The objective of this study was to examine individual and social influences on college student compliance with a gluten-free diet. Individual semi-structured interviews were conducted in 2016-2017 with undergraduates (n=24) at a large public university. Eligible participants were 18-25 years old with self-reported physician-diagnosed celiac disease. The interview question path was based on the Health Belief Model and assessed individual and social influences on eating choices. Transcribed interviews were independently coded by three researchers (ICR=.82) then analyzed for themes using the Health Belief Model as a guiding framework. Barriers to complying with a gluten-free diet included the college meal plan, meal planning and cooking skills, negotiating social interactions around food, financial burden of purchasing gluten-free food, and intentionally eating gluten as self-confidence grew. Students often reframed non-compliance as a “cheat day,” discussing it as they would a reduced-calorie diet, which could reduce perceived threat. Providers discussed threats to health in terms of long-term outcomes but students focused on the immediate impact of eating gluten. Nutritionists, parents, and health care providers can better prepare college students for the transition to independent food choices and how to remain compliant with a gluten-free diet within the challenging college food environment.
{"title":"Individual and Social Influences on College Student Compliance with a Gluten-Free Diet","authors":"Crystal Sparks, Taylor Zingg, M. Cheney","doi":"10.12691/ijcd-7-3-6","DOIUrl":"https://doi.org/10.12691/ijcd-7-3-6","url":null,"abstract":"The objective of this study was to examine individual and social influences on college student compliance with a gluten-free diet. Individual semi-structured interviews were conducted in 2016-2017 with undergraduates (n=24) at a large public university. Eligible participants were 18-25 years old with self-reported physician-diagnosed celiac disease. The interview question path was based on the Health Belief Model and assessed individual and social influences on eating choices. Transcribed interviews were independently coded by three researchers (ICR=.82) then analyzed for themes using the Health Belief Model as a guiding framework. Barriers to complying with a gluten-free diet included the college meal plan, meal planning and cooking skills, negotiating social interactions around food, financial burden of purchasing gluten-free food, and intentionally eating gluten as self-confidence grew. Students often reframed non-compliance as a “cheat day,” discussing it as they would a reduced-calorie diet, which could reduce perceived threat. Providers discussed threats to health in terms of long-term outcomes but students focused on the immediate impact of eating gluten. Nutritionists, parents, and health care providers can better prepare college students for the transition to independent food choices and how to remain compliant with a gluten-free diet within the challenging college food environment.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"201 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2019-12-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"76990844","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
H. Rahmoune, N. Boutrid, M. Amrane, Tahar Kherkhache, B. Bioud
Gluten-related disorders are an emerging worldwide with a global prevalence estimated at 5% in the general population. They include celiac disease, nonceliac gluten sensitivity and wheat allergy. Specifically, the peculiar wheat-dependent exercise-induced anaphylaxis is a particular allergy that occurs after wheat ingestion followed by physical exercise.
{"title":"Wheat-dependent Exercise-induced Anaphylaxis","authors":"H. Rahmoune, N. Boutrid, M. Amrane, Tahar Kherkhache, B. Bioud","doi":"10.12691/ijcd-7-3-2","DOIUrl":"https://doi.org/10.12691/ijcd-7-3-2","url":null,"abstract":"Gluten-related disorders are an emerging worldwide with a global prevalence estimated at 5% in the general population. They include celiac disease, nonceliac gluten sensitivity and wheat allergy. Specifically, the peculiar wheat-dependent exercise-induced anaphylaxis is a particular allergy that occurs after wheat ingestion followed by physical exercise.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"16 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2019-12-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"76568577","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
The etiology and pathogenesis of celiac disease remains obscure but remains the focus of intense research investigation. It is generally believed to be an immune-mediated small intestinal mucosal disorder that can cause diarrhea, impaired nutrient assimilation and weight loss. A key component in this process occurs at the intestinal epithelial cell surface that is closely associated with the luminal intestinal microbiome. Here, epithelial membrane glycoproteins and glycolipids are present along with adsorbed molecules that permit interaction with the intestinal microbiome. In recent years, use of specific sugar residue seeking proteins, lectins, that can be found in the diet have been employed topographically to map the small intestinal cell surface and goblet cell secretory mucins to further elucidate the structure and function of this tissue. Evidence has accumulated to indicate that this microenvironment may be critically important in further understanding the etiology and pathogenesis of celiac disease and other sprue-like intestinal disorders.
{"title":"Topographic Lectin Mapping of the Epithelial Cell Surface in Normal Intestine and Celiac Disease","authors":"H. Freeman","doi":"10.12691/ijcd-7-3-3","DOIUrl":"https://doi.org/10.12691/ijcd-7-3-3","url":null,"abstract":"The etiology and pathogenesis of celiac disease remains obscure but remains the focus of intense research investigation. It is generally believed to be an immune-mediated small intestinal mucosal disorder that can cause diarrhea, impaired nutrient assimilation and weight loss. A key component in this process occurs at the intestinal epithelial cell surface that is closely associated with the luminal intestinal microbiome. Here, epithelial membrane glycoproteins and glycolipids are present along with adsorbed molecules that permit interaction with the intestinal microbiome. In recent years, use of specific sugar residue seeking proteins, lectins, that can be found in the diet have been employed topographically to map the small intestinal cell surface and goblet cell secretory mucins to further elucidate the structure and function of this tissue. Evidence has accumulated to indicate that this microenvironment may be critically important in further understanding the etiology and pathogenesis of celiac disease and other sprue-like intestinal disorders.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"73 5 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2019-12-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"91554949","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Here we are continuing interview articles and now Assistant Professor Hakim Rahmoune from Department of Paediatrics of Setif University Hospital & University of Setif-1 is questioned.
{"title":"A Dialogue with Assistant Professor Hakim Rahmoune","authors":"P. Makovicky","doi":"10.12691/IJCD-7-2-4","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-4","url":null,"abstract":"Here we are continuing interview articles and now Assistant Professor Hakim Rahmoune from Department of Paediatrics of Setif University Hospital & University of Setif-1 is questioned.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"31 1","pages":"63-65"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"88171335","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Anti deamidated gliadin peptides antibodies are considered as celiac disease associated diagnostic antibodies. They are in clinical use for almost the last two decades. In the first decade they were preferentially used in early childhood, in face of IgA deficiency and occasionally recommended as the prime serological marker, outperforming the anti-tissue transglutaminase autoantibody. Notably, they were recommended in combination with the tissue transglutaminase as enhancer of the diagnostic performances. No more, the circle turned over. In the second decade (2012-2019), most of the studies limited and criticized their past published advantages. They suggested that deamidated gliadin peptides antibodies do not have any advantage over anti-tissue transglutaminase autoantibodies in terms of early childhood, IgA deficiency, diagnostic performances and when both antibodies are combined. It seems that the deamidated gliadin peptide are losing their place in the celiac disease algorithmic diagnostic flow chart.
{"title":"Deamidated Gliadin Peptide Antibodies in Celiac Disease: A Diagnostic Driver or just along for the Ride?","authors":"Lerner Aaron, Haimi Motti, Matthias Torsten","doi":"10.12691/IJCD-7-2-6","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-6","url":null,"abstract":"Anti deamidated gliadin peptides antibodies are considered as celiac disease associated diagnostic antibodies. They are in clinical use for almost the last two decades. In the first decade they were preferentially used in early childhood, in face of IgA deficiency and occasionally recommended as the prime serological marker, outperforming the anti-tissue transglutaminase autoantibody. Notably, they were recommended in combination with the tissue transglutaminase as enhancer of the diagnostic performances. No more, the circle turned over. In the second decade (2012-2019), most of the studies limited and criticized their past published advantages. They suggested that deamidated gliadin peptides antibodies do not have any advantage over anti-tissue transglutaminase autoantibodies in terms of early childhood, IgA deficiency, diagnostic performances and when both antibodies are combined. It seems that the deamidated gliadin peptide are losing their place in the celiac disease algorithmic diagnostic flow chart.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"1 1","pages":"42-45"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"90330686","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Celiac disease (CD) is the most common permanent T-cell mediated gluten intolerance, caused by the dietary gluten in individuals who are genetically susceptible to the disease. CD characterized by small intestine mucosal lesions, subtotal, or total intestinal villi atrophy and nutrient malabsorptions. Sometimes the diagnosis of CD is so challenging and needs a combination of clinical, serological, histopathological and genetic evaluations. The well-known cornerstone treatment of CD is gluten-free diet (GFD) and there is need to regular monitoring of patients after starting the GFD. The main aim of this review was to discussing the definition and pathogenesis of CD, together with the diagnostics, treatment and follow-up strategies in this disease.
{"title":"The Facts of Celiac Disease; A Comprehensive Review","authors":"N. Asri, M. Rostami-Nejad","doi":"10.12691/IJCD-7-2-7","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-7","url":null,"abstract":"Celiac disease (CD) is the most common permanent T-cell mediated gluten intolerance, caused by the dietary gluten in individuals who are genetically susceptible to the disease. CD characterized by small intestine mucosal lesions, subtotal, or total intestinal villi atrophy and nutrient malabsorptions. Sometimes the diagnosis of CD is so challenging and needs a combination of clinical, serological, histopathological and genetic evaluations. The well-known cornerstone treatment of CD is gluten-free diet (GFD) and there is need to regular monitoring of patients after starting the GFD. The main aim of this review was to discussing the definition and pathogenesis of CD, together with the diagnostics, treatment and follow-up strategies in this disease.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"21 1","pages":"48-52"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74526229","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Celiac disease (CD) is an autoimmune disorder precipitated generally in genetically susceptible individuals by the ingestion of gluten and is becoming a major communal health problem throughout the globe. Initially the disease was reported in few of the countries specifically in predominant Caucasian populations, but now a day it is being reported from almost all parts of the world. Simplification of the diagnostic criteria and widespread use of serologic tests have made it possible to estimate the prevalence of CD in the general population. But the exact global prevalence of the celiac disease is quite ambiguous due to the fact that when the disease is diagnosed with tTG test it is generally not confirmed with the biopsy examinations whereas, diagnosis should be based on the combination of both as per current guidelines on diagnosis of CD. The seropositive based (anti-tissue transglutaminase and/or anti-endomysial antibodies) data shows a high global prevalence of 1.4% whereas biopsy-confirmation based data shows a global prevalence of only 0.7%. Although prevalence of celiac disease varies with respect to sex, age, and location etc. but its accurate prevalence is not being reported due to various reasons. At the same time, a classified data of prevalence of the celiac disease based on multiple factors does not exist. This manuscript intends to highlight various underlying reasons responsible for generating vague prevalence data worldwide along with an awareness note regarding practical implications of diagnostic modalities.
{"title":"Seroprevalence vs Biopsy Prevalence of Celiac Disease: A Bird’s Eye View","authors":"Moni Kumari, M. Arora, N. Kaur, P. Bansal","doi":"10.12691/IJCD-7-2-3","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-3","url":null,"abstract":"Celiac disease (CD) is an autoimmune disorder precipitated generally in genetically susceptible individuals by the ingestion of gluten and is becoming a major communal health problem throughout the globe. Initially the disease was reported in few of the countries specifically in predominant Caucasian populations, but now a day it is being reported from almost all parts of the world. Simplification of the diagnostic criteria and widespread use of serologic tests have made it possible to estimate the prevalence of CD in the general population. But the exact global prevalence of the celiac disease is quite ambiguous due to the fact that when the disease is diagnosed with tTG test it is generally not confirmed with the biopsy examinations whereas, diagnosis should be based on the combination of both as per current guidelines on diagnosis of CD. The seropositive based (anti-tissue transglutaminase and/or anti-endomysial antibodies) data shows a high global prevalence of 1.4% whereas biopsy-confirmation based data shows a global prevalence of only 0.7%. Although prevalence of celiac disease varies with respect to sex, age, and location etc. but its accurate prevalence is not being reported due to various reasons. At the same time, a classified data of prevalence of the celiac disease based on multiple factors does not exist. This manuscript intends to highlight various underlying reasons responsible for generating vague prevalence data worldwide along with an awareness note regarding practical implications of diagnostic modalities.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"17 1","pages":"33-41"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74239477","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
N. Boutrid, H. Rahmoune, M. Amrane, R. Belbouab, B. Bioud
The expanding spectrum of gluten-related autoimmune disorders may encompass distal tubular acidosis. We report a case of an infant presenting a classical digestive celiac disease along with a transient, reversible tubular acidosis that vanished after gluten free diet.
{"title":"Gluten-induced Distal Tubular Acidosis?","authors":"N. Boutrid, H. Rahmoune, M. Amrane, R. Belbouab, B. Bioud","doi":"10.12691/IJCD-7-2-1","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-1","url":null,"abstract":"The expanding spectrum of gluten-related autoimmune disorders may encompass distal tubular acidosis. We report a case of an infant presenting a classical digestive celiac disease along with a transient, reversible tubular acidosis that vanished after gluten free diet.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"31 1","pages":"56-57"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"73961393","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Elena Cristina Garbovan, C. Aldea, G. Sur, C. Silaghi, Anamaria Garbovan
The association of celiac disease with chronic hepatitis C virus infection was intensely debated. We aimed to study the prevalence of celiac disease in patients with chronic hepatitis C virus infection.
{"title":"Celiac Disease and Chronic Hepatitis C Virus Infection – Prevalence Studies","authors":"Elena Cristina Garbovan, C. Aldea, G. Sur, C. Silaghi, Anamaria Garbovan","doi":"10.12691/IJCD-7-2-8","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-8","url":null,"abstract":"The association of celiac disease with chronic hepatitis C virus infection was intensely debated. We aimed to study the prevalence of celiac disease in patients with chronic hepatitis C virus infection.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"1 1","pages":"46-47"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"80059365","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
A woman with long-standing weight loss and malabsorption demonstrated a severe sprue-like enteropathy. She insidiously developed persistent and progressive muscle weakness caused by inclusion body myositis, an uncommon muscle disorder. Treatment with a gluten-free diet, steroids, calcium, zinc and vitamin supplements, including empirical vitamin E resulted in weight gain, but failed to histologically improve her small intestinal mucosa or the muscle weakness which became profound. The myopathic process could reflect a co-existent autoimmune disorder, or, possibly a direct result of long-standing and superimposed nutrient deficits. An alternative explanation may be a hitherto unrecognized syndrome manifested as inclusion body myositis and a form of sprue-like enteropathy.
{"title":"Sprue-Like Intestinal Disease Complicated by Inclusion Body Myositis","authors":"H. Freeman","doi":"10.12691/IJCD-7-2-5","DOIUrl":"https://doi.org/10.12691/IJCD-7-2-5","url":null,"abstract":"A woman with long-standing weight loss and malabsorption demonstrated a severe sprue-like enteropathy. She insidiously developed persistent and progressive muscle weakness caused by inclusion body myositis, an uncommon muscle disorder. Treatment with a gluten-free diet, steroids, calcium, zinc and vitamin supplements, including empirical vitamin E resulted in weight gain, but failed to histologically improve her small intestinal mucosa or the muscle weakness which became profound. The myopathic process could reflect a co-existent autoimmune disorder, or, possibly a direct result of long-standing and superimposed nutrient deficits. An alternative explanation may be a hitherto unrecognized syndrome manifested as inclusion body myositis and a form of sprue-like enteropathy.","PeriodicalId":13927,"journal":{"name":"International Journal of Celiac Disease","volume":"26 1","pages":"53-55"},"PeriodicalIF":0.0,"publicationDate":"2019-08-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"82867859","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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