergy severity and poor mental health was weak, and positive associations observed were likely due to familial confounding.

背景：有证据表明食物过敏和心理健康状况不佳之间存在联系，然而，这可能是由共同的遗传和环境因素来解释的。我们旨在探讨不同严重程度的食物过敏与儿童心理健康的关系，以及家族因素的作用。方法：这项以人群为基础的纵向队列研究基于瑞典的儿童和青少年双胞胎研究，调查问卷数据由父母和/或儿童报告。9-12岁报告“曾经”食物过敏和医生诊断，最近≥1次肾上腺素分配作为当前严重食物过敏的标志。使用有效的焦虑问卷确定结果；儿童焦虑相关障碍筛查（SCARED）；力量与困难问卷（SDQ）与抑郁；短期情绪和感觉问卷（SMFQ），流行病学研究中心抑郁量表（CES-D）和精神障碍诊断与统计手册（DSM-IV-MDE），并在9-12岁，15岁和18岁时报告。多变量线性和逻辑模型应用于整个队列，并采用双胎对照方法消除家族因素的混淆。结果：共有3039名（8.9%）儿童有父母报告的食物过敏。其中未确诊非严重食物过敏1292例（43.5%），确诊非严重食物过敏1490例（49%），重度食物过敏257例（8.5%）。与没有食物过敏的儿童相比，9-12岁诊断为非严重食物过敏的儿童与父母报告的焦虑/抑郁相关；9-12岁时为SCARED (adjOR 2.10, 95% CI 1.48-2.98), SMFQ (adjOR 1.92, 95% CI 1.19-3.10)， 15岁时为SDQ （adjβ 0.2, 95% CI 0.0-0.4）。所有其他的关联都是无效的，包括那些严重食物过敏的人。使用双胎对照，整个队列中所有阳性估计都减弱了。结论：儿童食物过敏严重程度与心理健康不良相关的证据较弱，观察到的正相关可能是由于家族混杂。

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引用次数: 0

Advocacy in Action: International Patient Group Improves Hereditary Angioedema Diagnosis and Care Across the Asia-Pacific. 倡导在行动：国际患者团体改善遗传性血管性水肿的诊断和护理在亚太地区。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-12 DOI: 10.1111/cea.14623

Jane C Y Wong, Cheryl C W Tsui, Kristie C W Lao, Jovilia Abong, Adli Ali, Dharmagat Bhattarai, Michihiro Hide, Ankur Jindal, Anthony Jordan, Hye-Ryun Kang, Constance H Katelaris, Narissara Suratannon, Sze-Chin Tan, Yong-Hao Lim, Deborah Corcoran, Fiona Wardman, Henrik Balle Boysen, Anthony J Castaldo, Philip H Li

This study demonstrates that patient advocacy groups significantly enhance medication availability and improve diagnosis of hereditary angioedema (HAE), particularly in emerging economies within the Asia-Pacific region. This study supports integrating patient advocacy group involvement into management guidelines, emphasising their role in improving access to diagnostics and treatment for HAE.

这项研究表明，患者权益团体能显著提高药物的可获得性，改善遗传性血管性水肿（HAE）的诊断，尤其是在亚太地区的新兴经济体。这项研究支持将患者权益团体的参与纳入管理指南，强调他们在改善 HAE 诊断和治疗方面的作用。

引用次数: 0

A Scoping Review of Cost Questionnaires Aimed at Measuring the Household Financial Burden of Food Allergy. 旨在衡量食物过敏家庭经济负担的成本问卷的范围审查。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-08 DOI: 10.1111/cea.14622

Zoe Harbottle, Michael A Golding, Ayel Luis R Batac, Andrew T Fong, Mê-Linh Lê, Elissa M Abrams, Edmond S Chan, Moshe Ben-Shoshan, Peter S Hsu, Jodi A Shroba, Juho E Kivistö, Matthew J Greenhawt, Gregory Mason, Mika J Mäkelä, Antonella Muraro, Staffan Ahlstedt, Jennifer L P Protudjer

引用次数: 0

How to Recognise Mastocytosis? 如何识别肥大细胞增多症？

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-07 DOI: 10.1111/cea.14608

Clive Grattan, Sigurd Broesby-Olsen, Knut Brockow

引用次数: 0

Fatal Food Anaphylaxis in Children: A Statutory Review in England 儿童致死性食物过敏反应：英国的法定审查。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-07 DOI: 10.1111/cea.14614

Sylvia Stoianova, Vibha Sharma, Robert J. Boyle

<p>Fatal anaphylaxis is a very rare, unpredictable tragedy. For children and young people, most fatal anaphylaxis is caused by food allergy, and thus, carers of children and young people with food allergy may become preoccupied about the possibility of sudden, unexpected fatal anaphylaxis. Other unexpected causes of death such as severe acute asthma are less rare than fatal anaphylaxis, but the rapidity of fatal food anaphylaxis and its propensity to affect adolescents and young adults generate continued concern and societal interest.</p><p>The burden of avoiding known food allergens falls largely on people living with allergies and their families. Thus, an exposure leading to a fatal anaphylactic reaction may inspire fear and guilt in families and also in other carers, caterers and health professionals. Most anaphylactic reactions to food self-revert, with or without medical intervention [<span>1</span>]. In the past 30 years, several society-wide changes have been made to try to prevent fatal food anaphylaxis. In many countries such as the UK, these include widespread provision of adrenaline autoinjectors, new food allergen labelling laws, increased food allergy diagnostics and improved emergency service awareness of anaphylaxis and its management [<span>2, 3</span>]. Yet, fatal food anaphylaxis remains as common as it was 30 years ago, suggesting we need to go back a step and learn more about the condition [<span>4</span>]. Fatal food anaphylaxis is difficult to study prospectively, because it is very rare, unpredictable and usually occurs in the community. It is therefore important that we learn as much as we can from each tragic occurrence, and the recent statutory review of childhood fatal anaphylaxis and asthma provides an opportunity to do this (Figure 1, Table 1).</p><p>England is one of very few countries with a national, statutory, multi-professional review of all child deaths [<span>5</span>]. Since 2019, the National Child Mortality Database (NCMD) collates and analyses information about child deaths. For all deaths under age 18, a comprehensive summary of the circumstances of death and background information from professionals is collated. A final record summarises conclusions of a multi-agency panel documenting contributory, modifiable factors and learning. The latest NCMD thematic report analysed child deaths in England because of asthma or anaphylaxis. It identified key findings and made recommendations for policy, practice and research [<span>6</span>].</p><p>The report documents 54 child deaths from asthma and 19 from anaphylaxis in a 4-year period from 1 April 2019 to 31 March 2023. Many (54%) of the children who died from asthma also had a food allergy. However, the cause of death in these cases was thought to be asthma rather than food anaphylaxis. Fatal anaphylaxis was triggered by food allergy (<i>n</i> = 18) and in one case by anaesthesia, a rare cause of fatal anaphylaxis in childhood [<span>7</span>]. Just over half of

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引用次数: 0

Role of MicroRNAs in Allergy and Basophil Activation Test for IgE-Mediated Drug Allergy microrna在ige介导的药物过敏和嗜碱性粒细胞激活试验中的作用。

IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-07 DOI: 10.1111/cea.14616

Mohamed H. Shamji, Robert J. Boyle

<p>In this month's editorial, the Editors of the journal have highlighted two fascinating studies that are included in this issue. The first article provides evidence that miR-107 is involved in the allergic response to house dust mites (HDM) in children with asthma [<span>1</span>]. Allergic asthma (AA) is a prevalent phenotype of asthma that presents atopic sensitisations in asthma patients exposed to allergens [<span>2, 3</span>]. Some, but not all, studies have suggested a possible increase in AA over time [<span>4</span>]. HDM are a significant trigger for AA in many regions, with HDM sensitisation potentially leading to severe, and in some instances, life-threatening asthma symptoms. In the study by Kim et al. [<span>1</span>], the relationship between microRNAs (miRNAs) and HDM sensitisation in children with asthma was investigated. The researchers examined serum samples from 1126 children in the Genetics of Asthma in Costa Rica Study (GACRS) and also replicated their findings in the Childhood Asthma Management Program (CAMP). Initially, the study revealed that 17 miRNAs were differentially expressed between HDM-sensitised and non-sensitised children in the GACRS group. Of the 17, miR-642a, let-7c-5p and miR-107 showed the strongest association with HDM sensitisation. Moreover, the CAMP cohort successfully replicated the elevated expression of miR-107 in HDM-sensitised children. Additional mediation analysis also presented significant effects of miR-107 on eosinophil count and total IgE after HDM sensitisation. These findings suggest new insights into the molecular mechanisms underlying HDM sensitisation in paediatric asthma and emphasise the potential of miRNAs, particularly, miR-107, which can be studied further for application as biomarkers or therapeutic targets for AA (Figure 1).</p><p>This issue's second editor's choice article reports on the optimisation of the basophil activation assay for identifying IgE-mediated drug allergies [<span>5</span>]. Diagnosing drug allergies is a complex and nuanced process that presents several challenges. Allergic reactions can manifest with a broad spectrum of clinical symptoms, ranging from mild rashes to life-threatening anaphylaxis, making it difficult to establish consistent diagnostic criteria. Reactions may occur immediately or be delayed, sometimes appearing days after exposure [<span>6</span>]. This variability complicates the temporal connection between drug intake and allergic symptoms. Additionally, patients may experience allergic reactions to drug molecules with similar structures, further complicating the identification of the exact trigger, especially in cases involving structurally similar drugs. Unlike testing for environmental allergens, there are limited standardised and clinically validated laboratory tests available for many drug allergies. The reliability and availability of tests like the Basophil Activation Test (BAT) can vary for different drugs [<span>7, 8</span>]. While o

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引用次数: 0

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IF 6.3 2区医学 Q1 ALLERGY

Clinical and Experimental Allergy

Pub Date : 2025-01-07 DOI: 10.1111/cea.14621

Shican Zhou, Ju Lai, Na Che, Kai Fan, Chuanliang Zhao, Bojin Long, Chunyan Yao, Yu Zeng, Shaoqing Yu

The cover image is based on the article Emerging Role of SAMSN1⁺Mast Cells: Insights From Mendelian Randomisation and Transcriptomic Analyses on Chronic Sinusitis and Obesity by Shaoqing Yu et al.,https://doi.org/10.1111/cea.14529.

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