Pub Date : 2021-07-06DOI: 10.5935/1984-0063.20220014
Michael John Stacey Pengelly, J. Guy, Nathan Elsworthy, A. Scanlan, M. Lastella
Athlete chronotype has been documented to underpin diurnal variations in skill execution across various team sports. However, no research has explored the effects of athlete chronotype on basketball-specific skills at different times of the day. Therefore, the aim of this study was to explore diurnal variations in basketball shooting accuracy according to chronotype. Professional, male basketball players (n = 13) completed a Morningness- Eveningness Questionnaire and were categorised into chronotypes using a tertile split technique (morning-types: n = 4; neither-types: n = 4; evening-types: n = 5). Players completed separate trials of a shooting accuracy test in the morning (08:00-09:30h) and afternoon (15:00-16:30h) with each trial consisting of 20 shots attempted from four court locations at either two- or three-point distances and one-shot location from the free-throw line (100 shots in total). Each shot attempt was scored using a 0-3-point scale with higher scores awarded to more accurate shots. Non-significant ( p >0.05) differences in shooting scores were evident between morning and afternoon trials for each chronotype group, with small-large effects in shooting scores favouring the morning across groups. Moreover, non-significant ( p >0.05) differences in shooting scores were apparent between chronotype groups in the morning ( small-large effects) and afternoon ( moderate-large effects). Shooting accuracy appears to remain consistent across morning and afternoon performances irrespective of player chronotype in a professional basketball team, suggesting coaches may not need to schedule training sessions involving shooting tasks at specific times of the day to optimise shooting accuracy in players.
{"title":"Player chronotype does not affect shooting accuracy at different times of the day in a professional, male basketball team: a pilot study","authors":"Michael John Stacey Pengelly, J. Guy, Nathan Elsworthy, A. Scanlan, M. Lastella","doi":"10.5935/1984-0063.20220014","DOIUrl":"https://doi.org/10.5935/1984-0063.20220014","url":null,"abstract":"Athlete chronotype has been documented to underpin diurnal variations in skill execution across various team sports. However, no research has explored the effects of athlete chronotype on basketball-specific skills at different times of the day. Therefore, the aim of this study was to explore diurnal variations in basketball shooting accuracy according to chronotype. Professional, male basketball players (n = 13) completed a Morningness- Eveningness Questionnaire and were categorised into chronotypes using a tertile split technique (morning-types: n = 4; neither-types: n = 4; evening-types: n = 5). Players completed separate trials of a shooting accuracy test in the morning (08:00-09:30h) and afternoon (15:00-16:30h) with each trial consisting of 20 shots attempted from four court locations at either two- or three-point distances and one-shot location from the free-throw line (100 shots in total). Each shot attempt was scored using a 0-3-point scale with higher scores awarded to more accurate shots. Non-significant ( p >0.05) differences in shooting scores were evident between morning and afternoon trials for each chronotype group, with small-large effects in shooting scores favouring the morning across groups. Moreover, non-significant ( p >0.05) differences in shooting scores were apparent between chronotype groups in the morning ( small-large effects) and afternoon ( moderate-large effects). Shooting accuracy appears to remain consistent across morning and afternoon performances irrespective of player chronotype in a professional basketball team, suggesting coaches may not need to schedule training sessions involving shooting tasks at specific times of the day to optimise shooting accuracy in players.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"24 1","pages":"149 - 155"},"PeriodicalIF":1.6,"publicationDate":"2021-07-06","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"85860350","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-06-24DOI: 10.5935/1984-0063.20220018
M. Hein, J. Lanquart, A. Mungo, G. Loas
Objective In the general population, co-morbid insomnia and sleep apnoea (COMISA) is associated with higher risk of cardiovascular diseases (CVD). However, despite a high prevalence of COMISA in type 2 diabetics, no study has investigated its potential implication in the negative cardiovascular outcome of this particular subpopulation. The aim of this study was therefore to examine the risk of CVD associated with COMISA in type 2 diabetics. Methods Data from 471 type 2 diabetics recruited from the clinical database of the Erasme Hospital sleep laboratory were analysed. Only type 2 diabetics with SCORE index ≥5% were included in the group at high risk of CVD. Logistic regression analyses were conducted to examine the risk of CVD associated with COMISA in type 2 diabetics. Results A high risk of CVD was present in 32.9% of type 2 diabetics. After adjustment for the main confounding factors associated with cardiovascular risk, multivariate logistic regression analysis revealed that unlike obstructive sleep apnoea syndrome or insomnia alone, only COMISA was associated with higher risk of CVD in type 2 diabetics. Discussion In our study, we have demonstrated that unlike its components alone, only COMISA was associated with higher risk of CVD in type 2 diabetics, which highlights the importance of the central role played by the negative synergistic effect of COMISA on the cardiovascular outcome in this particular subpopulation. Thus, given these elements, more systematic research and adequate therapeutic management of COMISA seem to be necessary to allow better cardiovascular prevention in type 2 diabetics.
{"title":"Cardiovascular risk associated with co-morbid insomnia and sleep apnoea (COMISA) in type 2 diabetics","authors":"M. Hein, J. Lanquart, A. Mungo, G. Loas","doi":"10.5935/1984-0063.20220018","DOIUrl":"https://doi.org/10.5935/1984-0063.20220018","url":null,"abstract":"Objective In the general population, co-morbid insomnia and sleep apnoea (COMISA) is associated with higher risk of cardiovascular diseases (CVD). However, despite a high prevalence of COMISA in type 2 diabetics, no study has investigated its potential implication in the negative cardiovascular outcome of this particular subpopulation. The aim of this study was therefore to examine the risk of CVD associated with COMISA in type 2 diabetics. Methods Data from 471 type 2 diabetics recruited from the clinical database of the Erasme Hospital sleep laboratory were analysed. Only type 2 diabetics with SCORE index ≥5% were included in the group at high risk of CVD. Logistic regression analyses were conducted to examine the risk of CVD associated with COMISA in type 2 diabetics. Results A high risk of CVD was present in 32.9% of type 2 diabetics. After adjustment for the main confounding factors associated with cardiovascular risk, multivariate logistic regression analysis revealed that unlike obstructive sleep apnoea syndrome or insomnia alone, only COMISA was associated with higher risk of CVD in type 2 diabetics. Discussion In our study, we have demonstrated that unlike its components alone, only COMISA was associated with higher risk of CVD in type 2 diabetics, which highlights the importance of the central role played by the negative synergistic effect of COMISA on the cardiovascular outcome in this particular subpopulation. Thus, given these elements, more systematic research and adequate therapeutic management of COMISA seem to be necessary to allow better cardiovascular prevention in type 2 diabetics.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"88 1","pages":"184 - 194"},"PeriodicalIF":1.6,"publicationDate":"2021-06-24","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"75708602","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-06-21DOI: 10.5935/1984-0063.20220022
A. Orjuela-Cañón, Andrés L. Jutinico, M. Bazurto-Zapata, E. Duenas-Meza
Objective Physiological networks have recently been employed as an alternative to analyze the interaction of the human body. Within this option, different systems are analyzed as nodes inside a communication network as well how information fows. Several studies have been proposed to study sleep subjects with the help of the Granger causality computation over electroencephalographic and heart rate variability signals. However, following this methodology, novel approximations for children subjects are presented here, where comparison between adult and children sleep is followed through the obtained connectivities. Methods Data from ten adults and children were retrospectively extracted from polysomnography records. Database was extracted from people suspected of having sleep disorders who participated in a previous study. Connectivity was computed based on Granger causality, according to preprocessing of similar studies in this feld. A comparison for adults and children groups with a chi-square test was followed, employing the results of the Granger causality measures. Results Results show that differences were mainly established for nodes inside the brain network connectivity. Additionally, for interactions between brain and heart networks, it was brought to light that children physiology sends more information from heart to brain nodes compared to the adults group. Discussion This study represents a frst sight to children sleep analysis, employing the Granger causality computation. It contributes to understand sleep in children employing measurements from physiological signals. Preliminary fndings suggest more interactions inside the brain network for children group compared to adults group.
{"title":"Sleep physiological network analysis in children","authors":"A. Orjuela-Cañón, Andrés L. Jutinico, M. Bazurto-Zapata, E. Duenas-Meza","doi":"10.5935/1984-0063.20220022","DOIUrl":"https://doi.org/10.5935/1984-0063.20220022","url":null,"abstract":"Objective Physiological networks have recently been employed as an alternative to analyze the interaction of the human body. Within this option, different systems are analyzed as nodes inside a communication network as well how information fows. Several studies have been proposed to study sleep subjects with the help of the Granger causality computation over electroencephalographic and heart rate variability signals. However, following this methodology, novel approximations for children subjects are presented here, where comparison between adult and children sleep is followed through the obtained connectivities. Methods Data from ten adults and children were retrospectively extracted from polysomnography records. Database was extracted from people suspected of having sleep disorders who participated in a previous study. Connectivity was computed based on Granger causality, according to preprocessing of similar studies in this feld. A comparison for adults and children groups with a chi-square test was followed, employing the results of the Granger causality measures. Results Results show that differences were mainly established for nodes inside the brain network connectivity. Additionally, for interactions between brain and heart networks, it was brought to light that children physiology sends more information from heart to brain nodes compared to the adults group. Discussion This study represents a frst sight to children sleep analysis, employing the Granger causality computation. It contributes to understand sleep in children employing measurements from physiological signals. Preliminary fndings suggest more interactions inside the brain network for children group compared to adults group.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"374 1","pages":"215 - 223"},"PeriodicalIF":1.6,"publicationDate":"2021-06-21","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"75123613","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-06-19DOI: 10.5935/1984-0063.20220001
Rashmi Soori, Nandakishore Baikunje, I. D’sa, N. Bhushan, B. Nagabhushana, G. Hosmane
Obstructive sleep apnoea (OSA) is the major underlying co-morbidity in many of the non-communicable diseases (NCD) due to obesity as a common risk factor. Incidence and prevalence of OSA is on the constant rise ever since this entity came to forefront three decades ago. Precise treatment of underlying OSA is extremely important in major NCDs like diabetes mellitus, hypertension, endocrine disorders and vascular diseases. OSA is subcategorized in to mild, moderate and severe based of apnoea-hypopnea index (AHI). Based on the severity grading, treatment of OSA ranges from life style modifications to oral appliances, continuous positive airway pressure (CPAP) and surgeries. AHI system of severity grading in OSA has several inherent shortcomings and using AHI system for severity grading as the holy grail is likely to be counter-productive. AHI system equates apnoea and hypopnea as equal events, whereas physiological effects vary significantly. AHI system does not account duration of apnoea or body position during apnoeic events. We discuss at length the pitfalls of AHI system of severity grading in OSA.
{"title":"Pitfalls of AHI system of severity grading in obstructive sleep apnoea","authors":"Rashmi Soori, Nandakishore Baikunje, I. D’sa, N. Bhushan, B. Nagabhushana, G. Hosmane","doi":"10.5935/1984-0063.20220001","DOIUrl":"https://doi.org/10.5935/1984-0063.20220001","url":null,"abstract":"Obstructive sleep apnoea (OSA) is the major underlying co-morbidity in many of the non-communicable diseases (NCD) due to obesity as a common risk factor. Incidence and prevalence of OSA is on the constant rise ever since this entity came to forefront three decades ago. Precise treatment of underlying OSA is extremely important in major NCDs like diabetes mellitus, hypertension, endocrine disorders and vascular diseases. OSA is subcategorized in to mild, moderate and severe based of apnoea-hypopnea index (AHI). Based on the severity grading, treatment of OSA ranges from life style modifications to oral appliances, continuous positive airway pressure (CPAP) and surgeries. AHI system of severity grading in OSA has several inherent shortcomings and using AHI system for severity grading as the holy grail is likely to be counter-productive. AHI system equates apnoea and hypopnea as equal events, whereas physiological effects vary significantly. AHI system does not account duration of apnoea or body position during apnoeic events. We discuss at length the pitfalls of AHI system of severity grading in OSA.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"22 1","pages":"285 - 288"},"PeriodicalIF":1.6,"publicationDate":"2021-06-19","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"72643319","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-06-18DOI: 10.5935/1984-0063.20220031
André Oliveira, Beatriz Pereira, P. Rosário, P. Magalhães
ABSTRACT Introduction: Bedtime procrastination is the deliberate delay of the time an individual goes to bed in the absence of external reasons. The COVID-19 pandemic has pushed families to establish new routines and ways of managing newfound roles and responsibilities. This scenario is likely to exacerbate bedtime procrastination due to, for example, a challenge in balancing professional and personal life. Objective: The aim is to present preliminary findings regarding bedtime procrastination and its relation to sociodemographic characteristics, sleep routines, perceived daily fatigue, dinnertime, and activities performed near bedtime, during the second lockdown in Portugal. Material and Methods: A cross-sectional online survey was conducted with a sample of 560 adults. Results: During home confinement, most people (79.46%) delayed their bedtime. However, this delay does not seem to be affecting the number of hours of sleep, as 88.60% were sleeping the recommended or appropriate number of hours. Nevertheless, most of the participants reported feeling tired throughout the day (53.04%), and individuals who reported to have procrastinated their sleep are those who reported more tiredness ( r pb =.33, p <.01). Additionally, bedtime procrastination is positively associated with findings related to dinnertime (e.g., dinner between 9 p.m. and 10 p.m., r pb =.19, p<.01) and with engagement in activities near bedtime (e.g., studying/working, r pb =.39, p <.01). Conclusion: Current data shows relationships between bedtime procrastination and most of the studied variables. Specifically, findings indicate that lack of routines, especially scheduled nighttime routines (e.g., studying/working near bedtime), may have contributed to bedtime procrastination during the second lockdown in Portugal.
{"title":"Bedtime procrastination during the second COVID-19 lockdown in Portugal","authors":"André Oliveira, Beatriz Pereira, P. Rosário, P. Magalhães","doi":"10.5935/1984-0063.20220031","DOIUrl":"https://doi.org/10.5935/1984-0063.20220031","url":null,"abstract":"ABSTRACT Introduction: Bedtime procrastination is the deliberate delay of the time an individual goes to bed in the absence of external reasons. The COVID-19 pandemic has pushed families to establish new routines and ways of managing newfound roles and responsibilities. This scenario is likely to exacerbate bedtime procrastination due to, for example, a challenge in balancing professional and personal life. Objective: The aim is to present preliminary findings regarding bedtime procrastination and its relation to sociodemographic characteristics, sleep routines, perceived daily fatigue, dinnertime, and activities performed near bedtime, during the second lockdown in Portugal. Material and Methods: A cross-sectional online survey was conducted with a sample of 560 adults. Results: During home confinement, most people (79.46%) delayed their bedtime. However, this delay does not seem to be affecting the number of hours of sleep, as 88.60% were sleeping the recommended or appropriate number of hours. Nevertheless, most of the participants reported feeling tired throughout the day (53.04%), and individuals who reported to have procrastinated their sleep are those who reported more tiredness ( r pb =.33, p <.01). Additionally, bedtime procrastination is positively associated with findings related to dinnertime (e.g., dinner between 9 p.m. and 10 p.m., r pb =.19, p<.01) and with engagement in activities near bedtime (e.g., studying/working, r pb =.39, p <.01). Conclusion: Current data shows relationships between bedtime procrastination and most of the studied variables. Specifically, findings indicate that lack of routines, especially scheduled nighttime routines (e.g., studying/working near bedtime), may have contributed to bedtime procrastination during the second lockdown in Portugal.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"38 1","pages":"105 - 111"},"PeriodicalIF":1.6,"publicationDate":"2021-06-18","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"81386227","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-05-25DOI: 10.5935/1984-0063.20220024
Laila Taghvaee, Amir Ali Mazandarani
Objective The current study examines the relationship between sleep duration and variability with sensation- seeking and risky behaviors in a sample of Iranian undergraduate students. Methods The participants in this study were 385 undergraduate students in Tehran. To collect data, we used the Sensation Seeking Scale, Risk Behavior Scale, and two questions related to sleep duration and sleep variability. Results The results showed a positive relationship between sleep deprivation and irregular sleep schedule with sensation-seeking and risk behavior tendencies in students. Discussion This association could point to an indirect impact of sleep deprivation on sensationseeking and risk behavior, for example, by altering brain regions responsible for self- control.
{"title":"Poor sleep is associated with sensation-seeking and risk behavior in college students","authors":"Laila Taghvaee, Amir Ali Mazandarani","doi":"10.5935/1984-0063.20220024","DOIUrl":"https://doi.org/10.5935/1984-0063.20220024","url":null,"abstract":"Objective The current study examines the relationship between sleep duration and variability with sensation- seeking and risky behaviors in a sample of Iranian undergraduate students. Methods The participants in this study were 385 undergraduate students in Tehran. To collect data, we used the Sensation Seeking Scale, Risk Behavior Scale, and two questions related to sleep duration and sleep variability. Results The results showed a positive relationship between sleep deprivation and irregular sleep schedule with sensation-seeking and risk behavior tendencies in students. Discussion This association could point to an indirect impact of sleep deprivation on sensationseeking and risk behavior, for example, by altering brain regions responsible for self- control.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"18 1","pages":"249 - 256"},"PeriodicalIF":1.6,"publicationDate":"2021-05-25","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77807271","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-05-13DOI: 10.5935/1984-0063.20220012
M. Guimarães, Pedro Azevedo, S. Barros-Vieira, M. Elbaz, D. Léger, Ana Paula Hermont
Aims A cross-sectional study was designed to evaluate the effectiveness of a mandibular advancement device (MAD) with respect to respiratory and sleep parameters among miners with obstructive sleep apnea syndrome (OSAS) and primary snore. Methods The target sample was composed by 102 Brazilian miners with a history of non-adherence to continuous positive airway pressure. All patients were treated with a MAD and underwent pre and post-treatment full-night polysomnography. Ethical approval and consents were obtained. Bivariate and logistic regression analyses were conducted. The level of statistical significance was set at 5%. Results After the treatment with MAD, 71.8% of patients presented a decrease ≥ 50% in the basal apnea-hypopnea index (AHI), 51.2% presented an AHI < 5 events/h and 83.3% reached an AHI<10/h, whereas 22.5% did not show any changes and 7.5% of the sample presented an increase in the AHI (p<0.05). There was an increase in the mean SpO2 nadir (p<0.001) and in the baseline duration of the REM sleep stage (p<0.05). The MAD significantly decreased snore events (p<0.05). Multivariate analysis did not identify predictive factors related to therapy success (decrease ≥ 50% of AHI). However, basal AHI was a significant predictor related to the secondary endpoint (AHI<10/h) (OR= 1.06, IC 95%1.00-1.13, p=0.007). Conclusions The MAD therapy showed significant improvements in AHI, minimum oxygen saturation, REM sleep and snoring.
目的横断面研究旨在评估下颌推进装置(MAD)在阻塞性睡眠呼吸暂停综合征(OSAS)和原发性打鼾矿工的呼吸和睡眠参数方面的有效性。方法目标样本由102名巴西矿工组成，他们有不坚持持续气道正压的历史。所有患者都接受了MAD治疗，并在治疗前后进行了夜间多导睡眠图检查。获得了伦理批准和同意。进行了双变量和逻辑回归分析。统计学显著性水平设为5%。结果经MAD治疗后，71.8%的患者基础呼吸暂停低通气指数(AHI)下降≥50%，51.2%的患者AHI< 5事件/h, 83.3%的患者AHI<10事件/h, 22.5%的患者AHI无变化，7.5%的患者AHI升高(p<0.05)。平均SpO2最低点增加(p<0.001)， REM睡眠阶段基线持续时间增加(p<0.05)。MAD显著降低了打鼾事件(p<0.05)。多变量分析未发现与治疗成功相关的预测因素(AHI降低≥50%)。然而，基础AHI是与次要终点(AHI<10/h)相关的重要预测因子(OR= 1.06, IC 95%1.00-1.13, p=0.007)。结论MAD治疗对AHI、最低血氧饱和度、快速眼动睡眠和打鼾均有显著改善。
{"title":"When adherence to CPAP fails, how do we treat workers with obstructive sleep apnea?","authors":"M. Guimarães, Pedro Azevedo, S. Barros-Vieira, M. Elbaz, D. Léger, Ana Paula Hermont","doi":"10.5935/1984-0063.20220012","DOIUrl":"https://doi.org/10.5935/1984-0063.20220012","url":null,"abstract":"Aims A cross-sectional study was designed to evaluate the effectiveness of a mandibular advancement device (MAD) with respect to respiratory and sleep parameters among miners with obstructive sleep apnea syndrome (OSAS) and primary snore. Methods The target sample was composed by 102 Brazilian miners with a history of non-adherence to continuous positive airway pressure. All patients were treated with a MAD and underwent pre and post-treatment full-night polysomnography. Ethical approval and consents were obtained. Bivariate and logistic regression analyses were conducted. The level of statistical significance was set at 5%. Results After the treatment with MAD, 71.8% of patients presented a decrease ≥ 50% in the basal apnea-hypopnea index (AHI), 51.2% presented an AHI < 5 events/h and 83.3% reached an AHI<10/h, whereas 22.5% did not show any changes and 7.5% of the sample presented an increase in the AHI (p<0.05). There was an increase in the mean SpO2 nadir (p<0.001) and in the baseline duration of the REM sleep stage (p<0.05). The MAD significantly decreased snore events (p<0.05). Multivariate analysis did not identify predictive factors related to therapy success (decrease ≥ 50% of AHI). However, basal AHI was a significant predictor related to the secondary endpoint (AHI<10/h) (OR= 1.06, IC 95%1.00-1.13, p=0.007). Conclusions The MAD therapy showed significant improvements in AHI, minimum oxygen saturation, REM sleep and snoring.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"64 5 1","pages":"135 - 142"},"PeriodicalIF":1.6,"publicationDate":"2021-05-13","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"77884162","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-05-04DOI: 10.5935/1984-0063.20220002
Jenny E. Simon
Cardiovascular diseases (CVDs) are the leading cause of death globally. Among the objectives of preventive cardiology is the design to understand and neutralise the clinical overlap between disordered sleep and CVDs. Seldom do studies measure ‘sleep health’ beyond the absence of disease. Explored herein are the cardiovascular (CV) outcomes of sleep health on the grounds that, more than a corollary of sleep disorder research, sleep health constitutes a critical determinant of cardiac health and disease not unlike diet and physical activity. That sleep interventions can reverse the CV consequences of poor sleep habits lends credence to the notion that sleep health benefits CV health, and that the importance of sleep health percolates far beyond sleep disorder research. Overall, sleep health, and its practicable correlate: sleep hygiene, are clinical imperatives in the foreseeable future of cardiology in the 24-hour society.
{"title":"Beyond the absence of sleep disorder: spotlighting the cardiovascular benefits of sleep health","authors":"Jenny E. Simon","doi":"10.5935/1984-0063.20220002","DOIUrl":"https://doi.org/10.5935/1984-0063.20220002","url":null,"abstract":"Cardiovascular diseases (CVDs) are the leading cause of death globally. Among the objectives of preventive cardiology is the design to understand and neutralise the clinical overlap between disordered sleep and CVDs. Seldom do studies measure ‘sleep health’ beyond the absence of disease. Explored herein are the cardiovascular (CV) outcomes of sleep health on the grounds that, more than a corollary of sleep disorder research, sleep health constitutes a critical determinant of cardiac health and disease not unlike diet and physical activity. That sleep interventions can reverse the CV consequences of poor sleep habits lends credence to the notion that sleep health benefits CV health, and that the importance of sleep health percolates far beyond sleep disorder research. Overall, sleep health, and its practicable correlate: sleep hygiene, are clinical imperatives in the foreseeable future of cardiology in the 24-hour society.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"9 1","pages":"289 - 292"},"PeriodicalIF":1.6,"publicationDate":"2021-05-04","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"74626365","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
Pub Date : 2021-04-26DOI: 10.5935/1984-0063.20220019
Huriye Çakmak
Quarantine and isolation are the most important public health measures used to prevent the transmission of infectious diseases among individuals. Quarantine and social isolation cause some health problems, such as depressive symptoms, sleep disturbances, anxiety, irritability, feeling of loneliness, helplessness, and trauma and post-traumatic stress symptoms. In this study, we aimed to examine early phase of COVID-19 quarantine impacted insomnia symptoms in Turkish families. The population of the study comprised students attending the Department of Healthcare Services of Eldivan Health Vocational School at Çankırı Karatekin University and their families. In total, 564 students and their families completed online surveys. Data was collected with Google forms. Snowball sampling was used as the data collection method. Female participants, those who gained weight during home quarantine, those who put their phones under their pillow or at the bedside and in another room, and those who used a phone or tablet before sleeping were found to have significantly more insomnia compared to others. The mean day of uninterrupted home quarantine was higher for participants experiencing insomnia (p<0.05). This study showed that individuals experienced sleep problems during the home quarantine.
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Pub Date : 2021-04-15DOI: 10.5935/1984-0063.20220003
F. Ozen, Z. Yeğin, Z. Sağlam, F. Yavlal, H. Koç, Celal Ulaşoğlu
Excessive daytime sleepiness (EDS) is a common complaint encountered in clinical practice with serious consequences both for individual and society since it can increase the ratio of motor vehicle accidents, work- related incidents, and deaths. Moreover, it also manifests less serious individual consequences. This study aimed to investigate the potential role of PER3-VNTR, 5-HTT-LPR, and 5-HTT-VNTR in terms of constituting liability to EDS. Two hundred eighteen participants (93 complaining about daytime sleepiness and 125 individuals with no serious complaint) were recruited in the study. General daytime of sleepiness was quantified with Epworth sleepiness scale (ESS). DNA extractions were performed from collected blood samples with standart salting-out procedure and genotyped. ESS scores displayed difference between individuals suffering from sleep disturbances and other individuals with values of 12.75±4.55 and 6.34±4.26, respectively. PER3- VNTR and 5-HTT-LPR genotypes did not display association with mean ESS scores. However, 5-HTT-VNTR genotypes showed significant association with mean ESS scores; individuals with 10/10 genotypes had the highest ESS score reflecting this genotype as a liability factor for EDS. We strongly recommend further studies based on circadian/serotonin pathway genes in different populations to reach to a consensus and highlight sleep genetic marker genes which then can be the future targets of pharmacological treatment studies for sleep problems.
{"title":"Association analysis of Epworth Sleepiness Scale (ESS) scores with serotonin transporter (5-HTT- LPR, 5-HTT-VNTR) and circadian (PER3-VNTR) genes","authors":"F. Ozen, Z. Yeğin, Z. Sağlam, F. Yavlal, H. Koç, Celal Ulaşoğlu","doi":"10.5935/1984-0063.20220003","DOIUrl":"https://doi.org/10.5935/1984-0063.20220003","url":null,"abstract":"Excessive daytime sleepiness (EDS) is a common complaint encountered in clinical practice with serious consequences both for individual and society since it can increase the ratio of motor vehicle accidents, work- related incidents, and deaths. Moreover, it also manifests less serious individual consequences. This study aimed to investigate the potential role of PER3-VNTR, 5-HTT-LPR, and 5-HTT-VNTR in terms of constituting liability to EDS. Two hundred eighteen participants (93 complaining about daytime sleepiness and 125 individuals with no serious complaint) were recruited in the study. General daytime of sleepiness was quantified with Epworth sleepiness scale (ESS). DNA extractions were performed from collected blood samples with standart salting-out procedure and genotyped. ESS scores displayed difference between individuals suffering from sleep disturbances and other individuals with values of 12.75±4.55 and 6.34±4.26, respectively. PER3- VNTR and 5-HTT-LPR genotypes did not display association with mean ESS scores. However, 5-HTT-VNTR genotypes showed significant association with mean ESS scores; individuals with 10/10 genotypes had the highest ESS score reflecting this genotype as a liability factor for EDS. We strongly recommend further studies based on circadian/serotonin pathway genes in different populations to reach to a consensus and highlight sleep genetic marker genes which then can be the future targets of pharmacological treatment studies for sleep problems.","PeriodicalId":21848,"journal":{"name":"Sleep Science","volume":"25 1","pages":"110 - 115"},"PeriodicalIF":1.6,"publicationDate":"2021-04-15","publicationTypes":"Journal Article","fieldsOfStudy":null,"isOpenAccess":false,"openAccessPdf":"","citationCount":null,"resultStr":null,"platform":"Semanticscholar","paperid":"78066432","PeriodicalName":null,"FirstCategoryId":null,"ListUrlMain":null,"RegionNum":0,"RegionCategory":"","ArticlePicture":[],"TitleCN":null,"AbstractTextCN":null,"PMCID":"","EPubDate":null,"PubModel":null,"JCR":null,"JCRName":null,"Score":null,"Total":0}
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