Pub Date : 2004-01-01DOI: 10.1080/13590840410001735027
K. K. E. L. L. Lrfpsg
{"title":"Do Yeasts Play Any Part in \"Candida\" (Fungal-type Dysbiosis)?: Discussion Paper","authors":"K. K. E. L. L. Lrfpsg","doi":"10.1080/13590840410001735027","DOIUrl":"https://doi.org/10.1080/13590840410001735027","url":null,"abstract":"","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"1 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2004-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840410001735027","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59777478","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2004-01-01DOI: 10.1080/1359084021000036792
K. K. E. L. L. Lrfpsg
{"title":"Moulds, Yeasts, Ascospores, Basidiospores, Algae and Lichens: Toxic and Allergic Reactions","authors":"K. K. E. L. L. Lrfpsg","doi":"10.1080/1359084021000036792","DOIUrl":"https://doi.org/10.1080/1359084021000036792","url":null,"abstract":"","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"1 1","pages":""},"PeriodicalIF":0.0,"publicationDate":"2004-01-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/1359084021000036792","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59775145","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-12-01DOI: 10.1080/13590840310001642003
M. Moss, R. Waring
Purpose: To explore the possible manifestations of abnormal levels of either cysteine or sulphate, whether high or low, and the ratio between them, in human subjects. Design: A case–control study of the plasma cysteine and sulphate levels and cysteine/sulphate ratio in chronically ill patients. Materials and Methods: Eighty-one chronically ill patients of a nutrition clinic were suspected of having abnormal levels of sulphate and/or cysteine. Their plasma was checked to determine their cysteine and sulphate levels and cysteine/sulphate ratio. These were compared with the results of 177 controls. They were grouped according to their results, and their symptoms were listed. Results: Only one patient had a ratio within the reference range, whereas 175 of the controls did. Patients already being treated with relevant nutritional supplements at the time of testing were less likely to have a ratio over 1000. Some close relatives shared abnormal test results, but manifested different symptoms. Conclusion: Patients with chronic conditions including myalgic encephalomyelitis, irritable bowel syndrome, migraine, arthritis, multiple chemical sensitivity and depression are likely to benefit from tests for cysteine and sulphate, and from treatment designed to improve these levels. Oral fish oil, vitamin B2, pantothenate and molybdenum, and Epsom salt baths may help patients with low sulphate. Vitamins B2 and B6, zinc and magnesium, and a low protein diet may reduce high cysteine levels. N-acetyl cysteine, zinc and vitamin C may help those with low cysteine levels. Patients with abnormal levels of sulphate might be counselled against working in polluted conditions, where efficient sulphate conjugation is required, and against using pesticides. They might be advised to be cautious in their use of drugs, and possibly vaccines too. Further work is suggested, to investigate to what extent abnormalities in cysteine and sulphate levels are genetically determined, and to test the efficacy of the treatments outlined, both at improving the cysteine and sulphate levels, and health.
{"title":"The Plasma Cysteine/Sulphate Ratio: A Possible Clinical Biomarker","authors":"M. Moss, R. Waring","doi":"10.1080/13590840310001642003","DOIUrl":"https://doi.org/10.1080/13590840310001642003","url":null,"abstract":"Purpose: To explore the possible manifestations of abnormal levels of either cysteine or sulphate, whether high or low, and the ratio between them, in human subjects. Design: A case–control study of the plasma cysteine and sulphate levels and cysteine/sulphate ratio in chronically ill patients. Materials and Methods: Eighty-one chronically ill patients of a nutrition clinic were suspected of having abnormal levels of sulphate and/or cysteine. Their plasma was checked to determine their cysteine and sulphate levels and cysteine/sulphate ratio. These were compared with the results of 177 controls. They were grouped according to their results, and their symptoms were listed. Results: Only one patient had a ratio within the reference range, whereas 175 of the controls did. Patients already being treated with relevant nutritional supplements at the time of testing were less likely to have a ratio over 1000. Some close relatives shared abnormal test results, but manifested different symptoms. Conclusion: Patients with chronic conditions including myalgic encephalomyelitis, irritable bowel syndrome, migraine, arthritis, multiple chemical sensitivity and depression are likely to benefit from tests for cysteine and sulphate, and from treatment designed to improve these levels. Oral fish oil, vitamin B2, pantothenate and molybdenum, and Epsom salt baths may help patients with low sulphate. Vitamins B2 and B6, zinc and magnesium, and a low protein diet may reduce high cysteine levels. N-acetyl cysteine, zinc and vitamin C may help those with low cysteine levels. Patients with abnormal levels of sulphate might be counselled against working in polluted conditions, where efficient sulphate conjugation is required, and against using pesticides. They might be advised to be cautious in their use of drugs, and possibly vaccines too. Further work is suggested, to investigate to what extent abnormalities in cysteine and sulphate levels are genetically determined, and to test the efficacy of the treatments outlined, both at improving the cysteine and sulphate levels, and health.","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"215-229"},"PeriodicalIF":0.0,"publicationDate":"2003-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001642003","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776731","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-12-01DOI: 10.1080/13590840310001649899
A. Ali
Purpose: Honey is widely used all over the world in complementary and alternative medicine in various disorders, including gastrointestinal lesions. Unfortunately, the therapeutic potential of honey has so far been neglected in modern medicine owing to a lack of systematic scientific studies. Recent studies using different models of experimentally induced acute and chronic gastric lesions in rats have demonstrated the gastric protection effects and molecular mechanisms of honey. As ammonia is highly cytotoxic, its production by urease of Helicobacter pylori makes it an important aggressive factor in mucosal injury. The present work was undertaken to evaluate potential gastric protection by honey and its possible mechanism(s) against ammonia‐induced gastric lesions in rats.Materials and Methods: Twenty‐four hour fasted rats were given 1 ml of ammonium hydroxide 1% intragastrically and were killed 1 hour later under deep ether anaesthesia. The gastric lesion index was calculated according to the method of T...
{"title":"Prevention of ammonia-induced gastric lesions in rats by natural honey","authors":"A. Ali","doi":"10.1080/13590840310001649899","DOIUrl":"https://doi.org/10.1080/13590840310001649899","url":null,"abstract":"Purpose: Honey is widely used all over the world in complementary and alternative medicine in various disorders, including gastrointestinal lesions. Unfortunately, the therapeutic potential of honey has so far been neglected in modern medicine owing to a lack of systematic scientific studies. Recent studies using different models of experimentally induced acute and chronic gastric lesions in rats have demonstrated the gastric protection effects and molecular mechanisms of honey. As ammonia is highly cytotoxic, its production by urease of Helicobacter pylori makes it an important aggressive factor in mucosal injury. The present work was undertaken to evaluate potential gastric protection by honey and its possible mechanism(s) against ammonia‐induced gastric lesions in rats.Materials and Methods: Twenty‐four hour fasted rats were given 1 ml of ammonium hydroxide 1% intragastrically and were killed 1 hour later under deep ether anaesthesia. The gastric lesion index was calculated according to the method of T...","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"239-246"},"PeriodicalIF":0.0,"publicationDate":"2003-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001649899","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776864","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-12-01DOI: 10.1080/13590840310001641987
R. Singh, D. Pella, R. Chopra, G. Cornelissen, F. Halberg
{"title":"Overview of Ubiquinone, in Memory of a Champion, Dr Emile G. Bliznakov","authors":"R. Singh, D. Pella, R. Chopra, G. Cornelissen, F. Halberg","doi":"10.1080/13590840310001641987","DOIUrl":"https://doi.org/10.1080/13590840310001641987","url":null,"abstract":"","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"211-214"},"PeriodicalIF":0.0,"publicationDate":"2003-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001641987","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776223","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-12-01DOI: 10.1080/13590840310001641996
P. Whiteley, P. MRPharmS
Purpose: To assess the prevalence of trans‐indolyl‐3‐acryloylglycine (IAcrGly) in the urine of people with a pervasive developmental disorder (PDD) compared with controls.Design: A case–control study of urinary IAcrGly levels.Materials and Methods: The electronic records of developmental and current medical status and urinary results of 500 patients formally diagnosed with PDD (autism, Asperger syndrome and autism spectrum disorder) and controls (no diagnosis) were examined for the presence of urinary IAcrGly identified by gradient elution high‐performance liquid chromatography (HPLC), co‐morbidity of epilepsy and current use of medication.Results: Urinary IAcrGly was identified in more samples from the PDD groups than the controls, although this was not significant (p=0.063, Fisher's exact test, two‐sided). The accompanying rates of epileptic disorders and current use of medication known to affect HPLC results were, however, also significantly higher in the PDD groups (χ2=8.070, df=1, p<0.004) (χ2=4.203,...
{"title":"What Makes Trans‐indolyl‐3‐acryloylglycine Identified by High‐performance Liquid Chromatography Relevant to Pervasive Developmental Disorders?","authors":"P. Whiteley, P. MRPharmS","doi":"10.1080/13590840310001641996","DOIUrl":"https://doi.org/10.1080/13590840310001641996","url":null,"abstract":"Purpose: To assess the prevalence of trans‐indolyl‐3‐acryloylglycine (IAcrGly) in the urine of people with a pervasive developmental disorder (PDD) compared with controls.Design: A case–control study of urinary IAcrGly levels.Materials and Methods: The electronic records of developmental and current medical status and urinary results of 500 patients formally diagnosed with PDD (autism, Asperger syndrome and autism spectrum disorder) and controls (no diagnosis) were examined for the presence of urinary IAcrGly identified by gradient elution high‐performance liquid chromatography (HPLC), co‐morbidity of epilepsy and current use of medication.Results: Urinary IAcrGly was identified in more samples from the PDD groups than the controls, although this was not significant (p=0.063, Fisher's exact test, two‐sided). The accompanying rates of epileptic disorders and current use of medication known to affect HPLC results were, however, also significantly higher in the PDD groups (χ2=8.070, df=1, p<0.004) (χ2=4.203,...","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"231-237"},"PeriodicalIF":0.0,"publicationDate":"2003-12-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001641996","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776299","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-09-01DOI: 10.1080/13590840310001619405
L. Plaskett
Purpose: To overview the physiological and health implications of carbohydrate deprivation as in the so‐called ‘Stone Age diet’ being used for allergy avoidance and weight loss.Design: To consider the types of diet that place humans at or beyond the borderline of ketosis and to define the conditions that trigger the strongly ketotic state in non‐diabetic subjects. To consider the health consequences of ketotic acidosis and the wider health impact of diets made up principally of protein and fat.Conclusions: Carbohydrate depletion occurs in starvation or at times when only protein and fat are being eaten. The resulting ketosis is due to the body's inability to carry out net synthesis of new carbohydrate from ketogenic metabolites. The resulting ketosis leads to acidosis, which may induce respiratory and oxygenation effects and negative effects upon renal function, bone mineral, calcium and nitrogen balance and upon the nervous system. The body's ability to form limited carbohydrate from either protein or gl...
{"title":"On the essentiality of dietary carbohydrate","authors":"L. Plaskett","doi":"10.1080/13590840310001619405","DOIUrl":"https://doi.org/10.1080/13590840310001619405","url":null,"abstract":"Purpose: To overview the physiological and health implications of carbohydrate deprivation as in the so‐called ‘Stone Age diet’ being used for allergy avoidance and weight loss.Design: To consider the types of diet that place humans at or beyond the borderline of ketosis and to define the conditions that trigger the strongly ketotic state in non‐diabetic subjects. To consider the health consequences of ketotic acidosis and the wider health impact of diets made up principally of protein and fat.Conclusions: Carbohydrate depletion occurs in starvation or at times when only protein and fat are being eaten. The resulting ketosis is due to the body's inability to carry out net synthesis of new carbohydrate from ketogenic metabolites. The resulting ketosis leads to acidosis, which may induce respiratory and oxygenation effects and negative effects upon renal function, bone mineral, calcium and nitrogen balance and upon the nervous system. The body's ability to form limited carbohydrate from either protein or gl...","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"161-168"},"PeriodicalIF":0.0,"publicationDate":"2003-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001619405","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776261","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-09-01DOI: 10.1080/13590840310001619414
N. Egaña, Mph Tm
Purpose: To present one possible framework by which to appraise the potential for golden rice to prevent vitamin A deficiency (VAD)/xerophthalmia.Design: Literature review.Method: Twenty‐nine articles were read and the term ‘issue’ was used for factors that the authors of the articles or secondary sources identified as being either causative or protective of VAD/xerophthalmia. The term ‘issue’ was not used exclusively to indicate that the factor caused VAD/xerophthalmia.Setting: VAD and xerophthalmia in developing countries.Main Outcome Measure: The number of issues identified and the frequency with which they occurred.Results: Twenty issues were identified with varying degrees of frequency. Socio‐economic/environmental, parasitic infestation and dark green leafy vegetables were the issues mentioned most frequently. Each issue was addressed in relation to golden rice. It is difficult to ascertain whether the frequency with which an issue was raised is relative to how important it is in relation to the oth...
{"title":"Vitamin A Deficiency and Golden Rice—A Literature Review","authors":"N. Egaña, Mph Tm","doi":"10.1080/13590840310001619414","DOIUrl":"https://doi.org/10.1080/13590840310001619414","url":null,"abstract":"Purpose: To present one possible framework by which to appraise the potential for golden rice to prevent vitamin A deficiency (VAD)/xerophthalmia.Design: Literature review.Method: Twenty‐nine articles were read and the term ‘issue’ was used for factors that the authors of the articles or secondary sources identified as being either causative or protective of VAD/xerophthalmia. The term ‘issue’ was not used exclusively to indicate that the factor caused VAD/xerophthalmia.Setting: VAD and xerophthalmia in developing countries.Main Outcome Measure: The number of issues identified and the frequency with which they occurred.Results: Twenty issues were identified with varying degrees of frequency. Socio‐economic/environmental, parasitic infestation and dark green leafy vegetables were the issues mentioned most frequently. Each issue was addressed in relation to golden rice. It is difficult to ascertain whether the frequency with which an issue was raised is relative to how important it is in relation to the oth...","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"169-184"},"PeriodicalIF":0.0,"publicationDate":"2003-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001619414","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776325","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2003-09-01DOI: 10.1080/13590840310001619397
S. Lindeberg, M. Phd, S. Boyd, Eaton Md
Purpose: To explore the possibility that a Palaeolithic diet, i.e. one that corresponds to what was available in any of the ecological niches of pre‐agricultural humans (1.5 million–10,000 years bp), is optimal in the prevention of age‐related degenerative disease.Design: Literature review.Materials and Methods: Between 1985 and December 2002, more than 200 scientific journals in medicine, nutrition, biology and anthropology were systematically screened for relevant papers. Computer‐based searches and studies of reference lists in journals and books provided a vast number of additional papers.Results: Increasing evidence suggests that a Palaeolithic diet based on lean meat, fish, vegetables and fruit may be effective in the prevention and treatment of common Western diseases. Avoiding dairy products, margarine, oils, refined sugar and cereals, which provide 70% or more of the dietary intake in northern European populations, may be advisable. Atherosclerosis is highly dependent on dietary manipulation in a...
{"title":"Biological and Clinical Potential of a Palaeolithic Diet","authors":"S. Lindeberg, M. Phd, S. Boyd, Eaton Md","doi":"10.1080/13590840310001619397","DOIUrl":"https://doi.org/10.1080/13590840310001619397","url":null,"abstract":"Purpose: To explore the possibility that a Palaeolithic diet, i.e. one that corresponds to what was available in any of the ecological niches of pre‐agricultural humans (1.5 million–10,000 years bp), is optimal in the prevention of age‐related degenerative disease.Design: Literature review.Materials and Methods: Between 1985 and December 2002, more than 200 scientific journals in medicine, nutrition, biology and anthropology were systematically screened for relevant papers. Computer‐based searches and studies of reference lists in journals and books provided a vast number of additional papers.Results: Increasing evidence suggests that a Palaeolithic diet based on lean meat, fish, vegetables and fruit may be effective in the prevention and treatment of common Western diseases. Avoiding dairy products, margarine, oils, refined sugar and cereals, which provide 70% or more of the dietary intake in northern European populations, may be advisable. Atherosclerosis is highly dependent on dietary manipulation in a...","PeriodicalId":88013,"journal":{"name":"Journal of nutritional & environmental medicine","volume":"13 1","pages":"149-160"},"PeriodicalIF":0.0,"publicationDate":"2003-09-01","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"https://sci-hub-pdf.com/10.1080/13590840310001619397","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"59776481","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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