Psychological review最新文献

Science and religion are influential social forces, and their interplay has been subject to many public and scholarly debates. The present article addresses how people mentally conceptualize the relationship between science and religion and how these conceptualizations can be systematized. To that end, we provide a comprehensive, integrative review of the pertinent literature. Moreover, we discuss how cognitive (in particular, epistemic beliefs) and motivational factors (in particular, epistemic needs, identity, and moral beliefs), as well as personality and contextual factors (e.g., rearing practices and cross-cultural exposure), are related to these mental conceptualizations. And finally, we provide a flowchart detailing the psychological processes leading to these mental conceptualizations. A comprehensive understanding of how individuals perceive the science-religion relationship is interesting in and of itself and practically relevant for managing societal challenges, such as science denial. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

Some neural representations gradually change across multiple timescales. Here we argue that modeling this "drift" could help explain the spacing effect (the long-term benefit of distributed learning), whereby differences between stored and current temporal context activity patterns produce greater error-driven learning. We trained a neurobiologically realistic model of the entorhinal cortex and hippocampus to learn paired associates alongside temporal context vectors that drifted between learning episodes and/or before final retention intervals. In line with spacing effects, greater drift led to better model recall after longer retention intervals. Dissecting model mechanisms revealed that greater drift increased error-driven learning, strengthened weights in slower drifting temporal context neurons (temporal abstraction), and improved direct cue-target associations (decontextualization). Intriguingly, these results suggest that decontextualization-generally ascribed only to the neocortex-can occur within the hippocampus itself. Altogether, our findings provide a mechanistic formalization for established learning concepts such as spacing effects and errors during learning. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

Experiencing stressful or traumatic events can lead to a range of responses, from mild disruptions to severe and persistent mental health issues. Understanding the various trajectories of response to adversity is crucial for developing effective interventions and support systems. Researchers have identified four commonly observed response trajectories to adversity, from which the resilient is the most common one. Resilience refers to the maintenance of healthy psychological functioning despite facing adversity. However, it remains an open question how to understand and anticipate resilience, due to its dynamic and multifactorial nature. This article presents a novel formalized framework to conceptualize resilience from a complex systems perspective. We use the network theory of psychopathology, which states that mental disorders are self-sustaining endpoints of direct symptom-symptom interactions organized in a network system. The internal structure of the network determines the most likely trajectory of symptom development. We introduce the resilience quadrant, which organizes the state of symptom networks on two domains: (1) healthy versus dysfunctional and (2) stable versus unstable. The quadrant captures the four commonly observed response trajectories to adversity along those dimensions: resilient trajectories in the face of adversity, as well as persistent symptoms despite treatment interventions. Subsequently, an empirical illustration, by means of a proof-of-principle, shows how simulated observations from four different network architectures lead to the four commonly observed responses to adversity. As such, we present a novel outlook on resilience by combining existing statistical symptom network models with simulation techniques. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

The present research develops a predictive model of prejudice. For nearly a century, psychology and other fields have sought to scientifically understand and describe the causes of prejudice. Numerous theories of prejudice now exist. Yet these theories are overwhelmingly defined verbally and thus lack the ability to precisely predict when and to what extent prejudice will emerge. The abundance of theory also raises the possibility of undetected overlap between constructs theorized to cause prejudice. Predictive models enable falsification and provide a way for the field to move forward. To this end, here we present 18 studies with ∼5,000 participants in seven phases of model development. After initially identifying major theorized causes of prejudice in the literature, we used a model selection approach to winnow constructs into a parsimonious predictive model of prejudice (Phases I and II). We confirm this model in a preregistered out-of-sample test (Phase III), test variations in operationalizations and boundary conditions (Phases IV and V), and test generalizability on a U.S. representative sample, an Indian sample, and a U.K. sample (Phase VI). Finally, we consulted the predictions of experts in the field to examine how well they align with our results (Phase VII). We believe this initial predictive model is limited and bad, but by developing a model that makes highly specific predictions, drawing on the state of the art, we hope to provide a foundation from which research can build to improve science of prejudice. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

Everything that happens has a multitude of causes, but people make causal judgments effortlessly. How do people select one particular cause (e.g., the lightning bolt that set the forest ablaze) out of the set of factors that contributed to the event (the oxygen in the air, the dry weather … )? Cognitive scientists have suggested that people make causal judgments about an event by simulating alternative ways things could have happened. We argue that this counterfactual theory explains many features of human causal intuitions, given two simple assumptions. First, people tend to imagine counterfactual possibilities that are both a priori likely and similar to what actually happened. Second, people judge that a factor C caused effect E if C and E are highly correlated across these counterfactual possibilities. In a reanalysis of existing empirical data, and a set of new experiments, we find that this theory uniquely accounts for people's causal intuitions. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

The optimal way to make decisions in many circumstances is to track the difference in evidence collected in favor of the options. The drift diffusion model (DDM) implements this approach and provides an excellent account of decisions and response times. However, existing DDM-based models of confidence exhibit certain deficits, and many theories of confidence have used alternative, nonoptimal models of decisions. Motivated by the historical success of the DDM, we ask whether simple extensions to this framework might allow it to better account for confidence. Motivated by the idea that the brain will not duplicate representations of evidence, in all model variants decisions and confidence are based on the same evidence accumulation process. We compare the models to benchmark results, and successfully apply four qualitative tests concerning the relationships between confidence, evidence, and time, in a new preregistered study. Using computationally cheap expressions to model confidence on a trial-by-trial basis, we find that a subset of model variants also provide a very good to excellent account of precise quantitative effects observed in confidence data. Specifically, our results favor the hypothesis that confidence reflects the strength of accumulated evidence penalized by the time taken to reach the decision (Bayesian readout), with the penalty applied not perfectly calibrated to the specific task context. These results suggest there is no need to abandon the DDM or single accumulator models to successfully account for confidence reports. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

Perception and action rely on integrating or binding different features of stimuli and responses. Such bindings are short-lived, but they can be retrieved for a limited amount of time if any of their features is reactivated. This is particularly true for stimulus-response bindings, allowing for flexible recycling of previous action plans. A relation to learning of stimulus-response associations suggests itself, and previous accounts have proposed binding as an initial step of forging associations in long-term memory. The evidence for this claim is surprisingly mixed, however. Here we propose a framework that explains previous failures to detect meaningful relations of binding and learning by highlighting the joint contribution of three variables: (a) decay, (b) the number of repetitions, and (c) the time elapsing between repetitions. Accounting for the interplay of these variables provides a promising blueprint for innovative experimental designs that bridge the gap between immediate bindings on the one hand and lasting associations in memory on the other hand. (PsycInfo Database Record (c) 2024 APA, all rights reserved).

To navigate changes within a highly dynamic and complex environment, it is crucial to compare current visual representations of a scene to previously formed representations stored in memory. This process of mental comparison requires integrating information from multiple sources to inform decisions about changes within the environment. In the present article, we combine a novel systems factorial technology change detection task (Blunden et al., 2022) with a set size manipulation. Participants were required to detect 0, 1, or 2 changes of low and high detectability between a memory and probe array of 1-4 spatially separated luminance discs. Analyses using systems factorial technology indicated that the processing architecture was consistent across set sizes but that capacity was always limited and decreased as the number of distractors increased. We developed a novel model of change detection based on the statistical principles of basic sampling theory (Palmer, 1990; Sewell et al., 2014). The sample size model, instantiated parametrically, predicts the architecture and capacity results a priori and quantitatively accounted for several key results observed in the data: (a) increasing set size acted to decrease sensitivity (d') in proportion to the square root of the number of items in the display; (b) the effect of redundancy benefited performance by a factor of the square root of the number of changes; and (c) the effect of change detectability was separable and independent of the sample size costs and redundancy benefits. (PsycInfo Database Record (c) 2024 APA, all rights reserved).